A short note on spin pumping theory with Landau-Lifshitz-Gilbert equation under quantum fluctuation; necessity for quantization of localized spin

We would like to point out the blind spots of the approach combining the spin pumping theory proposed by Tserkovnyak et al. with the Landau-Lifshitz-Gilbert equation; this method has been widely used for interpreting vast experimental results. The essence of the spin pumping effect is the quantum fluctuation. Thus, localized spin degrees of freedom should be quantized, i.e. be treated as magnons not as classical variables. Consequently, the precessing ferromagnet can be regarded as a magnon battery. This point of view will be useful for further progress of spintronics.Comment: 10pages, 1 figure. This article is closely related to the work by K. N.; arXiv:1201.194

Measurement of minority carrier lifetime, mobility and diffusion length in heavily doped silicon

Carrier transport and recombination parameters in heavily doped silicon were examined. Data were presented for carrier diffusivity in both p- and n-type heavily doped silicon covering a broad range of doping concentrations from 10 to the 15th power to 10 to the 20th power atoms/cu cm. One of the highlights of the results showed that minority carrier diffusivities are higher by a factor of 2 in silicon compared to majority carrier diffusivities

Node synchronization schemes for the Big Viterbi Decoder

The Big Viterbi Decoder (BVD), currently under development for the DSN, includes three separate algorithms to acquire and maintain node and frame synchronization. The first measures the number of decoded bits between two consecutive renormalization operations (renorm rate), the second detects the presence of the frame marker in the decoded bit stream (bit correlation), while the third searches for an encoded version of the frame marker in the encoded input stream (symbol correlation). A detailed account of the operation is given, as well as performance comparison, of the three methods

Dynamics and Decay of Heavy-Light Hadrons

Recent signals for narrow hadrons containing heavy and light flavours are compared with quark model predictions for spectroscopy, strong decays, and radiative transitions. In particular, the production and identification of excited charmed and cs states are examined with emphasis on elucidating the nature of $0^+$ and $1^+$ states. Roughly 200 strong decay amplitudes of $D$ and $D_s$ states up to 3.3 GeV are presented. Applications include determining flavour content in $\eta$ mesons and the mixing angle in $P$ and $D$ wave states and probes of putative molecular states. We advocate searching for radially excited $D_s^*$ states in B decays.Comment: 19 pages, 7 figures, revtex. A numerical error is corrected. Some strong decay rates have change

Modelling DNA Response to THz Radiation

Collective response of DNA to THz electric fields is studied in a simple pair bond model. We confirm, with some caveats, a previous observation of destabilising DNA breather modes and explore the parameter-dependence of these modes. It is shown that breather modes are eliminated under reasonable physical conditions and that thermal effects are significant.Comment: 6 pages, 3 figures. version to appear in Phys. Rev.

A central role for C1q/TNF-related protein 13 (CTRP13) in modulating food intake and body weight.

C1q/TNF-related protein 13 (CTRP13), a hormone secreted by adipose tissue (adipokines), helps regulate glucose metabolism in peripheral tissues. We previously reported that CTRP13 expression is increased in obese and hyperphagic leptin-deficient mice, suggesting that it may modulate food intake and body weight. CTRP13 is also expressed in the brain, although its role in modulating whole-body energy balance remains unknown. Here, we show that CTRP13 is a novel anorexigenic factor in the mouse brain. Quantitative PCR demonstrated that food restriction downregulates Ctrp13 expression in mouse hypothalamus, while high-fat feeding upregulates expression. Central administration of recombinant CTRP13 suppressed food intake and reduced body weight in mice. Further, CTRP13 and the orexigenic neuropeptide agouti-related protein (AgRP) reciprocally regulate each other's expression in the hypothalamus: central delivery of CTRP13 suppressed Agrp expression, while delivery of AgRP increased Ctrp13 expression. Food restriction alone reduced Ctrp13 and increased orexigenic neuropeptide gene (Npy and Agrp) expression in the hypothalamus; in contrast, when food restriction was coupled to enhanced physical activity in an activity-based anorexia (ABA) mouse model, hypothalamic expression of both Ctrp13 and Agrp were upregulated. Taken together, these results suggest that CTRP13 and AgRP form a hypothalamic feedback loop to modulate food intake and that this neural circuit may be disrupted in an anorexic-like condition