Genome-wide cis-eQTL results (SNPs with P<0.05)

Genome-wide cis-eQTL results (SNPs with P<0.05

Association between smoking and mortality from IHD and stroke.

a<p>Adjusted for baseline age (years), body mass index, and educational attainment.</p>b<p>Non-cigarette/bidi smokers as the reference group.</p>c<p>Categories in ever smokers were based on meaningful cut points for easy interpretation.</p>d<p>Non-hookah smokers as the reference group.</p

Mean urinary creatinine-adjusted As levels for participants with an unsafe well at baseline () and for those with a safe well at baseline ()

<p><b>Copyright information:</b></p><p>Taken from "Reduction in Urinary Arsenic Levels in Response to Arsenic Mitigation Efforts in Araihazar, Bangladesh"</p><p></p><p>Environmental Health Perspectives 2007;115(6):917-923.</p><p>Published online 5 Feb 2007</p><p>PMCID:PMC1892113.</p><p>This is an Open Access article: verbatim copying and redistribution of this article are permitted in all media for any purpose, provided this notice is preserved along with the article's original DOI</p> Additional adjustments were made for age, sex, and BMI. Values above the bars are average baseline well As concentration and number

Survival curves for overall survival by never and ever cigarette/bidi smoking.

<p>(<b>A</b>) Overall survival among men. (<b>B</b>) Overall survival among women. Cox proportional hazards regression analyses were adjusted for baseline age (years), body mass index, and educational attainment (years).</p

Association between smoking and mortality from all-cause, cancer, and cardiovascular diseases.

a<p>Adjusted for baseline age (years), body mass index, and educational attainment.</p>b<p>Non-cigarette/bidi smokers as the reference group.</p>c<p>Categories in ever smokers were based on meaningful cut points for easy interpretation.</p>d<p>Non-hookah smokers as the reference group.</p

Genome-wide SNP association signals for number of pregnancies (a) number of children (b) and ≥6 vs. ≤1 children (c) for overall study population (N = 1,686).

<p>Genome-wide SNP association signals for number of pregnancies (a) number of children (b) and ≥6 vs. ≤1 children (c) for overall study population (N = 1,686).</p

Prevalence of smoking and age at starting smoking.

<p>(<b>A</b>) Prevalence of cigarette/bidi smoking and hookah smoking by age groups (17–29, 30–39, 40–49, and 50+) among men and women. (<b>B</b>) Mean age at starting cigarette/bidi smoking by age groups among men and women.</p

Prevalence of ever cigarette/bidi smoking by baseline characteristics.^a

a<p>Data were missing on cigarette/bidis smoking for 8 subjects; on education for 11 subjects; on religion for 1 subject; on body mass index for 281 subjects; on hookah smoking for 8295 subjects; on betel quid chewing for 34 subjects; on systolic blood pressure for 260 subjects; on diastolic blood pressure for 268 subjects; and on history of diabetes for 321 subjects.</p>b<p><i>P</i>-value from the chi-square test or <i>t</i>-test.</p

SNPs with number of pregnancies number of children (linear) or extreme number children (≥6 compared to ≤1) in Bangladeshi women (p<1X10–5) for entire study population and excluding hormonal contraceptive users.

<p>SNPs with number of pregnancies number of children (linear) or extreme number children (≥6 compared to ≤1) in Bangladeshi women (p<1X10–5) for entire study population and excluding hormonal contraceptive users.</p

Association of age at starting cigarette/bidi smoking and time since quitting smoking with mortality from all-cause, cancer, and IHD among men.

a<p>Adjusted for baseline age (years), BMI, and educational attainment.</p>b<p>Non-cigarette/bidi smokers as the reference group.</p>c<p>Among current smokers.</p>d<p>Among past smokers.</p