Derivation of the action and symmetries of the q-deformed AdS5×S5 superstring

This article is distributed under the terms of the Creative Commons Attribution License (CC-BY 4.0), which permits any use, distribution and reproduction in any medium, provided the original author(s) and source are credited.We recently proposed an integrable q-deformation of the AdS5 × S 5 superstring action. Here we give details on the hamiltonian origin and construction of this deformation. The procedure is a generalization of the one previously developed for deforming principal chiral and symmetric space σ-modelsPeer reviewedFinal Published versio

First exclusive measurements of the K^-pp state - populated in the pp to K^+ Lambda p reaction at 2.85 GeV

We have analyzed data of the DISTO experiment on the exclusive pp to K^+ Lambda p process at T_p = 2.85$GeV to search for a K^-pp (= X) nuclear bound state to be formed in the pp to K^+ + X reaction. The deviation spectra of the K^+ missing-mass MM (K^+) and Lambda p invariant-mass M(Lambda-p) with selection of large-angle proton emission revealed a structure with M_X = 2265 +- 2 MeV/c2 and Gamma_X = 118 +- 8$ MeV.Comment: 6 pages, 4 figues, Proceedings of EXA0

The Role of Host Genetics in Susceptibility to Influenza: A Systematic Review

Background: The World Health Organization has identified studies of the role of host genetics on susceptibility to severe influenza as a priority. A systematic review was conducted to summarize the current state of evidence on the role of host genetics in susceptibility to influenza (PROSPERO registration number: CRD42011001380). Methods and Findings: PubMed, Web of Science, the Cochrane Library, and OpenSIGLE were searched using a pre-defined strategy for all entries up to the date of the search. Two reviewers independently screened the title and abstract of 1,371 unique articles, and 72 full text publications were selected for inclusion. Mouse models clearly demonstrate that host genetics plays a critical role in susceptibility to a range of human and avian influenza viruses. The Mx genes encoding interferon inducible proteins are the best studied but their relevance to susceptibility in humans is unknown. Although the MxA gene should be considered a candidate gene for further study in humans, over 100 other candidate genes have been proposed. There are however no data associating any of these candidate genes to susceptibility in humans, with the only published study in humans being under-powered. One genealogy study presents moderate evidence of a heritable component to the risk of influenza-associated death, and while the marked familial aggregation of H5N1 cases is suggestive of host genetic factors, this remains unproven. Conclusion: The fundamental question ‘‘Is susceptibility to severe influenza in humans heritable?’ ’ remains unanswered. No