247 research outputs found

    How and what kind of cities benefit from the development of digital inclusive finance? Evidence from the upgrading of export in Chinese cities

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    Employing Chinese customs data and the Peking University Digital Financial Inclusion Index of China, this paper studies the impact of China’s digital finance development on the upgrading of export at the city level and further explores the heterogeneity across cities and the mechanisms through which digital finance influences export upgrading. Benchmark results suggest that digital inclusive finance can significantly promote the upgrading of export. The heterogeneity analysis shows that cities with a smaller size, lower wage, higher human capital level, and better location advantage experience greater facilitating effects of digital inclusive finance on promoting export upgrading. It suggests that, compared with ‘icing on the cake’, the digital inclusive finance plays a better role in ‘offering fuel in snowy weather’, whereas full exertion of the inclusiveness of digital finance requires higher human capital and location advantage. Further mechanism analysis shows that innovation effect and market effect are the main channels where digital inclusive finance promotes the upgrading of a city’s export

    Spin Squeezing through Collective Spin-Spin Interactions

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    Spin squeezing provides crucial quantum resource for quantum metrology and quantum information science. Here we propose that one axis-twisted (OAT) spin squeezing can be generated from free evolution under a general coupled-spin model with collective spin-spin interactions. We further propose pulse schemes to recover squeezing from parameter imperfections, and reach the extreme squeezing with Heisenberg-limited measurement precision scaling as 1/N1/N for NN particles. This work provides a feasible method for generating extreme spin squeezing

    catena-Poly[[bis­(1H-benzimidazole-κN 3)palladium(II)]-μ-benzene-1,4-dicarboxyl­ato-κ2 O 1:O 4]

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    In the title compound, [Pd(C8H4O4)(C7H6N2)2]n, the Pd atom is tetra­coordinated by two carboxyl­ate O atoms from two benzene-1,4-dicarboxyl­ate (bdc) dianions and two N atoms from two benzimidazole ligands, resulting in a slightly distorted tetra­hedral PdO2N2 geometry. The bdc ligand acts as a bridge, linking the Pd atoms into a chain. Inter-chain N—H⋯O hydrogen bonds help to stabilize the crystal structure

    MicroRNA-33b inhibits liver cancer cell proliferation, migration and invasion via down-regulation of Fli-1 and MMP-2 protein expressions

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    Purpose: To study the influence of microRNA-33b (miR-33b) on liver cancer cell proliferation, migration and invasiveness, and the mechanism involved. Methods: MicroRNA-33b or Fli-1 overexpression plasmid was transfected into liver cancer (SMMC7721) cells. Cell proliferation, migration, and invasiveness were determined using cell counting kit 8 (CCK-8), scratch test, and Transwell invasion assay, respectively. The amounts of miR-33b and Fli-1 in liver cancer tissues,  paracancerous normal tissues, and miR-33b overexpression and control groups were measured using qRT-PCR, while protein concentration of matrix metalloproteinase 2 (MMP-2) was assayed using Western blotting. Results: Fli-1 protein was markedly upregulated in liver cancerous cells, relative to paracancerous normal tissues (p < 0.05). MicroRNA-33b protein expression was also significantly upregulated in miR33b overexpression group, but the corresponding Fli-1 expression was downregulated in miR-33b overexpression group, relative to control (p < 0.05). MicroRNA-33b overexpression significantly and time-dependently inhibited SMMC7721 cell proliferation and migration, but it reduced the degree of apoptosis (p < 0.05). Liver cancer (SMMC7721) cells in miR-33b overexpression group were less invasive than the control group (p < 0.05). Similarly, miR-33b overexpression significantly downregulated MMP-2 protein expression in SMMC7721cells (p < 0.05). Conclusion: Overexpression of miR-33b suppresses the proliferation, migratory and invasive potential of hepatic cancer cells via down-regulation of Fli-1 and MMP-2 protein expression. This finding may be useful in the identification of new liver cancer drugs

    μ-Oxido-bis­({4,4′-dibromo-2,2′-ethane-1,2-diylbis(nitrilo­methyl­idyne)]diphenolato}iron(III))

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    In the title compound, [Fe2(C16H12Br2N2O2)2O], the complete mol­ecule is generated by twofold symmetry, with the bridging O atom, which links the iron centres, lying on the roatation rotation axis. The Fe(III) ion is chelated by the N,N,O,O-tetra­dentate Schiff base dianion, resulting in an FeN2O3 square-based pyramid, with the two N atoms in the basal plane

    μ-Oxido-bis­{chlorido[tris­(2-pyridylmethyl)amine]manganese(III)} bis­(hexa­fluorido­phosphate)

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    In the title compound, [Mn2O(C18H18ClN4)2](PF6)2, the Mn atom is chelated by a tetra­dentate ligand via four N atoms, and further bonded to one chloride ion and one bridging oxide, to give a centrosymmetric cation and distorted octa­hedral coordination geometry

    Dynamic Analysis of a Predator-Prey (Pest) Model with Disease in Prey and Involving an Impulsive Control Strategy

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    The dynamic behaviors of a predator-prey (pest) model with disease in prey and involving an impulsive control strategy to release infected prey at fixed times are investigated for the purpose of integrated pest management. Mathematical theoretical works have been pursuing the investigation of the local asymptotical stability and global attractivity for the semitrivial periodic solution and population persistent, which depicts the threshold expression of some critical parameters for carrying out integrated pest management. Numerical analysis indicates that the impulsive control strategy has a strong effect on the dynamical complexity and population persistent using bifurcation diagrams and power spectra diagrams. These results show that if the release amount of infective prey can satisfy some critical conditions, then all biological populations will coexist. All these results are expected to be of use in the study of the dynamic complexity of ecosystems
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