Real-Time Depth-Based Hand Detection and Tracking

This paper illustrates the hand detection and tracking method that operates in real time on depth data. To detect a hand region, we propose the classifier that combines a boosting and a cascade structure. The classifier uses the features of depth-difference at the stage of detection as well as learning. The features of each candidate segment are to be computed by subtracting the averages of depth values of subblocks from the central depth value of the segment. The features are selectively employed according to their discriminating power when constructing the classifier. To predict a hand region in a successive frame, a seed point in the next frame is to be determined. Starting from the seed point, a region growing scheme is applied to obtain a hand region. To determine the central point of a hand, we propose the so-called Depth Adaptive Mean Shift algorithm. DAM-Shift is a variant of CAM-Shift (Bradski, 1998), where the size of the search disk varies according to the depth of a hand. We have evaluated the proposed hand detection and tracking algorithm by comparing it against the existing AdaBoost (Friedman et al., 2000) qualitatively and quantitatively. We have analyzed the tracking accuracy through performance tests in various situations

Distal Pectoralis Major Tears

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/135433/1/jum201332122075.pd

Akt1-Inhibitor of DNA binding2 is essential for growth cone formation and axon growth and promotes central nervous system axon regeneration.

Mechanistic studies of axon growth during development are beneficial to the search for neuron-intrinsic regulators of axon regeneration. Here, we discovered that, in the developing neuron from rat, Akt signaling regulates axon growth and growth cone formation through phosphorylation of serine 14 (S14) on Inhibitor of DNA binding 2 (Id2). This enhances Id2 protein stability by means of escape from proteasomal degradation, and steers its localization to the growth cone, where Id2 interacts with radixin that is critical for growth cone formation. Knockdown of Id2, or abrogation of Id2 phosphorylation at S14, greatly impairs axon growth and the architecture of growth cone. Intriguingly, reinstatement of Akt/Id2 signaling after injury in mouse hippocampal slices redeemed growth promoting ability, leading to obvious axon regeneration. Our results suggest that Akt/Id2 signaling is a key module for growth cone formation and axon growth, and its augmentation plays a potential role in CNS axonal regeneration