1,986 research outputs found

    B and D physics from the Tevatron

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    The CDF and D0 experiments at the Tevatron ppˉp\bar{p} collider established that extensive and detailed exploration of the b-quark dynamics is possible in hadron collisions, with results competitive and supplementary to B-factories. In this paper we review the current state of Tevatron's heavy flavor measurements considering two main categories: searches for non standard model physics (results on rare decays and CP-violation) and determinations of standard model parameters (annihilation in Bh+hB \to h^+ h^- decays and γ\gamma angle measurement through BDKB\to DK modes)

    CKM physics at CDF

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    A precise knowledge of the CKM matrix elements is one of the primary goals of the CDF experiment. The Tevatron collider at Fermilab, operating at s = 1.96 , collected 1 f b −1 of data corresponding to a huge b b ¯ sample. In this paper the recent measurements performed in the CKM sector will be presented

    Recent results on the Unitarity Triangle from Tevatron

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    I report on the recent results of Tevatron experiments on the Unitarity Triangle. In particular the measurements of the direct CP asymmetry (ACP ) and of the double ratio of CP-even (D → K−K+ or π−π+) to flavor (D → K−π+) eigenstate branching fraction (RCP) in the B → DK decays, that can be used to improve the determination of the CKM angle γ, and the search for new physics in the CP-violating Bs phase βs using the B0s → J/ψφ decay

    Brunetto Latini, Tresor.

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    Nuova edizione critica del testo antico-francese, con traduzione e commento. Sergio vatteroni ha curato e tradotto il libro terz

    Urocortins in the mammalian endocrine system

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    Urocortins (Ucns), peptides belonging to the corticotropin-releasing hormone (CRH) family, are classified into Ucn1, Ucn2, and Ucn3. They are involved in regulating several body functions by binding to two G protein-coupled recep- tors: receptor type 1 (CRHR1) and type 2 (CRHR2). In this review, we provide a historical overview of research on Ucns and their receptors in the mammalian endocrine system. Although the literature on the topic is limited, we focused our attention particularly on the main role of Ucns and their receptors in regulating the hypothalamic–pituitary–adre- nal and thyroid axes, reproductive organs, pancreas, gastrointestinal tract, and other tissues characterized by “diffuse” endocrine cells in mammals. The prominent function of these peptides in health conditions led us to also hypothesize an action of Ucn agonists/antagonists in stress and in various diseases with its critical consequences on behavior and physiology. The potential role of the urocortinergic system is an intriguing topic that deserves further in-depth investi- gations to develop novel strategies for preventing stress-related conditions and treating endocrine diseases

    Effects of castration on the immunoreactivity to NGF, BDNF and their receptors in the pelvic ganglia of the male rat

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    Nerve growth factor (NGF) and brain derived neurotrophic factor (BDNF) and are members of the neurotrophin family, a family of neurotrophic factors that also includes neurotrophin (NT) 3 and NT4/5. Neurotrophins have essential roles in the survival, development and differentiation of neurons in the central and peripheral nervous systems. Neurotrophins exert their effects by binding to corresponding receptors which are formed by the tyrosine protein kinases TrkA, TrkB and TrkC, and the low affinity neurotrophic receptor (p75NTR). In the present study, using immunohistochemistry and quantitative analysis, we have investigated immunoreactivity to BDNF, NGF, TrkB, p75NTR and TrkA in the pelvic ganglia of normal and castrated rats. Neurons of the pelvic ganglia expressed both these neurotrophins and their receptors. After castration the immunoreactivity persisted. However, the number of BDNF- and p75NTR–IR cells statistically significant decreased after castration. These results suggest that castration modulates the expression of neurotrophins and their receptors in pelvic autonomic neurons
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