Anomalous electromagnetic coupling via entanglement at the nanoscale

This is the final version of the article. Available from IoP Publishing via the DOI in this record.Understanding unwanted mutual interactions between devices at the nanoscale is crucial for the study of the electromagnetic compatibility in nanoelectronic and nanophotonic systems. Anomalous electromagnetic coupling (crosstalk) between nanodevices may arise from the combination of electromagnetic interaction and quantum entanglement. In this paper we study in detail the crosstalk between two identical nanodevices, each consisting of a quantum emitter (atom, quantum dot, etc), capacitively coupled to a pair of nanoelectrodes. Using the generalized susceptibility concept, the overall system is modeled as a two-port within the framework of the electrical circuit theory and it is characterized by the admittance matrix. We show that the entanglement changes dramatically the physical picture of the electromagnetic crosstalk. In particular, the excitation produced in one of the ports may be redistributed in equal parts between both the ports, in spite of the rather small electromagnetic interactions. Such an anomalous crosstalk is expected to appear at optical frequencies in lateral GaAs double quantum dots. A possible experimental set up is also discussed. The classical concepts of interference in the operation of electronic devices, which have been known since the early days of radio-communications and are associated with electromagnetic compatibility, should then be reconsidered at the nanoscale.This research was supported in part by the EU Horizon 2020 project H2020-MSCA-RISE-2014-644076 CoExAN and EU FP7 projects, FP7-PEOPLE-2012-IRSES-316432 QOCaN and FP7-PEOPLE-2013-IRSES-612285 CANTOR. Discussions of the basic ideas underlying this work with Dr S Starobinets and Dr D Mogilevtsev are acknowledged

Quantum entanglement in electric circuits: From anomalous crosstalk to electromagnetic compatibility in nano-electronics

This is the author accepted manuscript. The final version is available from the publisher via the DOI in this record..We show that the electromagnetic coupling at the nanoscale may be accompanied by another coupling mechanism, related to quantum entanglement. Consequently, a combined 'electromagnetic-quantum' coupling is created, which stipulates long-distance and long-living interactions in electric circuits. Manifestation of this effect in electromagnetic compatibility (EMC) is discussed. An efficient theoretical framework for EMC analysis in nanoelectronics is developed based on the generalized theory of electric circuits. It is shown that the action of quantum entanglement is equivalent to an addition of the supplementary elements in electric circuit with the effective admittances defined as general susceptibilities that can be calculated using the Kubo-technique.This work was supported in part by EU grants FP7-PEOPLE-2009-IRSES- 247007 CACOMEL and FP7-PEOPLE-2013-IRSES- 612285 CANTOR

Nanoscale Electromagnetic Compatibility: Quantum Coupling and Matching in Nanocircuits

AcceptedArticle in PressThe paper investigates two typical electromagnetic compatibility (EMC) problems, namely, coupling and matching in nanoscale circuits composed of nano-interconnects and quantum devices in entangled state. Nano-interconnects under consideration are implemented by using carbon nanotubes or metallic nanowires (NWs), while quantum devices by semiconductor quantum dots. Equivalent circuits of such nanocircuits contain additional elements arising at nanoscale due to quantum effects. As a result, the notions of coupling and impedance matching are reconsidered. Two examples are studied: in the first one, electromagnetically coupled NWs are connected to classical lumped devices; in the second one, electromagnetically uncoupled transmission lines are terminated on quantum devices in entangled states. In both circuits, the EMC features qualitatively and quantitatively differ from their classical analogs. In the second example, we demonstrate the existence of quantum coupling, due to the entanglement, which exists in spite of the absence of classical electromagnetic coupling. The entanglement also modifies the matching condition introducing a dependence of the optimal value of load impedance on the line length

Fast Discrete Diagnostics of EMC of Complex Co-Located Radio Systems by Using Worst-Case Models of Electromagnetic Spurious Couplings

A technique of high computational efficiency for EMC diagnostics of complex co-located set of radio systems is presented. The technique is based on the following: the use of worst-case models of electromagnetic spurious couplings between antennas (in order to eliminate the second-type errors in detection of dangerous couplings), iterative refinement of these models for the potentially dangerous couplings in the process of solving an EMC problem, and the extra efficient discrete technique for nonlinear behavior simulation of radio receiver operation in complex electromagnetic environment

Making investment decisions based on dcf-modeling

This article provides how the DCF-model is calculated and how an investor, based on it, makes a decision to buy / sell / hold shares

Guanidino anthrathiophenediones as G-quadruplex binders: uptake, intracellular localization, and anti-Harvey-Ras gene activity in bladder cancer cells.

We prepared a series of anthrathiophenediones (ATPDs) with guanidino-alkyl side chains of different length (compounds 1, 10−13). The aim was to investigate their interaction with DNA and RNA G-quadruplexes, their uptake in malignant and nonmalignant cells, and their capacity to modulate gene expression and inhibit cell growth. Flow cytometry showed that the ATPDs enter more efficiently in malignant T24 bladder cells than in nonmalignant embryonic kidney 293 or fibroblast NIH 3T3 cells. In T24 malignant cells, compound 1, with two ethyl side chains, is taken up by endocytosis, while 12 and 13, with respectively propyl and butyl side chains, are transported by passive diffusion. The designed ATPDs localize in the cytoplasm and nucleus and tightly bind to DNA and RNA G-quadruplexes. They also decrease HRAS expression, increase the cell population in G0/G1, and strongly inhibit proliferation in malignant T24 bladder cells, but not in nonmalignant 293 or NIH 3T3 cells