High-Energy Gamma-Rays from GRB X-ray Flares

The recent detection of X-ray flares during the afterglow phase of gamma-ray bursts (GRBs) suggests an inner-engine origin, at radii inside the forward shock. There must be inverse Compton (IC) emission arising from such flare photons scattered by forward shock afterglow electrons when they are passing through the forward shock. We find that this IC emission produces high energy gamma-ray flares, which may be detected by AGILE, GLAST and ground-based TeV telescopes. The anisotropic IC scattering between flare photons and forward shock electrons does not affect the total IC component intensity, but cause a time delay of the IC component peak relative to the flare peak. The anisotropic scattering effect may also weaken, to some extent, the suppression effect of the afterglow intensity induced by the enhanced electron cooling due to flare photons. We speculate that this IC component may already have been detected by EGRET from a very strong burst--GRB940217. Future observations by GLAST may help to distinguish whether X-ray flares originate from late central engine activity or from external shocks.Comment: 4 pages, Contributed talk presented at "The First GLAST Symposium", Feb.5-8 2007, Stanford Universit

On the magnetization of gamma-ray burst blast waves

The origin of magnetic fields that permeate the blast waves of gamma-ray bursts (GRBs) is a long-standing problem. The present paper argues that in four GRBs revealing extended emission at >100 MeV, with follow-up in the radio, optical and X-ray domains at later times, this magnetization can be described as the partial decay of the micro-turbulence that is generated in the shock precursor. Assuming that the bulk of the extended emission >100 MeV can be interpreted as synchrotron emission of shock accelerated electrons, we model the multi-wavelength light curves of GRB 090902B, GRB 090323, GRB 090328 and GRB 110731A, using a simplified then a full synchrotron calculation with power-law-decaying microturbulence \epsilon_B \propto t^{\alpha_t} (t denotes the time since injection through the shock, in the comoving blast frame). We find that these models point to a consistent value of the decay exponent -0.5 < \alpha_t < -0.4.Comment: 8 pages, 4 figures - discussion added, conclusions unchanged - version to appear in MNRA

GeV-TeV and X-ray flares from gamma-ray bursts

The recent detection of delayed X-ray flares during the afterglow phase of gamma-ray bursts (GRBs) suggests an inner-engine origin, at radii inside the deceleration radius characterizing the beginning of the forward shock afterglow emission. Given the observed temporal overlapping between the flares and afterglows, there must be inverse Compton (IC) emission arising from such flare photons scattered by forward shock afterglow electrons. We find that this IC emission produces GeV-TeV flares, which may be detected by GLAST and ground-based TeV telescopes. We speculate that this kind of emission may already have been detected by EGRET from a very strong burst--GRB940217. The enhanced cooling of the forward shock electrons by the X-ray flare photons may suppress the synchrotron emission of the afterglows during the flare period. The detection of GeV-TeV flares combined with low energy observations may help to constrain the poorly known magnetic field in afterglow shocks. We also consider the self-IC emission in the context of internal-shock and external-shock models for X-ray flares. The emission above GeV from internal shocks is low, while the external shock model can also produce GeV-TeV flares, but with a different temporal behavior from that caused by IC scattering of flare photons by afterglow electrons. This suggests a useful approach for distinguishing whether X-ray flares originate from late central engine activity or from external shocks.Comment: slightly shortened version, accepted for publication in ApJ Letters, 4 emulateapj pages, no figure

Jet-cloud/star interaction as an interpretation of neutrino outburst from the blazar TXS 0506+056

Recently, a high-energy neutrino event IceCube-170922A in the spatial and temporal coincidence with the flaring gamma-ray blazar TXS 0506+056 was reported. A neutrino outburst between September 2014 and March 2015 was discovered in the same direction by a further investigation of $9.5$ years of IceCube data, while the blazar is in a quiescent state during the outburst with a gamma-ray flux only about one-fifth of the neutrino flux. In this letter, we propose the neutrino outburst originates from the interaction between a relativistic jet and a dense gas cloud which may be formed via the tidally disrupted envelope of a red giant being blown by the impact of the jet. Gamma-ray photons and electron/positron pairs that are produced correspondingly will induce electromagnetic cascades. Comptonization of the cascade emission inside the cloud forms an X-ray photon field with Wien distribution. GeV flux is suppressed due to the absorption by the Comptonized photon field and, as a result, a hard spectrum above 10 GeV is formed. The gamma-ray spectrum predicted in our model is consistent with the Fermi-LAT data of TXS 0506+056.Comment: 6 pages, 3 figure

The standard cohomology of regular Courant algebroids

For any regular Courant algebroid $E$ over a smooth manifold $M$ with characteristic distribution $F$ and ample Lie algebroid $A_E$, we prove that there exists a canonical homological vector field on the graded manifold $A_E[1] \oplus (TM/F)^\ast[2]$ such that the associated dg manifold $\mathcal{M}_E$, which we call the minimal model of the Courant algebroid $E$, encodes all cohomological data of $E$. Thereby, the standard cohomology $H^\bullet_{\operatorname{st}}(E)$ of $E$ can be identified with the cohomology $H^\bullet(\mathcal{M}_E)$ of the function space on $\mathcal{M}_E$. To compute it, we find a natural transgression map [d_T] \colon H^{\bullet}_{\operatorname{CE}}\big(A_E; S^{\diamond}(TM/F[-2])\big) \to H^{\bullet+3}_{\CE}\big(A_E; S^{\diamond-1}(TM/F[-2])\big) from which we construct a spectral sequence which converges to $H^\bullet_{\operatorname{st}}(E)$. Moreover, we give applications to generalized exact Courant algebroids and those arising from regular Lie algebroids .Comment: 41 pages; the main body is rewritten; typos remove

The geometric constraints on Filippov algebroids

Filippov n-algebroids are introduced by Grabowski and Marmo as a natural generalization of Lie algebroids. In this note, we characterize Filippov n-algebroid structures by considering certain multi-input connections, which we call Filippov connections, on the underlying vector bundle. Through this approach, we are able to express the n-ary bracket of any Filippov n-algebroid using a torsion-free type formula. Additionally, we transform the generalized Jacobi identity of the Filippov n-algebroid into the Bianchi-Filippov identity. Furthermore, in the case of rank n vector bundles, we provide a characterization of linear Nambu-Poisson structures using Filippov connections

Spinal Microglial Motility is Independent of Neuronal Activity and Plasticity in Adult Mice

Microglia are the resident macrophages in the central nervous system. In the spinal cord dorsal horn, microglia stay in resting condition during physiological sensory processing, and are activated under pathological conditions such as peripheral nerve injury. In cases such as this, the nearby resting microglia increase their motility and accumulate at the site of injury. However, direct evidence to support that nerve activity can enhance the motility of microglia has not yet to be reported. In this study we investigated whether the activation of spinal microglia under in vivo nerve injury may be mimicked by neuronal activity in the spinal cord slice preparation. We found that local application of spinal excitatory neurotransmitters, such as glutamate and substance P did not cause any change in the motility of microglial cells in the spinal cord dorsal horn. The motility of microglial cells is unlikely modulated by other transmitters, neuromodulators and chemokines, because similar applications such as GABA, serotonin, noradrenaline, carbachol, fractalkine or interleukin did not produce any obvious effect. Furthermore, low or high frequency stimulation of spinal dorsal root fibers at noxious intensities failed to cause any enhanced extension or retraction of the microglia processes. By contrast, focal application of ATP triggered rapid and robust activation of microglial cells in the spinal dorsal horn. Our results provide the first evidence that the activation of microglia in the spinal cord after nerve injury is unlikely due solely to neuronal activity, non-neuronal factors are likely responsible for the activation of nerve injury-related microglial cells in the spinal dorsal horn