26,840 research outputs found

    On Lorentz violation in e ⁣ ⁣+ ⁣e+ ⁣ ⁣μ ⁣ ⁣+ ⁣μ+e^{-}\!\!+\!e^{+}\!\rightarrow\!\mu^{-}\!\!+\!\mu^{+} scattering at finite temperature

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    Small violation of Lorentz and CPT symmetries may emerge in models unifying gravity with other forces of nature. An extension of the standard model with all possible terms that violate Lorentz and CPT symmetries are included. Here a CPT-even non-minimal coupling term is added to the covariant derivative. This leads to a new interaction term that breaks the Lorentz symmetry. Our main objective is to calculate the cross section for the e ⁣ ⁣+ ⁣e+ ⁣ ⁣μ ⁣ ⁣+ ⁣μ+e^{-}\!\!+\!e^{+}\!\rightarrow\!\mu^{-}\!\!+\!\mu^{+} scattering in order to investigate any violation of Lorentz and/or CPT symmetry at finite temperature. Thermo Field Dynamics formalism is used to consider finite temperature effects.Comment: 12 pages, 1 figure, accepted for publication in PL

    A Flexible Implementation of a Matrix Laurent Series-Based 16-Point Fast Fourier and Hartley Transforms

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    This paper describes a flexible architecture for implementing a new fast computation of the discrete Fourier and Hartley transforms, which is based on a matrix Laurent series. The device calculates the transforms based on a single bit selection operator. The hardware structure and synthesis are presented, which handled a 16-point fast transform in 65 nsec, with a Xilinx SPARTAN 3E device.Comment: 4 pages, 4 figures. IEEE VI Southern Programmable Logic Conference 201

    Early appraisal of the fixation probability in directed networks

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    In evolutionary dynamics, the probability that a mutation spreads through the whole population, having arisen in a single individual, is known as the fixation probability. In general, it is not possible to find the fixation probability analytically given the mutant's fitness and the topological constraints that govern the spread of the mutation, so one resorts to simulations instead. Depending on the topology in use, a great number of evolutionary steps may be needed in each of the simulation events, particularly in those that end with the population containing mutants only. We introduce two techniques to accelerate the determination of the fixation probability. The first one skips all evolutionary steps in which the number of mutants does not change and thereby reduces the number of steps per simulation event considerably. This technique is computationally advantageous for some of the so-called layered networks. The second technique, which is not restricted to layered networks, consists of aborting any simulation event in which the number of mutants has grown beyond a certain threshold value, and counting that event as having led to a total spread of the mutation. For large populations, and regardless of the network's topology, we demonstrate, both analytically and by means of simulations, that using a threshold of about 100 mutants leads to an estimate of the fixation probability that deviates in no significant way from that obtained from the full-fledged simulations. We have observed speedups of two orders of magnitude for layered networks with 10000 nodes
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