219 research outputs found
Dynamics of entanglement for a two-parameter class of states in a qubit-qutrit system
We investigate the dynamics of entanglement for a two-parameter class of
states in a hybrid qubit-qutrit system under the influence of various
dissipative channels. Our results show that entanglement sudden death (ESD) is
a general phenomenon and it usually takes place in a qubit-qutrit system
interacting with various noisy channels, not only the case with dephasing and
depolarizing channels observed by others. ESD can only be avoided for some
initially entangled states under some particular noisy channels. Moreover, the
environment affects the entanglement and the coherence of the system in very
different ways.Comment: 9 pages, 6 figures, 1 table. A negligible error for type on Eq(7) is
correcte
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Activated N-Ras signaling regulates arterial-venous specification in zebrafish
Background: The aberrant activation of Ras signaling is associated with human diseases including hematological malignancies and vascular disorders. So far the pathological roles of activated Ras signaling in hematopoiesis and vasculogenesis are largely unknown. Methods: A conditional Cre/loxP transgenic strategy was used to mediate the specific expression of a constitutively active form of human N-Ras in zebrafish endothelial and hematopoietic cells driven by the zebrafish lmo2 promoter. The expression of hematopoietic and endothelial marker genes was analyzed both via whole mount in situ hybridization (WISH) assay and real-time quantitative PCR (qPCR). The embryonic vascular morphogenesis was characterized both by living imaging and immunofluorescence on the sections with a confocal microscopy, and the number of endothelial cells in the embryos was quantified by flow cytometry. The functional analyses of the blood circulation were carried out by fluorescence microangiography assay and morpholino injection. Results: In the activated N-Ras transgenic embryos, the primitive hematopoiesis appeared normal, however, the definitive hematopoiesis of these embryos was completely absent. Further analysis of endothelial cell markers confirmed that transcription of arterial marker ephrinB2 was significantly decreased and expression of venous marker flt4 excessively increased, indicating the activated N-Ras signaling promotes the venous development at the expense of arteriogenesis during zebrafish embryogenesis. The activated N-Ras-expressing embryos showed atrophic axial arteries and expansive axial veins, leading to no definitive hematopoietic stem cell formation, the blood circulation failure and subsequently embryonic lethality. Conclusions: Our studies revealed for the first time that activated N-Ras signaling during the endothelial differentiation in vertebrates can disrupt the balance of arterial-venous specification, thus providing new insights into the pathogenesis of the congenital human vascular disease and tumorigenic angiogenesis
Calcium-sensing receptors regulate cardiomyocyte Ca2+ signaling via the sarcoplasmic reticulum-mitochondrion interface during hypoxia/reoxygenation
Communication between the SR (sarcoplasmic reticulum, SR) and mitochondria is important for cell survival and apoptosis. The SR supplies Ca2+ directly to mitochondria via inositol 1,4,5-trisphosphate receptors (IP3Rs) at close contacts between the two organelles referred to as mitochondrion-associated ER membrane (MAM). Although it has been demonstrated that CaR (calcium sensing receptor) activation is involved in intracellular calcium overload during hypoxia/reoxygenation (H/Re), the role of CaR activation in the cardiomyocyte apoptotic pathway remains unclear. We postulated that CaR activation plays a role in the regulation of SR-mitochondrial inter-organelle Ca2+ signaling, causing apoptosis during H/Re. To investigate the above hypothesis, cultured cardiomyocytes were subjected to H/Re. We examined the distribution of IP3Rs in cardiomyocytes via immunofluorescence and Western blotting and found that type 3 IP3Rs were located in the SR. [Ca2+]i, [Ca2+]m and [Ca2+]SR were determined using Fluo-4, x-rhod-1 and Fluo 5N, respectively, and the mitochondrial membrane potential was detected with JC-1 during reoxygenation using laser confocal microscopy. We found that activation of CaR reduced [Ca2+]SR, increased [Ca2+]i and [Ca2+]m and decreased the mitochondrial membrane potential during reoxygenation. We found that the activation of CaR caused the cleavage of BAP31, thus generating the pro-apoptotic p20 fragment, which induced the release of cytochrome c from mitochondria and the translocation of bak/bax to mitochondria. Taken together, these results reveal that CaR activation causes Ca2+ release from the SR into the mitochondria through IP3Rs and induces cardiomyocyte apoptosis during hypoxia/reoxygenation
Single-photon-assisted entanglement concentration of a multi-photon system in a partially entangled W state with weak cross-Kerr nonlinearity
We propose a nonlocal entanglement concentration protocol (ECP) for
-photon systems in a partially entangled W state, resorting to some
ancillary single photons and the parity-check measurement based on cross-Kerr
nonlinearity. One party in quantum communication first performs a parity-check
measurement on her photon in an -photon system and an ancillary photon, and
then she picks up the even-parity instance for obtaining the standard W state.
When she obtains an odd-parity instance, the system is in a less-entanglement
state and it is the resource in the next round of entanglement concentration.
By iterating the entanglement concentration process several times, the present
ECP has the total success probability approaching to the limit in theory. The
present ECP has the advantage of a high success probability. Moreover, the
present ECP requires only the -photon system itself and some ancillary
single photons, not two copies of the systems, which decreases the difficulty
of its implementation largely in experiment. It maybe have good applications in
quantum communication in future.Comment: 7 pages, 3 figure
Superconductivity and Charge-density-wave-like Transition in Th2Cu4As5
We report the synthesis, crystal structure, and physical properties of a
novel ternary compound, ThCuAs. The material crystallizes in a
tetragonal structure with lattice parameters {\AA} and
{\AA}. Its structure can be described as an alternating stacking
of fluorite-type ThAs layers with antifluorite-type double-layered
CuAs slabs. The measurement of electrical resistivity, magnetic
susceptibility and specific heat reveals that ThCuAs undergoes bulk
superconducting transition at 4.2 K. Moreover, all these physical quantities
exhibit anomalies at 48 K, where the Hall coefficient change the sign. These
findings suggest a charge-density-wave-like (CDW) transition, making
ThCuAs a rare example for studying the interplay between CDW and
superconductivity.Comment: 11 pages, 6 figures, and 1 tabl
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