Performance evaluation of real-time indoor positioning with active RFID and ZigBee-based WSN systems

This study measures the accuracy of two radio signal-based positioning methods to track targets in an indoor environment using radio frequency identification (RFID) and a wireless sensor network (WSN), the advantages and disadvantages of which are compared, analyzed, and discussed. We propose a specific configuration to perform measurements in a real environment using various positioning algorithms. The experimental results show that the number and the quality of the reference tags influence the accuracy of positioning when using RFID devices. Although WSN devices require fewer sensors, the unit price of sensors is comparatively high. In addition, adjusting the antenna angle achieves a stronger received signal strength indicator variation when using RFID devices. For WSN devices, the key is to avoid interference between sensors because it causes large errors when estimating the distance between 2 points. The proposed positioning methods are suitable for patrol systems, and can be employed to obtain security service personnel location data and confirm their patrol routes. This study adopts active RFID technology and nano real-time positioning WSN technology to test the indoor positioning accuracy of these technologies and related system construction issues. The findings provide a reference for future research on indoor positioning method selection

Development of a microarray for simultaneous detection and differentiation of different tospoviruses that are serologically related to Tomato spotted wilt virus

Nucleotide (above diagonal) and amino acid (below diagonal) identities (%) of the N genes among the members of TSWV serogroup. (DOCX 68 kb

A hybrid switched reactive-based visual servo control of 5-DOF robot manipulators for pick-and-place tasks

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The nucleolar protein NIFK promotes cancer progression via CK1α/β-catenin in metastasis and Ki-67-dependent cell proliferation.

Nucleolar protein interacting with the FHA domain of pKi-67 (NIFK) is a Ki-67-interacting protein. However, its precise function in cancer remains largely uninvestigated. Here we show the clinical significance and metastatic mechanism of NIFK in lung cancer. NIFK expression is clinically associated with poor prognosis and metastasis. Furthermore, NIFK enhances Ki-67-dependent proliferation, and promotes migration, invasion in vitro and metastasis in vivo via downregulation of casein kinase 1α (CK1α), a suppressor of pro-metastatic TCF4/β-catenin signaling. Inversely, CK1α is upregulated upon NIFK knockdown. The silencing of CK1α expression in NIFK-silenced cells restores TCF4/β-catenin transcriptional activity, cell migration, and metastasis. Furthermore, RUNX1 is identified as a transcription factor of CSNK1A1 (CK1α) that is negatively regulated by NIFK. Our results demonstrate the prognostic value of NIFK, and suggest that NIFK is required for lung cancer progression via the RUNX1-dependent CK1α repression, which activates TCF4/β-catenin signaling in metastasis and the Ki-67-dependent regulation in cell proliferation