Analyse de l’endommagement et ses conséquences sur les propriétés mécaniques

Le vieillissement thermique d’un jonc composite carbone/époxy a été étudié dans l’air entre 160 et 220 °C par tomographie X, microscopie optique et flexion trois points. La thermo-oxydation de la matrice affecte une couche superficielle trop fine pour pouvoir altérer les propriétés mécaniques. La thermolyse de la matrice est donc le principal mode de dégradation. Elle conduit à la formation de longs microcanaux parallèles aux fibres dans les régions les plus riches en matrice du cœur du jonc. Leur multiplication et leur coalescence entraînent une chute progressive du module d’élasticité et, dès que le taux de porosité atteint une valeur critique de l’ordre de 3,6 %, une chute catastrophique de la contrainte à rupture. Sur la base de ces résultats, des équations reliant l’évolution des propriétés mécaniques à des critères d’endommagement dérivés du taux de porosité ont été établies

Development of the One-Sided Nonlinear Adaptive Doppler Shift Estimation

The new development of a one-sided nonlinear adaptive shift estimation technique (NADSET) is introduced. The background of the algorithm and a brief overview of NADSET are presented. The new technique is applied to the wind parameter estimates from a 2-micron wavelength coherent Doppler lidar system called VALIDAR located in NASA Langley Research Center in Virginia. The new technique enhances wind parameters such as Doppler shift and power estimates in low Signal-To-Noise-Ratio (SNR) regimes using the estimates in high SNR regimes as the algorithm scans the range bins from low to high altitude. The original NADSET utilizes the statistics in both the lower and the higher range bins to refine the wind parameter estimates in between. The results of the two different approaches of NADSET are compared

PowerDuck: A GOOSE Data Set of Cyberattacks in Substations

Power grids worldwide are increasingly victims of cyberattacks, where attackers can cause immense damage to critical infrastructure. The growing digitalization and networking in power grids combined with insufficient protection against cyberattacks further exacerbate this trend. Hence, security engineers and researchers must counter these new risks by continuously improving security measures. Data sets of real network traffic during cyberattacks play a decisive role in analyzing and understanding such attacks. Therefore, this paper presents PowerDuck, a publicly available security data set containing network traces of GOOSE communication in a physical substation testbed. The data set includes recordings of various scenarios with and without the presence of attacks. Furthermore, all network packets originating from the attacker are clearly labeled to facilitate their identification. We thus envision PowerDuck improving and complementing existing data sets of substations, which are often generated synthetically, thus enhancing the security of power grids

When and How to Aggregate Message Authentication Codes on Lossy Channels?

Aggregation of message authentication codes (MACs) is a proven and efficient method to preserve valuable bandwidth in resource-constrained environments: Instead of appending a long authentication tag to each message, the integrity protection of multiple messages is aggregated into a single tag. However, while such aggregation saves bandwidth, a single lost message typically means that authentication information for multiple messages cannot be verified anymore. With the significant increase of bandwidth-constrained lossy communication, as applications shift towards wireless channels, it thus becomes paramount to study the impact of packet loss on the diverse MAC aggregation schemes proposed over the past 15 years to assess when and how to aggregate message authentication. Therefore, we empirically study all relevant MAC aggregation schemes in the context of lossy channels, investigating achievable goodput improvements, the resulting verification delays, processing overhead, and resilience to denial-of-service attacks. Our analysis shows the importance of carefully choosing and configuring MAC aggregation, as selecting and correctly parameterizing the right scheme can, e.g., improve goodput by 39% to 444%, depending on the scenario. However, since no aggregation scheme performs best in all scenarios, we provide guidelines for network operators to select optimal schemes and parameterizations suiting specific network settings

Modulation of NK cell activation by exogenous calcium from alginate dressings in vitro

Natural Killer (NK) cells participate in the defense against infection by killing pathogens and infected cells and secreting immuno-modulatory cytokines. Defects in NK cell activity have been reported in obese, diabetic, and elderly patients that are at high risk of developing infected chronic wounds. Calcium alginate dressings are indicated for the debridement during the inflammatory phase of healing. Since calcium ions are major activators of NK cells, we hypothesized that these dressings could enhance NK functions, as investigated in vitro herein. Primary human blood NK cells were freshly-isolated from healthy volunteers and exposed to conditioned media (CM) from two alginate dressings, Algosteril® (ALG, pure Ca2+ alginate) and Biatain® Alginate (BIA, Ca2+ alginate with CMC), in comparison with an exogenous 3mM calcium solution. Our results demonstrated that exogenous calcium and ALG-CM, but not BIA-CM, induced NK cell activation and enhanced their capacity to kill their targets as a result of increased degranulation. NK cell stimulation by ALG depended on the influx of extracellular Ca2+via the SOCE Ca2+ permeable plasma membrane channels. ALG-CM also activated NK cell cytokine production of IFN-γ and TNF-α through a partly Ca2+-independent mechanism. This work highlights the non-equivalence between alginate dressings for NK cell stimulation and shows that the pure calcium alginate dressing Algosteril® enhances NK cell cytotoxic and immuno-modulatory activities. Altogether, these results underline a specific property of this medical device in innate defense that is key for the cutaneous wound healing process