Employee Recruitment and Selection in CIDEM Hranice, a.s. Company

Import 05/08/2014Tato práce řeší výběr a získávání zaměstnanců ve společnosti CIDEM Hranice, a.s. Cílem práce je zhodnotit současný stav získávání a výběru zaměstnanců ve společnosti CIDEM Hranice a navrhnout opatření pro zlepšení tohoto procesu. Zaměřila jsem se na podrobnou analýzu tohoto procesu. Na základě odborné literatury jsou vysvětleny důležité pojmy, které se týkají získávání a výběru zaměstnanců v teoretické části. Tyto poznatky jsou poté převedeny do praktické úrovně za pomoci interních dokumentů podniku. Pro analýzu procesu byl sestaven dotazník, který byl distribuován zaměstnancům podniku. Získaná data byla zpracována pomocí datové matice a převedena do grafické podoby. Hlavním výsledkem práce jsou návrhy a doporučení pro organizaci v procesu získávání a výběru zaměstnanců.This thesis deals with employee selection and acquirement in the company CIDEM Hranice, a.s. The goal of this thesis is to evaluate current state of emplyee selection and acquirement process in the company CIDEM Hranice and suggest steps to improve this process. My aim is to do a thorough analysis of this process. Based on a technical literature, the important terms concerning employee selection and acquirement are explained in the theoretical part of the thesis. This knowledge is then transferred to practical level using internal documentation of the company. For the purpose of process analysis, a quistionaire was created and distributed to the employees of the company. Collected data was processed using data matrix and rendered to graphical output. Main outcome of this thesis is collection of suggestions and recommendations for improving the process of employee selection and acquirement.115 - Katedra managementuvýborn

Endemic level of carbapenem resistant <i>P</i>. <i>aeruginosa</i> bacteremia per 1000 patient-days from March 2009 to December 2012.

<p>Endemic level of carbapenem resistant <i>P</i>. <i>aeruginosa</i> bacteremia per 1000 patient-days from March 2009 to December 2012.</p

Characterization of resistance mechanisms in carbapenem-resistant <i>P</i>. <i>aeruginosa</i> isolates, both multidrug-resistant and extensively drug-resistant.

<p>Characterization of resistance mechanisms in carbapenem-resistant <i>P</i>. <i>aeruginosa</i> isolates, both multidrug-resistant and extensively drug-resistant.</p

Phenotypic and genotypic profile of resistance, hospital unit and the date of isolation of <i>P</i>. <i>aeruginosa</i> samples characterized by PFGE.

<p>Phenotypic and genotypic profile of resistance, hospital unit and the date of isolation of <i>P</i>. <i>aeruginosa</i> samples characterized by PFGE.</p

Characterization of efflux pumps overexpression, overproduction of AmpC and loss/reduction OprD in <i>P</i>. <i>aeruginosa</i> isolates.

<p>Characterization of efflux pumps overexpression, overproduction of AmpC and loss/reduction OprD in <i>P</i>. <i>aeruginosa</i> isolates.</p

<i>Pseudomonas aeruginosa</i> antimicrobial resistance and sensitivity profiles to the main antimicrobials of isolates from 242 episodes of hospital infections.

<p><i>Pseudomonas aeruginosa</i> antimicrobial resistance and sensitivity profiles to the main antimicrobials of isolates from 242 episodes of hospital infections.</p

Survival curve using the Kaplan–Meier method for patients who received appropriate antimicrobial therapy compared with those who received inappropriate therapy.

<p><i>P</i> ≤ 0.05 –statistically significant.</p

UPGMA dendrogram of PFGE profiles of 21 clinical <i>P</i>. <i>aeruginosa</i> isolates used in this study using the Dice coefficient under 1% tolerance and 1% optimization.

<p>A similarity coefficient of 80% was chosen for cluster definition. ¹Plasmid-mediated quinolone resistance determinants or aminoglycoside 6′-N-acetyltransferase type Ib; ²Metallo-β-lactamase; ³negative; ⁴not tested.</p

Characterization of <i>P</i>. <i>aeruginosa</i> isolates resistant to fluoroquinolones and/or carbapenems with regard to the minimum inhibitory concentration, <i>bla</i>_SPM, <i>bla</i>_VIM, <i>qnrS</i>, <i>aac(6´)-Ib-cr</i> and <i>aac(6´)-Ib</i>_<i>7</i> genes, quinolone resistance determining region mutations and the PFGE patterns.

<p>Characterization of <i>P</i>. <i>aeruginosa</i> isolates resistant to fluoroquinolones and/or carbapenems with regard to the minimum inhibitory concentration, <i>bla</i>_SPM, <i>bla</i>_VIM, <i>qnrS</i>, <i>aac(6´)-Ib-cr</i> and <i>aac(6´)-Ib</i>_<i>7</i> genes, quinolone resistance determining region mutations and the PFGE patterns.</p

Clinical and Molecular Epidemiology of Multidrug-Resistant <i>P</i>. <i>aeruginosa</i> Carrying <i>aac(6')-Ib-cr</i>, <i>qnrS1</i> and <i>bla</i>_SPM Genes in Brazil

<div><p>We described a comprehensive analysis of the molecular epidemiology of multidrug-resistant (MDR) <i>P</i>. <i>aeruginosa</i>. Molecular analysis included typing by Pulsed Field Gel Electrophoresis, identification of genes of interest through PCR-based assays and sequencing of target genes. Case-control study was conducted to better understand the prognostic of patients and the impact of inappropriate therapy in patients with bacteremia, as well as the risk factors of MDR infections. We observed a high rate of MDR isolates (40.7%), and 51.0% of them was independently associated with inappropriate antibiotic therapy. Bacteremia was detected in 66.9% of patients, and prolonged hospital stay was expressive in those resistant to fluoroquinolone. Plasmid-mediated quinolone resistance genes (PMQR), <i>qnrS</i>_<i>1</i> and <i>aac(6’)Ib-cr</i>, were detected in two different nosocomial isolates (5.3%), and the <i>aac(6’)-Ib</i>_<i>7</i> variant was detected at a high frequency (87.5%) in those negative to PMQR. The presence of mutations in <i>gyrA</i> and <i>parC</i> genes was observed in 100% and 85% of selected isolates, respectively. Isolates harboring PMQR genes or mutations in <i>gyrA</i> and <i>parC</i> were not closely related, except in those containing SPM (São Paulo metallo-β-lactamase) clone. In addition, there is no study published in Brazil to date reporting the presence of <i>Pseudomonas aeruginosa</i> isolates harboring both <i>qnrS</i>_<i>1</i> and <i>aac(6’)Ib-cr</i> genes, with alarming frequency of patients with inappropriate therapy.</p></div