27 research outputs found
Hypernova Nucleosynthesis and Galactic Chemical Evolution
We study nucleosynthesis in 'hypernovae', i.e., supernovae with very large
explosion energies ( \gsim 10^{52} ergs) for both spherical and aspherical
explosions. The hypernova yields compared to those of ordinary core-collapse
supernovae show the following characteristics: 1) Complete Si-burning takes
place in more extended region, so that the mass ratio between the complete and
incomplete Si burning regions is generally larger in hypernovae than normal
supernovae. As a result, higher energy explosions tend to produce larger [(Zn,
Co)/Fe], small [(Mn, Cr)/Fe], and larger [Fe/O], which could explain the trend
observed in very metal-poor stars. 2) Si-burning takes place in lower density
regions, so that the effects of -rich freezeout is enhanced. Thus
Ca, Ti, and Zn are produced more abundantly than in normal
supernovae. The large [(Ti, Zn)/Fe] ratios observed in very metal poor stars
strongly suggest a significant contribution of hypernovae. 3) Oxygen burning
also takes place in more extended regions for the larger explosion energy. Then
a larger amount of Si, S, Ar, and Ca ("Si") are synthesized, which makes the
"Si"/O ratio larger. The abundance pattern of the starburst galaxy M82 may be
attributed to hypernova explosions. Asphericity in the explosions strengthens
the nucleosynthesis properties of hypernovae except for "Si"/O. We thus suggest
that hypernovae make important contribution to the early Galactic (and cosmic)
chemical evolution.Comment: To be published in "The Influence of Binaries on Stellar Population
Studies", ed. D. Vanbeveren (Kluwer), 200
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The first back-side illuminated types of Kyoto's X-ray astronomy SOIPIX
We have been developing Kyoto's X-ray astronomy SOI pixel sensors, called âXRPIXâ, aiming to extend the frontiers of X-ray astronomy with the wide-band imaging spectroscopy in the 0.5â40 keV band. A dead layer on the X-ray incident surface should ideally be as thin as possible to achieve a high sensitivity below 1 keV, and the depletion layer is required to be thick enough to detect 40 keV X-rays. Thus, we have started developing fully-depleted back-side illuminated (BI) types of XRPIXs. This paper reports on our first two BI devices and their X-ray evaluation (2.6â12 keV). The device named âXRPIX2b-FZ-LAâ successfully reaches a full depletion with a thickness of 500 ÎŒm. On the other hand, it has a dead layer with a thickness of 1.1â1.5 ÎŒm and struggles to achieve the requirement of 1.0 ÎŒm. The other device named âXRPIX2b-CZ-PZâ, which is applied with a thin Si sensor-layer and an improved back-side process, is found to satisfy the requirement with its thickness of 0.9â1.0 ÎŒm, including Al optical blocking filter of 0.2 ÎŒm, although the Si sensor-layer is rather thin with 62 ÎŒm. We also describe in this paper the X-ray calibration system that we have built for the X-ray evaluation of XRPIXs
Age Is a Determinant of Leukocyte Infiltration and Loss of Cortical Volume after Traumatic Brain Injury
There is increasing evidence that the inflammatory response differs in the injured developing brain as compared to the adult brain. Here we compared cerebral blood flow and profiled the inflammatory response in mice that had been subjected to traumatic brain injury (TBI) at postnatal day (P)21 or at adulthood. Relative blood flow, determined by laser Doppler, revealed a 30% decrease in flow immediately after injury followed by prominent hyperemia between 7 and 35 days after injury in both age groups. The animals were euthanized at 1â35 days after injury and the brains prepared for the immunolocalization and quantification of CD45-, GR-1-, CD4- and CD8-positive (+) cells. On average, the number of CD45+ leukocytes in the cortex was significantly higher in the P21 as compared to the adult group. A similar trend was seen for GR-1+ granulocytes, whereas no age-related differences were noted for CD4+ and CD8+ cells. While CD45+ and GR-1+ cells in the P21 group remained elevated, relative to shams, over the first 2 weeks after injury, the adult group showed a time course limited to the first 3 days after injury. The loss of ipsilateral cortical volumes at 2 weeks after injury was significantly greater in the adult relative to the P21 group. While the adult group showed no further change in cortical volumes, there was a significant loss of cortical volumes between 2 and 5 weeks after injury in the P21 group, reaching values similar to that of the adult group by 5 weeks after injury. Together, these findings demonstrate age-dependent temporal patterns of leukocyte infiltration and loss of cortical volume after TBI