31 research outputs found
Centromeric and telomeric genotype distribution.
<p>a) Genomic organisation of centromeric genotypes present in at least 2% of the study population, b) <i>KIR</i> genes present telomeric genotypes (those found in at least 2% of the study population). Filled box: gene is present; open box: gene is absent, x: known or novel motifs (e.g. Bx2: second novel B motif identified for the first time in this study), N: number of individuals carrying the genotype of interest.</p
Characteristics of study participants.
<p>Characteristics of study participants.</p
<i>KIR</i> genotypes and outcomes of hepatitis B infection.
<p><i>KIR</i> genotypes and outcomes of hepatitis B infection.</p
The genomic organisation of <i>KIR</i> genes on human chromosome 19.
<p><i>KIR</i> genes are tightly organised head-to-tail over approximately 150 kb within the Leukocyte Receptor Complex (LRC). Inhibitory <i>KIR</i> genes are shown in green, activating genes in orange, pseudogenes in yellow, and the recombination hotspot in black. <i>KIR</i> genes vary in size ranging from 10 kb to 16 kb and are separated from each other by about 2 kb of intergenic space, except for the 14 kb recombination hotspot zone upstream of <i>KIR2DL4</i> that separates telomeric from centromeric KIRs.</p
Characterising asset-based studies in public health: development of a framework
No description supplie
Association between <i>vacA</i> genotypes and clinical outcome.
<p>Incomplete <i>vacA</i> = either <i>vacA s</i> or <i>vacA m</i> regions were not detected (4/10 <i>vacAs1</i> was detected & <i>vacAm</i> was missing, 2/10 <i>vacAs2</i> detected and <i>vacAm</i> missing, 2/10 <i>vacAm1</i> detected and <i>vacAs</i> missing and for 2/10 both <i>vacAs</i> and <i>vacAm</i> were missing).</p
Association between <i>cagA</i> genotypes and disease type.
<p>DU = duodenal ulcer, GC = gastric carcinoma, GE = gastric erosion, GU = gastric ulcer, GUDU = gastric ulcer and duodenal ulcer, NUD = Non-ulcerative disease.</p