543 research outputs found
Thermal Control of the Intrinsic Magnetic Damping in a Ferromagnetic Metal
We report experiments on the control of intrinsic magnetic damping by thermal torque effects produced by the spin Seebeck effect and the anomalous Nernst effect in a thin layer of a ferromagnetic metal (Permalloy (Py), ). Damping is measured in ferromagnetic resonance (FMR) experiments on a sample excited by microwave radiation and detected by the dc-voltage-generated spin rectification and magnonic charge pumping in the Py film. Application of a temperature gradient in the longitudinal configuration increases or decreases the dc-voltage line width, depending on the sign of the thermal gradient, demonstrating that the magnetic damping in Py is controlled by currents generated by thermal effects. The absolute values of the line-width changes in Py may reach 1 order of magnitude larger than in the ferrimagnetic insulator yttrium iron garnet. The large change in magnetic damping is interpreted as a superposition of different phenomena in the same metallic ferromagnet
Readings By High School Students About The Beginning Of Quantum Physics Through Scientists' Texts
O objetivo deste estudo é compreender as interpretações de estudantes do ensino médio sobre as atividades de leitura por eles realizadas em uma aula de física num projeto de extensão de uma universidade estadual paulista, e sobre noções do início da Física Quântica, ao lerem textos escritos por cientistas. Utilizamos a Análise de Discurso, numa de suas vertentes francesas, para analisar as respostas dadas a um questionário aberto, com cinco perguntas, aplicado durante a aula. Constatamos que, prova velmente pelo fato de a leitura de textos de divulgação científica e de originais de cientistas não ser uma prática comum nas atividades escolares, a diversidade de sentidos produzidos pelos estudantes ao novo assunto, ao mesmo tempo que abre possibilidades para uma maior interação do professor, atenta para a necessidade de sua mediação quanto a dúvidas e equívocos.22229931
Rapid within- and transgenerational changes in thermal tolerance and fitness in variable thermal landscapes
Phenotypic plasticity may increase the performance and fitness and allow organisms to cope with variable environmental conditions. We studied within-generation plasticity and transgenerational effects of thermal conditions on temperature tolerance and demographic parameters in Drosophila melanogaster. We employed a fully factorial design, in which both parental (P) and offspring generations (F1) were reared in a constant or a variable thermal environment. Thermal variability during ontogeny increased heat tolerance in P, but with demographic cost as this treatment resulted in substantially lower survival, fecundity, and net reproductive rate. The adverse effects of thermal variability (V) on demographic parameters were less drastic in flies from the F1, which exhibited higher net reproductive rates than their parents. These compensatory responses could not totally overcome the challenges of the thermally variable regime, contrasting with the offspring of flies raised in a constant temperature (C) that showed no reduction in fitness with thermal variation. Thus, the parental thermal environment had effects on thermal tolerance and demographic parameters in fruit fly. These results demonstrate how transgenerational effects of environmental conditions on heat tolerance, as well as their potential costs on other fitness components, can have a major impact on populations’ resilience to warming temperatures and more frequent thermal extremes
Optimal Resource Allocation with Delay Guarantees for Network Slicing in Disaggregated RAN
In this article, we propose a novel formulation for the resource allocation
problem of a sliced and disaggregated Radio Access Network (RAN) and its
transport network. Our proposal assures an end-to-end delay bound for the
Ultra-Reliable and Low-Latency Communication (URLLC) use case while jointly
considering the number of admitted users, the transmission rate allocation per
slice, the functional split of RAN nodes and the routing paths in the transport
network. We use deterministic network calculus theory to calculate delay along
the transport network connecting disaggregated RANs deploying network functions
at the Radio Unit (RU), Distributed Unit (DU), and Central Unit (CU) nodes. The
maximum end-to-end delay is a constraint in the optimization-based formulation
that aims to maximize Mobile Network Operator (MNO) profit, considering a cash
flow analysis to model revenue and operational costs using data from one of the
world's leading MNOs. The optimization model leverages a Flexible Functional
Split (FFS) approach to provide a new degree of freedom to the resource
allocation strategy. Simulation results reveal that, due to its non-linear
nature, there is no trivial solution to the proposed optimization problem
formulation. Our proposal guarantees a maximum delay for URLLC services while
satisfying minimal bandwidth requirements for enhanced Mobile BroadBand (eMBB)
services and maximizing the MNO's profit.Comment: 21 pages, 10 figures. For the associated GitHub repository, see
https://github.com/LABORA-INF-UFG/paper-FGKCJ-202
Augmented β-cell function and mass in glucocorticoid-treated rodents are associated with increased islet ir-β /AKT/mTOR and decreased AMPK/ACC and AS160 signaling
FAPESP - FUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULOCNPQ - CONSELHO NACIONAL DE DESENVOLVIMENTO CIENTÍFICO E TECNOLÓGICOGlucocorticoid (GC) therapies may adversely cause insulin resistance (IR) that lead to a compensatory hyperinsulinemia due to insulin hypersecretion. The increased β-cell function is associated with increased insulin signaling that has the protein kinase B (AKT) substrate with 160 kDa (AS160) as an important downstream AKT effector. In muscle, both insulin and AMP-activated protein kinase (AMPK) signaling phosphorylate and inactivate AS160, which favors the glucose transporter (GLUT)-4 translocation to plasma membrane. Whether AS160 phosphorylation is modulated in islets from GC-treated subjects is unknown. For this, two animal models, Swiss mice and Wistar rats, were treated with dexamethasone (DEX) (1 mg/kg body weight) for 5 consecutive days. DEX treatment induced IR, hyperinsulinemia, and dyslipidemia in both species, but glucose intolerance and hyperglycemia only in rats. DEX treatment caused increased insulin secretion in response to glucose and augmented β-cell mass in both species that were associated with increased islet content and increased phosphorylation of the AS160 protein. Protein AKT phosphorylation, but not AMPK phosphorylation, was found significantly enhanced in islets from DEX-treated animals. We conclude that the augmented β-cell function developed in response to the GC-induced IR involves inhibition of the islet AS160 protein activity.Glucocorticoid (GC) therapies may adversely cause insulin resistance (IR) that lead to a compensatory hyperinsulinemia due to insulin hypersecretion. The increased β-cell function is associated with increased insulin signaling that has the protein kinase2014114FAPESP - FUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULOCNPQ - CONSELHO NACIONAL DE DESENVOLVIMENTO CIENTÍFICO E TECNOLÓGICOFAPESP - FUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULOCNPQ - CONSELHO NACIONAL DE DESENVOLVIMENTO CIENTÍFICO E TECNOLÓGICOsem informaçãosem informaçã
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