6 research outputs found
New insights into the genetic etiology of Alzheimer's disease and related dementias
- Author
- Aaltonen L.
- Aaltonen V.
- Aarsland Dag
- Aavikko M.
- Abalos M.S.
- Abdelnour Carla
- Abner E.
- Abraham R.
- Adams H.
- Adams P.M.
- Adarmes-GĂłmez A.
- Adarmes-GĂłmez A.D.
- Aguilera N.
- Aguilera N.
- Aguirre A.
- Ahmad S.
- Akinyemi R.O.
- Al-Chalabi A.
- AlarcĂłn-MartĂn Emilio
- Albert M.S.
- Albin R.L.
- Alcolea Daniel
- Alegret Montserrat
- Ali M.
- Allen M.
- Allende I.R.
- Alonso M.D.
- Alonso M.D.
- Alvarez I.
- Alvarez L.
- Amer-Ferrer G.
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- Amouyel Philippe
- Anastasiou A.
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- Publication venue
- Publication date
- 01/01/2022
- Field of study
Get PDFCharacterization of the genetic landscape of Alzheimer's disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/'proxy' AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE Δ4 allele
Reduction of a Starling Population at a Turkey Farm
- Publication venue
- 'Oxford University Press (OUP)'
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- Field of study
No full textProblems with Constructing Tests to Accept the Null Hypothesis
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
No full textFutility designs bhaveeen proposed and used by constructing classical (non-Bayesian) hypothesis tests such that the decision of therapeutic interest is to accept the null hypothesis.A consequence is that the probability of accepting (failing to reject) the null when the null is false is unknown. Reversal of the conventional null and alternative hypotheses is not required either to demonstrate futility/nonsuperiority, or to align type I and II errors with their consequences. Conventional methods to test whether the response to the investigational agent is superior to a comparative control (superiority trial) are preferable and in the case that the null hypothesis is rejected, the associated type I error is known
Replicability, Confidence, and Priors
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- Publication venue
- 'Wiley'
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No full textStatistical aspects of interpreting DNA profiling in legal cases
- Author
- Aitken C.G.G.
- Aitken C.G.G.
- Balding D.J.
- Balding D.J.
- Balding D.J.
- Brenner C.H.
- Buckleton J.S.
- Dawid A.P.
- Dawid A.P.
- DNA Advisory Board DAB
- Essen-Moller E.
- Evett I.W.
- Evett I.W.
- Evett I.W.
- Evett I.W.
- Gastwirth J.L.
- Geisser S.
- Jeffreys A.J.
- Jeffreys A.J.
- Lewontin R.C.
- Lewontin R.C.
- National Research Council
- National Research Council
- Perlin M.W.
- Robertson B.
- Roeder K.
- Royall R.
- Sjerps M.
- Stockmarr A.
- Thompson W.C.
- Weir B.S.
- Weir B.S.
- Weir B.S.
- Weir B.S.
- Publication venue
- Publication date
- Field of study
No full textNew insights into the genetic etiology of Alzheimerâs disease and related dementias
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
No full textCharacterization of the genetic landscape of Alzheimerâs disease (AD) and related dementias (ADD) provides a unique opportunity for a better understanding of the associated pathophysiological processes. We performed a two-stage genome-wide association study totaling 111,326 clinically diagnosed/âproxyâ AD cases and 677,663 controls. We found 75 risk loci, of which 42 were new at the time of analysis. Pathway enrichment analyses confirmed the involvement of amyloid/tau pathways and highlighted microglia implication. Gene prioritization in the new loci identified 31 genes that were suggestive of new genetically associated processes, including the tumor necrosis factor alpha pathway through the linear ubiquitin chain assembly complex. We also built a new genetic risk score associated with the risk of future AD/dementia or progression from mild cognitive impairment to AD/dementia. The improvement in prediction led to a 1.6- to 1.9-fold increase in AD risk from the lowest to the highest decile, in addition to effects of age and the APOE Δ4 allele. © 2022, The Author(s)
