Direct CP violation in τ±→K±ρ0(ω)ντ→K±π+π−ντ\tau^\pm\rightarrow K^\pm \rho^0 (\omega)\nu_\tau \rightarrow K^\pm \pi^+\pi^-\nu_\tau

We study the direct CP violation in the $\tau^\pm\rightarrow K^\pm \rho^0 (\omega)\nu_\tau \rightarrow K^\pm \pi^+\pi^-\nu_\tau$ decay process in the Standard Model. An interesting mechanism involving the charge symmetry violating mixing between $\rho^0$ and $\omega$ is applied to enlarge the CP asymmetry. With this mechanism, the maximum differential and localized integrated CP asymmetries can reach $-(5.6^{+2.9}_{-1.7})\times10^{-12}$ and $6.3^{+2.4}_{-3.3}\times 10^{-11}$, respectively, which still leave plenty room for CP-violating New Physics to be discovered through this process

Changes in Cyclic Guanosine Monophosphate Channel of 661w Cells In vitro with Excessive Light Time

Purpose: To determine the response time and protective mechanism of the cyclic guanosine monophosphate (cGMP) channel in 661w cells. Methods: 661w cells were exposed to 4500Lux visible light for three and four days at the following exposure time periods per day: 20, 30, 60, 90, 120, and 180. Cells were incubated for the rest of the time without any other treatment. Cell activity and cell death rates were measured with Hoechst/PI (diphenylmethane/propidium iodide) staining. Western Blot was used to detect the levels of guanylate cyclase-activating proteins 1 (GCAP1), cGMP, and phosphodiesterase (PDE)6 in the cGMP-gated channel. Results: 661w cells showed low mortality within three days. The mortality rate increased from the fourth day, especially during the longer times (120 and 180 min) of light exposure. After three-day illumination, the level of cGMP increased after 20 and 90 min and the level of GCAP1 increased after 60 and 90 min. After four days of illumination, the level of GCAP1 upregulated after a time of 20 and 60 min, while the cGMP level decreased from 30 min. The expression of PDE6 upregulated at each light period. Conclusion: The survival rate of 661w cells was relevant to the time of light exposure. The changes in GCAP1, cGMP, and PDE6 levels over time were possibly related to cell metabolism and restoration after light-induced damage

Ventilation of a monsoon‐dominated ocean : subduction and obduction in the North Indian Ocean

Author Posting. © American Geophysical Union, 2018. This article is posted here by permission of American Geophysical Union for personal use, not for redistribution. The definitive version was published in Journal of Geophysical Research: Oceans 123 (2018): 4449-4463, doi:10.1029/2017JC013719.Based on the characteristics of oceanic circulation in a monsoon‐dominated ocean, a new framework of annual ventilation, including subduction and obduction, is postulated and applied to the North Indian Ocean based on both SODA and GODAS. It is revealed that besides the winter season, ventilation can also occur in summer. Considering the horizontal resolution, SODA results are mainly discussed, with GODAS results given for validity of key conclusions. The annual subduction/obduction rate in the North Indian Ocean based on SODA is estimated at 10.2 Sv/11 Sv averaged from 1960 to 2009, with 4.2 Sv/6.2 Sv occurring during winter monsoon period and 6 Sv/4.8 Sv during summer monsoon period, respectively. Both subduction and obduction feature great interannual variability, with the vertical pumping term of decisive importance. Furthermore, the concepts of the penetration depth through subduction and the origin depth through obduction are postulated. The penetration depth in the Arabian Sea is on the order of 50 to 200 m; the origin depth through obduction in the Arabian Sea is deeper than that in the Bay of Bengal, with the deepest on the order of 200 to 250 m along the western boundary.AoShan Talents program Gr;ant Number: 2015ASTP; Global Change and Air‐Sea Interaction Grant Number: GASI‐IPOVAI‐04; National Key Research and Development Program of China Grant Number: 2017YFC14040022019-01-0

Possible open-charmed pentaquark molecule Ωc(3188)\Omega_c(3188) --- the DΞD \Xi bound state --- in the Bethe-Salpeter formalism

We study the $S$-wave $D\Xi$ bound state in the Bethe-Salpeter formalism in the ladder and instantaneous approximations. With the kernel generated by the hadronic effective Lagrangian, two open-charmed bound states, which quantum numbers are $I=0$, $J^P=(\frac{1}{2})^-$ and $I=1$, $J^P=(\frac{1}{2})^-$, respectively, are predicted as new candidates of hadronic pentaquark molecules in our formalism. If existing, they could contribute to the broad 3188 eV structure near the five new narrow $\Omega_c$ states observed recently by the LHCb Collaboration.Comment: 8 pages, 4 figures, accepted by Eur. Phys. J.

Value of superb microvascular imaging ultrasonography in the diagnosis of carpal tunnel syndrome: Compared with color Doppler and power Doppler.

The aim of this study was to compare the value of superb microvascular imaging (SMI) in carpal tunnel syndrome (CTS) with that of color Doppler ultrasonography (CDUS) and power Doppler ultrasonography (PDUS).Fifty patients with symptomatic CTS and 25 healthy volunteers were enrolled. The cross-sectional area (CSA), CDUS score, PDUS score, and SMI score of the median nerve (MN) at the carpal tunnel were recorded. The value of different ultrasonography (US) diagnostic strategies was calculated.The blood flow display ratio in the MN of the healthy volunteers had no statistical difference between CDUS, PDUS, and SMI (20%, 32%, and 48%, respectively, P \u3e.05). The blood flow display ratio for SMI in patients was significantly higher than that of CDUS and PDUS (90%, 52%, and 60%, respectively,

New estimation of the nuclear de-excitation line emission from the supernova remnant Cassiopeia A

MeV nuclear de-excitation lines serve as a unique tool to study low-energy cosmic rays (CRs), containing both spectral and elemental information of the interacting material. In this paper, we estimated the possible nuclear de-excitation lines from the young supernova remnant Cassiopeia A. Given different CR spectral shapes and interacting materials, we found the predicted fluxes of strong narrow line emissions from the remnant are highly model-dependent, ranging from about $1\times10^{-10}\,{\rm \,cm^{-2}\,s^{-1}}$ to $1\times10^{-6}\, {\rm \,cm^{-2}\,s^{-1}}$ for the 4.44 MeV narrow line and from about $4\times10^{-11}\,{\rm \,cm^{-2}\,s^{-1}}$ to $2\times10^{-7}{\rm \,cm^{-2}\,s^{-1}}$ for the 6.13 MeV narrow line, respectively. Based on the new estimation, we also discussed the detection probability of these line emissions against the MeV diffuse Galactic background under different assumptions of instrument response functions.Comment: 6 pages, 6 figures, accepted for publication in MNRA

Implications of C1q/TNF-related protein superfamily in patients with coronary artery disease.

The C1q complement/TNF-related protein superfamily (CTRPs) displays differential effects on the regulation of metabolic homeostasis, governing cardiovascular function. However, whether and how they may serve as predictor/pro-diagnosis factors for assessing the risks of coronary artery disease (CAD) remains controversial. Therefore, we performed a clinical study to elaborate on the implication of CTRPs (CTRP1, CTRP5, CTRP7, and CTRP15) in CAD. CTRP1 were significantly increased, whereas CTRP7 and CTRP15 levels were decreased in CAD patients compared to the non-CAD group. Significant differences in CTRP1 levels were discovered between the single- and triple-vascular-vessel lesion groups. ROC analysis revealed that CTRP7 and CTRP15 may serve as CAD markers, while CTRP1 may serve as a marker for the single-vessel lesion of CAD. CTRP1 and CTRP5 can serve as markers for the triple-vessel lesion. CTRP1 may serve as an independent risk predictor for triple-vessel lesion, whereas CTRP15 alteration may serve for a single-vessel lesion of CAD. CTRP1 may serve as a novel superior biomarker for diagnosis of severity of vessel-lesion of CAD patients. CTRP7, CTRP15 may serve as more suitable biomarker for the diagnosis of CAD patients, whereas CTRP5 may serve as an independent predictor for CAD. These findings suggest CTRPs may be the superior predictive factors for the vascular lesion of CAD and represent novel therapeutic targets against CAD