4,563 research outputs found
Adiabatic optical entanglement between electron spins in separate quantum dots
We present an adiabatic approach to the design of entangling quantum
operations with two electron spins localized in separate InAs/GaAs quantum dots
via the Coulomb interaction between optically-excited localized states.
Slowly-varying optical pulses minimize the pulse noise and the relaxation of
the excited states. An analytic "dressed state" solution gives a clear physical
picture of the entangling process, and a numerical solution is used to
investigate the error dynamics. For two vertically-stacked quantum dots we show
that, for a broad range of dot parameters, a two-spin state with concurrence
can be obtained by four optical pulses with durations
ns.Comment: 7 pages, 5 figure
Fast initialization of the spin state of an electron in a quantum dot in the Voigt configuration
We consider the initialization of the spin-state of a single electron trapped
in a self-assembled quantum dot via optical pumping of a trion level. We show
that with a magnetic field applied perpendicular to the growth direction of the
dot, a near-unity fidelity can be obtained in a time equal to a few times the
inverse of the spin-conserving trion relaxation rate. This method is several
orders-of-magnitude faster than with the field aligned parallel, since this
configuration must rely on a slow hole spin-flip mechanism. This increase in
speed does result in a limit on the maximum obtainable fidelity, but we show
that for InAs dots, the error is very small.Comment: 4 pages, 4 figure
Generation of frequency sidebands on single photons with indistinguishability from quantum dots
Generation and manipulation of the quantum state of a single photon is at the
heart of many quantum information protocols. There has been growing interest in
using phase modulators as quantum optics devices that preserve coherence. In
this Letter, we have used an electro-optic phase modulator to shape the state
vector of single photons emitted by a quantum dot to generate new frequency
components (modes) and explicitly demonstrate that the phase modulation process
agrees with the theoretical prediction at a single photon level. Through
two-photon interference measurements we show that for an output consisting of
three modes (the original mode and two sidebands), the indistinguishability of
the mode engineered photon, measured through the secondorder intensity
correlation (g2(0)) is preserved. This work demonstrates a robust means to
generate a photonic qubit or more complex state (e.g., a qutrit) for quantum
communication applications by encoding information in the sidebands without the
loss of coherence
Influence of misonidazole on the incidence of radiation-induced intestinal tumours in mice.
C57BL mice were given local irradiation to 2 cm2 of the lower abdomen in the dose range 16--24 Gy. There were some early deaths, but mice dying between 50--240 days predominantly developed invasive adenocarcinomas of the intestine. When the radiosensitizer misonidazole was given in a single dose shortly before irradiation the proportion of mice developing tumours was higher, but the difference was not statistically significant. However, there was a significant increase in the incidence of multiple tumours, largely attributable to tumours arising in the rectum
Myosin VIIA is required for aminoglycoside accumulation in cochlear hair cells.
Myosin VIIA is expressed by sensory hair cells and has a primary structure predicting a role in membrane trafficking and turnover, processes that may underlie the susceptibility of hair cells to aminoglycoside antibiotics. [3H]Gentamicin accumulation and the effects of aminoglycosides were therefore examined in cochlear cultures of mice with different missense mutations in the myosin VIIA gene, Myo7a, to see whether myosin VIIA plays a role in aminoglycoside ototoxicity. Hair cells from homozygous mutant Myo7a(sh1) mice, with a mutation in a non-conserved region of the myosin VIIA head, respond rapidly to aminoglycoside treatment and accumulate high levels of gentamicin. Hair cells from homozygous mutant Myo7a(6J) mice, with a mutation at a highly conserved residue close to the ATP binding site of the myosin VIIA head, do not accumulate [3H]gentamicin and are protected from aminoglycoside ototoxicity. Hair cells from heterozygotes of both alleles accumulate [3H]gentamicin and respond to aminoglycosides. Although aminoglycoside uptake is thought to be via apical surface-associated endocytosis, coated pit numbers on the apical membrane of heterozygous and homozygous Myo7a(6J) hair cells are similar. Pulse-chase experiments with cationic ferritin confirm that the apical endocytotic pathway is functional in homozygous Myo7a(6J) hair cells. Transduction currents can be recorded from both heterozygous and homozygous Myo7a(6J) hair cells, suggesting it is unlikely that the drug enters via diffusion through the mechanotransducer channel. The results show that myosin VIIA is required for aminoglycoside accumulation in hair cells. Myosin VIIA may transport a putative aminoglycoside receptor to the hair cell surface, indirectly translocate it to sites of membrane retrieval, or retain it in the endocytotic pathway
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