63,306 research outputs found
AgRISTARS: Renewable resources inventory. Land information support system implementation plan and schedule
The planning and scheduling of the use of remote sensing and computer technology to support the land management planning effort at the national forests level are outlined. The task planning and system capability development were reviewed. A user evaluation is presented along with technological transfer methodology. A land management planning pilot test of the San Juan National Forest is discussed
Evaluation of VICAR software capability for land information support system needs
A preliminary evaluation of the processing capability of the VICAR software for land information support system needs is presented. The geometric and radiometric properties of four sets of LANDSAT data taken over the Elk River, Idaho quadrangle were compared. Storage of data sets, the means of location, pixel resolution, and radiometric and geometric characteristics are described. Recommended modifications of VICAR programs are presented
Controlled cavity-QED using a photonic crystal waveguide-cavity system
We introduce a photonic crystal waveguide-cavity system for controlling
single photon cavity-QED processes. Exploiting Bloch mode analysis, and
medium-dependent Green function techniques, we demonstrate that the propagation
of single photons can be accurately described analytically, for integrated
periodic waveguides with little more than four unit cells, including an output
coupler. We verify our analytical approach by comparing to rigorous numerical
calculations for a range of photonic crystal waveguide lengths. This allows one
to nano-engineer various regimes of cavity-QED with unprecedented control. We
demonstrate Purcell factors of greater than 1000 and on-chip single photon beta
factors of about 80% efficiency. Both weak and strong coupling regimes are
investigated, and the important role of waveguide length on the output emission
spectra is shown, for vertically emitted emission and output waveguide
emission
Population-based incremental learning with associative memory for dynamic environments
Copyright © 2007 IEEE. Reprinted from IEEE Transactions on Evolutionary Computation.
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By choosing to view this document, you agree to all provisions of the copyright laws protecting it.In recent years there has been a growing interest in studying evolutionary algorithms (EAs) for dynamic optimization problems (DOPs) due to its importance in real world applications. Several approaches, such as the memory and multiple population schemes, have been developed for EAs to address dynamic problems. This paper investigates the application of the memory scheme for population-based incremental learning (PBIL) algorithms, a class of EAs, for DOPss. A PBIL-specific associative memory scheme, which stores best solutions as well as corresponding environmental information in the memory, is investigated to improve its adaptability in dynamic environments. In this paper, the interactions between the memory scheme and random immigrants, multi-population, and restart schemes for PBILs in dynamic environments are investigated. In order to better test the performance of memory schemes for PBILs and other EAs in dynamic environments, this paper also proposes a dynamic environment generator that can systematically generate dynamic environments of different difficulty with respect to memory schemes. Using this generator a series of dynamic environments are generated and experiments are carried out to compare the performance of investigated algorithms. The experimental results show that the proposed memory scheme is efficient for PBILs in dynamic environments and also indicate that different interactions exist between the memory scheme and random immigrants, multi-population schemes for PBILs in different dynamic environments
Dual population-based incremental learning for problem optimization in dynamic environments
Copyright @ 2003 Asia Pacific Symposium on Intelligent and Evolutionary SystemsIn recent years there is a growing interest in the research of evolutionary algorithms for dynamic optimization problems since real world problems are usually dynamic, which presents serious challenges to traditional evolutionary algorithms. In this paper, we investigate the application of Population-Based Incremental Learning (PBIL) algorithms, a class of evolutionary algorithms, for problem optimization under dynamic environments. Inspired by the complementarity mechanism in nature, we propose a Dual PBIL that operates on two probability vectors that are dual to each other with respect to the central point in the search space. Using a dynamic problem generating technique we generate a series of dynamic knapsack problems from a randomly generated stationary knapsack problem and carry out experimental study comparing the performance of investigated PBILs and one traditional genetic algorithm. Experimental results show that the introduction of dualism into PBIL improves its adaptability under dynamic environments, especially when the environment is subject to significant changes in the sense of genotype space
Experimental study on population-based incremental learning algorithms for dynamic optimization problems
Copyright @ Springer-Verlag 2005.Evolutionary algorithms have been widely used for stationary optimization problems. However, the environments of real world problems are often dynamic. This seriously challenges traditional evolutionary algorithms. In this paper, the application of population-based incremental learning (PBIL) algorithms, a class of evolutionary algorithms, for dynamic problems is investigated. Inspired by the complementarity mechanism in nature a Dual PBIL is proposed, which operates on two probability vectors that are dual to each other with respect to the central point in the genotype space. A diversity maintaining technique of combining the central probability vector into PBIL is also proposed to improve PBILs adaptability in dynamic environments. In this paper, a new dynamic problem generator that can create required dynamics from any binary-encoded stationary problem is also formalized. Using this generator, a series of dynamic problems were systematically constructed from several benchmark stationary problems and an experimental study was carried out to compare the performance of several PBIL algorithms and two variants of standard genetic algorithm. Based on the experimental results, we carried out algorithm performance analysis regarding the weakness and strength of studied PBIL algorithms and identified several potential improvements to PBIL for dynamic optimization problems.This work was was supported by
UK EPSRC under Grant GR/S79718/01
Marked central nervous system pathology in CD59 knockout rats following passive transfer of Neuromyelitis optica immunoglobulin G.
Neuromyelitis optica spectrum disorders (herein called NMO) is an inflammatory demyelinating disease of the central nervous system in which pathogenesis involves complement-dependent cytotoxicity (CDC) produced by immunoglobulin G autoantibodies targeting aquaporin-4 (AQP4-IgG) on astrocytes. We reported evidence previously, using CD59-/- mice, that the membrane-associated complement inhibitor CD59 modulates CDC in NMO (Zhang and Verkman, J. Autoimmun. 53:67-77, 2014). Motivated by the observation that rats, unlike mice, have human-like complement activity, here we generated CD59-/- rats to investigate the role of CD59 in NMO and to create NMO pathology by passive transfer of AQP4-IgG under conditions in which minimal pathology is produced in normal rats. CD59-/- rats generated by CRISPR/Cas9 technology showed no overt phenotype at baseline except for mild hemolysis. CDC assays in astrocyte cultures and cerebellar slices from CD59-/- rats showed much greater sensitivity to AQP4-IgG and complement than those from CD59+/+ rats. Intracerebral administration of AQP4-IgG in CD59-/- rats produced marked NMO pathology, with astrocytopathy, inflammation, deposition of activated complement, and demyelination, whereas identically treated CD59+/+ rats showed minimal pathology. A single, intracisternal injection of AQP4-IgG in CD59-/- rats produced hindlimb paralysis by 3 days, with inflammation and deposition of activated complement in spinal cord, optic nerves and brain periventricular and surface matter, with most marked astrocyte injury in cervical spinal cord. These results implicate an important role of CD59 in modulating NMO pathology in rats and demonstrate amplification of AQP4-IgG-induced NMO disease with CD59 knockout
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