Bi-local baryon interpolating fields with two flavours

We construct bi-local interpolating field operators for baryons consisting of three quarks with two flavors, assuming good isospin symmetry. We use the restrictions following from the Pauli principle to derive relations/identities among the baryon operators with identical quantum numbers. Such relations that follow from the combined spatial, Dirac, color, and isospin Fierz transformations may be called the (total/complete) Fierz identities. These relations reduce the number of independent baryon operators with any given spin and isospin. We also study the Abelian and non-Abelian chiral transformation properties of these fields and place them into baryon chiral multiplets. Thus we derive the independent baryon interpolating fields with given values of spin (Lorentz group representation), chiral symmetry ($U_L(2) \times U_R(2)$ group representation) and isospin appropriate for the first angular excited states of the nucleon.Comment: 15 pages, 4 tables, accepted by EPJ

Chiral Baryon Fields in the QCD Sum Rule

We study the structure of local baryon fields using the method of QCD sum rule. We only consider the single baryon fields and calculate their operator product expansions. We find that the octet baryon fields belonging to the chiral representations [(3,3*)+(3*,3)] and [(8,1)+(1,8)] and the decuplet baryon fields belonging to the chiral representations [(3,6)+(6,3)] lead to the baryon masses which are consistent with the experimental data of ground baryon masses. We also calculate their decay constants, check our normalizations for baryon fields in PRD81:054002(2010) and find that they are well-defined.Comment: 12 pages, 6 figure, 1 table, accepted by EPJ

Twist-3 Distribute Amplitude of the Pion in QCD Sum Rules

We apply the background field method to calculate the moments of the pion two-particles twist-3 distribution amplitude (DA) $\phi_p(\xi)$ in QCD sum rules. In this paper,we do not use the equation of motion for the quarks inside the pion since they are not on shell and introduce a new parameter $m_0^p$ to be determined. We get the parameter $m_0^p\approx1.30GeV$ in this approach. If assuming the expansion of $\phi_p(\xi)$ in the series in Gegenbauer polynomials $C_n^{1/2}(\xi)$, one can obtain its approximate expression which can be determined by its first few moments.Comment: 12 pages, 3 figure

Baryon Tri-local Interpolating Fields

We systematically investigate tri-local (non-local) three-quark baryon fields with U_L(2)*U_R(2) chiral symmetry, according to their Lorentz and isospin (flavor) group representations. We note that they can also be called as "nucleon wave functions" due to this full non-locality. We study their chiral transformation properties and find all the possible chiral multiplets consisting J=1/2 and J=3/2 baryon fields. We find that the axial coupling constant |g_A| = 5/3 is only for nucleon fields belonging to the chiral representation (1/2,1)+(1,1/2) which contains both nucleon fields and Delta fields. Moreover, all the nucleon fields belonging to this representation have |g_A| = 5/3.Comment: 8 pages, 3 tables, accepted by EPJ

Adaptive Robust Fault-Tolerant Control for Linear MIMO Systems with Unmatched Uncertainties

In this paper, two novel fault-tolerant control design approaches are proposed for linear MIMO systems with actuator additive faults, multiplicative faults and unmatched uncertainties. For time-varying multiplicative and additive faults, new adaptive laws and additive compensation functions are proposed. A set of conditions is developed such that the unmatched uncertainties are compensated by actuators in control. On the other hand, for unmatched uncertainties with their projection in unmatched space being not zero, based on a (vector) relative degree condition, additive functions are designed to compensate for the uncertainties from output channels in presence of actuator faults. The developed fault-tolerant control schemes are applied to two aircraft systems to demonstrate the efficiency of the proposed approaches

Unitary limit and quantum interference effect in disordered two-dimensional crystals with nearly half-filled bands

Based on the self-consistent $T$-matrix approximation, the quantum interference (QI) effect is studied with the diagrammatic technique in weakly-disordered two-dimensional crystals with nearly half-filled bands. In addition to the usual 0-mode cooperon and diffuson, there exist $\pi$-mode cooperon and diffuson in the unitary limit due to the particle-hole symmetry. The diffusive $\pi$-modes are gapped by the deviation from the exactly-nested Fermi surface. The conductivity diagrams with the gapped $\pi$-mode cooperon or diffuson are found to give rise to unconventional features of the QI effect. Besides the inelastic scattering, the thermal fluctuation is shown to be also an important dephasing mechanism in the QI processes related with the diffusive $\pi$-modes. In the proximity of the nesting case, a power-law anti-localization effect appears due to the $\pi$-mode diffuson. For large deviation from the nested Fermi surface, this anti-localization effect is suppressed, and the conductivity remains to have the usual logarithmic weak-localization correction contributed by the 0-mode cooperon. As a result, the dc conductivity in the unitary limit becomes a non-monotonic function of the temperature or the sample size, which is quite different from the prediction of the usual weak-localization theory.Comment: 21 pages, 4 figure

Integrating sequence and array data to create an improved 1000 Genomes Project haplotype reference panel

A major use of the 1000 Genomes Project (1000GP) data is genotype imputation in genome-wide association studies (GWAS). Here we develop a method to estimate haplotypes from low-coverage sequencing data that can take advantage of single-nucleotide polymorphism (SNP) microarray genotypes on the same samples. First the SNP array data are phased to build a backbone (or 'scaffold') of haplotypes across each chromosome. We then phase the sequence data 'onto' this haplotype scaffold. This approach can take advantage of relatedness between sequenced and non-sequenced samples to improve accuracy. We use this method to create a new 1000GP haplotype reference set for use by the human genetic community. Using a set of validation genotypes at SNP and bi-allelic indels we show that these haplotypes have lower genotype discordance and improved imputation performance into downstream GWAS samples, especially at low-frequency variants. © 2014 Macmillan Publishers Limited. All rights reserved

Towards genomic and proteomic studies of protein phosphorylation in plant-pathogen interactions

Phosphorylation is an effective method of post-translational protein modification but understanding its significance is hindered by its biological complexity. Many protein kinases and phosphatases have been identified that connect signal perception mechanisms to plant defence responses. Recent studies of mitogen-activated protein kinases, calcium-dependent protein kinases and other kinases and phosphatases have revealed some important mechanisms, but have also raised new questions. The regulation of any phosphorylation pathway is complex and dynamic. There are many protein kinases and phosphatases in the plant genome, which makes it hard to delineate the phosphorylation machinery fully. Genomics and proteomics have already identified new components and will continue to influence the study of phosphorylation profoundly in plant-pathogen interactions

Guided deletion and mutagenesis analysis identified a tMEK2-responsive region in tomato lepr1b1 promoter

TMEK2MUT is a constitutively activated form of tMEK2, a mitogen-activated protein kinase kinase (MAPKK). Overexpression of tMEK2MUT in tomato results in transcriptionally upregulated expression of PR1b1, a pathogenesis-related (PR) protein. To identify the responsive regions in the lepr1b1 promoter, Arabidopsis homologues to known mammalian and yeast transcription factors (TFs) downstream of MAPKs in MAPK pathways were identified through database searches. Binding sites in the lepr1b1 promoter for these homologous TFs were analyzed using Transfac, a TF database, and MatInspector program. The analysis indicated that the promoter contains potential binding sites for two TFs, Elf1 and AP1. A protoplast transient expression system was used in deletion and mutagenesis analysis. Mutation of the Elf1 binding site blocked activation of the LePR1b1 gene by tMEK2MUT. Mutation of the AP1 binding sites and of the GCC or W box, two regulatory elements found in some defense-related genes, had no effect on the expression of PR1b1. Our findings suggest that Elf1 is a downstream TF from MAPKK tMEK2 and is involved in mediating LePR1b1 gene expression in defense-related MAPK pathways