Metabolism and conidiation in Neurospora crassa

Metabolism and conidiation in Neurospora crass

The hexosemonophosphate shunt as an alternate metabolic pathway for conidial differentiation in Neurospora

The hexosemonophosphate shunt as an alternate metabolic pathway for conidial differentiation in Neurospor

Further studies on the metabolic control of conidiation of N.crassa

Metabolic control of conidiatio

Conidiation antigen and malate dehydrogenase isoenzyme activities

Conidiation antigen and malate dehydrogenase isoenzyme activitie

Isocitratase studies in Neurospora

Isocitratase studies in Neurospor

Improved techniques for study of caratenoid intermediates

Improved techniques for study of caratenoid intermediate

Preliminary studies on pigmentation and ultrastructure of microconidia of Neurospora crassa

Pigmentation and ultrastructure of microconidi

Construction of two Neurospora crassa Lambda ZapII-cDNA libraries

In the presence of the anti-microtubule agent benomyl, N. crassa synthesizes and secretes a major 60 kDa protein in the culture medium. This protein is absent in untreated cells (Rossier et al. 1989. Eur. J. Cell Biol. 50:333-339). To clone the gene coding for the 60 kDa protein and to unravel its function, we have constructed a cDNA library from benomyl-treated cells ( benomyl cDNA library). A second cDNA library was constructed from untreated cells in order to clone genes coding for cytoskeletal proteins ( standard cDNA library)

New mutants of Neurospora crassa highly resistant to the microtubule inhibitor benomyl

Mutants of N. crassa highly resistant to benomyl with respect to hyphal growth were obtained by mutagenizing wild type strain St. Lawrence 74A or mutant Bml 511(r) which is moderately resistant to the fungicide. One of these, strain E1-91 that has a mutation mapping at the Bml (beta-tubulin) locus, showed temporary sensitivity to the benzimidazole