Quiver Bundles and Wall Crossing for Chains

Holomorphic chains on a Riemann surface arise naturally as fixed points of the natural C*-action on the moduli space of Higgs bundles. In this paper we associate a new quiver bundle to the Hom-complex of two chains, and prove that stability of the chains implies stability of this new quiver bundle. Our approach uses the Hitchin-Kobayashi correspondence for quiver bundles. Moreover, we use our result to give a new proof of a key lemma on chains (due to \'Alvarez-C\'onsul, Garc\'ia-Prada and Schmitt), which has been important in the study of Higgs bundle moduli; this proof relies on stability and thus avoids the direct use of the chain vortex equations

Nucleophilic activity of a linked bis{guanidine} leading to formation of a dicationic C4N4-heterocycle

The methylene-linked bis{guanidine}, H2C{hpp}2 (hppH = 1,3,4,6,7,8-hexahydro-2H-pyrimido[1,2-a]pyrimidine), displays nucleophilic activity towards organic halides, including the activation of dichloromethane under ambient conditions to afford the heterocyclic dication, [H2C{hpp}2CH2]2+ [Cl]- 2. X-ray crystallography has been used as a probe of the bonding within the eight-membered ring of the dication, and DFT calculations have identified two stable conformations, in agreement with two different species observed in solution

Avoiding Cracks in Nanoparticle Films

A new method utilizing subsequent depositions of thin crack-free nanoparticle layers is demonstrated to avoid the formation of cracks within silica nanoparticle films. Using this method, films can be assembled with thicknesses exceeding the critical cracking values. Explanation of this observed phenomenon is hypothesized to mainly arise from chemical bond formation between neighboring silica nanoparticles. Application of this method for fabricating crack-free functional structures is demonstrated by producing crack-free Bragg reflectors that exhibit structural color

Additional file 7: of Isolation of T cell receptors targeting recurrent neoantigens in hematological malignancies

Engineering of mCALR-expressing target cells. (DOCX 488 kb

Additional file 5: of Isolation of T cell receptors targeting recurrent neoantigens in hematological malignancies

Detection of cancer-testes antigen T cell responses in the healthy donor T cell repertoire. (DOCX 1988 kb

Additional file 8: of Isolation of T cell receptors targeting recurrent neoantigens in hematological malignancies

Engineering of mFBXW7-expressing target cells. (DOCX 409 kb

Additional file 6: of Isolation of T cell receptors targeting recurrent neoantigens in hematological malignancies

mCALR-specific TCR gene rearrangments. (DOCX 1896 kb

Additional file 3: of Isolation of T cell receptors targeting recurrent neoantigens in hematological malignancies

Myeloproliferative neoplasm patient cohort. (DOCX 16 kb

Additional file 1: of Isolation of T cell receptors targeting recurrent neoantigens in hematological malignancies

Supplementary materials and methods. (DOCX 221 kb

Growth curves for SW480 cells (A) HCT-116 cells (B), HCT-15 cells (C), HT-29 (D) were plotted as average cell counts over time after treatment with 100 μM tocopherol at 1, 2, and 3 days

<p><b>Copyright information:</b></p><p>Taken from "Comparative effects of RRR-alpha- and RRR-gamma-tocopherol on proliferation and apoptosis in human colon cancer cell lines"</p><p>BMC Cancer 2006;6():13-13.</p><p>Published online 17 Jan 2006</p><p>PMCID:PMC1379650.</p><p>Copyright © 2006 Campbell et al; licensee BioMed Central Ltd.</p> and Live Cell Counts in CCD-112CoN cells following 100 μM tocopherol treatment at 72 hours (E). Values plotted are averages of three independent trials. Error bars represent standard deviation of the means. Positive controls used included (15 deoxy Δ 12,14 PGJ2, troglitzone and camptothecin). (Data are representative of three independent trials performed in triplicate. *p < 0.05 vs. vehicle at corresponding concentration.