53 research outputs found
Triple‐phased mesenteric CT angiography using a test bolus technique for evaluation of the mesenteric vasculature and small intestinal wall contrast enhancement in dogs
Hemodynamic Homeostasis during Acute Hypoxia in Septic and Nonseptic Piglets: Differential Role of Prostaglandins and Nitric Oxide
Bayesian inference of caprine herd growth and product yields using archaeological age-at-death profiles
International audienc
Bayesian inference of caprine herd growth and product yields using archaeological age-at-death profiles
International audienc
Schaffung eines nationalen Zentrums für Kinderneurochirurgie in der Slowakischen Republik - ist eine zentralisierte Spezialversorgung in der Kinderneurochirurgie erforderlich?
Using Deep Learning to Deconvolute Complex Spectra for Hyperspectral Imaging Applications
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Long-term delivery of FGF-6 changes the fiber type and fatigability of muscle reinnervated from embryonic neurons transplanted into adult rat peripheral nerve
Motoneuron death leads to muscle denervation and atrophy. Transplantation of embryonic neurons into peripheral nerves results in reinnervation and provides a strategy to rescue muscles from atrophy independent of neuron replacement in a damaged or diseased spinal cord. But the count of regenerating axons always exceeds the number of motor units in this model, so target-derived trophic factor levels may limit reinnervation. Our aim was to examine whether long-term infusion of fibroblast growth factor-6 (FGF-6) into denervated medial gastrocnemius muscles improved the function of muscles reinnervated from neurons transplanted into nerve of adult Fischer rats. Factor delivery (10 microg, 4 weeks) began after sciatic nerve transection. After a week of nerve degeneration, 1 million embryonic day 14-15 ventral spinal cord cells were transplanted into the distal tibial stump as a neuron source. Ten weeks later, neurons that expressed motoneuron markers survived in the nerves. More myelinated axons were in nerves to saline-treated muscles than in FGF-6-treated muscles. However, each group showed comparable reductions in muscle fiber atrophy because of reinnervation. Mean reinnervated fiber area was 43%-51% of non-denervated fibers. Denervated fiber area averaged 11%. FGF-6-treated muscles were more fatigable than other reinnervated muscles but had stronger motor units and fewer type I fibers than did saline-treated muscles. FGF-6 thus influenced function by changing the type of fiber reinnervated by transplanted neurons. Deficits in FGF-6 may also contribute to the increase in type I fibers in muscles reinnervated from peripheral axons, suggesting that the effects of FGF-6 on fiber type are independent of the neuron source used for reinnervation
Semiochemical Emission From Individual Galleries of the Southern Pine Beetle, (Coleoptera: Curculionidae: Scolytinae), Attacking Standing Trees
Interactions of light intensity, insecticide concentration, and time on the efficacy of systemic insecticides in suppressing populations of the sweetpotato whitefly (Hemiptera: Aleyrodidae) and the citrus mealybug (Hemiptera: Pseudococcidae)
The impact of light intensity on the uptake and persistence of the systemic neonicotinoid insecticides, imidacloprid and dinotefuran, were evaluated in poinsettia (Euphorbia pulcherrima Willd.) and yellow sage (Lantana camara L.). Insecticide residues were measured in leaves sampled from the treated plants at four time intervals after treatment to determine the relationship between insecticide concentration and efficacy against two insect pests: sweetpotato whitefly, Bemisia tabaci Gennadius, and the citrus mealybug, Planococcus citri Risso. The insecticides were evaluated at their respective label rate and at the comparable label rate of the other insecticide under two different light environments: ambient and shade. The uptake of dinotefuran into yellow sage was more rapid at both treatment rates than both rates of imidacloprid, resulting in higher percent mortality of whitefly nymphs (89.8–100) compared with imidacloprid (14.1–89.2) across all 4 wk. Additionally, plants that received both rates of dinotefuran had fewer whitefly pupae (<1.0) at week 4 compared with imidacloprid-treated plants (23.7–25.3). The uptake of dinotefuran into poinsettia plants was also more rapid and resulted in quicker and higher percent mortality of whitefly nymphs (89.5–99.6) compared with imidacloprid (14.1–89.2) across all 4 wk. However, despite efficient uptake, the efficacy of both systemic insecticides was less for citrus mealybug where percent mortality values were <50% among all the treatments across the 4 wk. The use of the two systemic insecticides evaluated in regards to pest management in horticultural cropping systems is discussed
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