235 research outputs found

    Reaching North: A Celebration of the Subarctic, by Jamie Bastedo

    Get PDF

    Wild Rivers, Wild Lands, by Ken Madsen

    Get PDF

    Injury patterns of South African international cricket players over a two-season period

    Get PDF
    Objective. The aim of the study was to determine the incidence and nature of injury patterns of South African international cricket players. Methods. A questionnaire was completed for each cricketer who presented with an injury during the 2004 - 2005 (S1) and 2005 - 2006 (S2) cricket seasons to determine the anatomical site, month, diagnosis and mechanism of injury. Results. The results showed that 113 injuries were sustained, with a match exposure time of 1 906 hours for one-day internationals (ODIs) and 5 070 hours for test matches. The injury prevalence was 4% per match, while the incidence of injury was 90 injuries per 10 000 hours of matches. Injuries occurred mostly to the lower limbs, back and trunk, upper limbs and head and neck. The injuries occurred primarily during test matches (43%), practices (20%) and practices and matches (19%). Acute injuries comprised 87% of the injuries. The major injuries during S1 were haematomas (20 %), muscle strains (14%) and other trauma (20%), while during S2 the injuries were primarily muscle strains (16%), other trauma (32%), tendinopathy (10%) and acute sprains (12%). The primary mechanisms of injury occurred when bowling (67%), on impact by the ball (batting – 65%, fielding – 26%) and when sliding for the ball (19%). Conclusion. The study provided prospective injury incidence and prevalence data for South African cricketers playing at international level over a two-season period, high-lighting the increased injury prevalence for away matches and an increased match injury incidence for test and ODI matches possibly as a result of increased match exposure time

    Progress towards the production of potatoes and cereals with low acrylamide-forming potential

    Get PDF
    The presence of acrylamide in foods derived from grains, tubers, storage roots, beans and other crop products has become a difficult problem for the food industry. Here we review how acrylamide is formed predominantly from free asparagine and reducing sugars, the relationship between precursor concentration and acrylamide formation, and the challenge of complying with increasingly stringent regulations. Progress made in reducing acrylamide levels in foods is assessed, along with the difficulty of dealing with a raw material that may be highly variable due to plant responses to nutrition, disease and cold storage. The potential for plant breeding and biotechnology to deliver low acrylamide varieties is assessed, in the context of a regulatory landscape covering acrylamide, crop biotechnology and crop protection

    Cereal asparagine synthetase genes

    Get PDF
    Asparagine synthetase catalyses the transfer of an amino group from glutamine to aspartate to form glutamate and asparagine. The accumulation of free (non-protein) asparagine in crops has implications for food safety because free asparagine is the precursor for acrylamide, a carcinogenic contaminant that forms during high-temperature cooking and processing. Here we review publicly-available genome data for asparagine synthetase genes from species of the Pooideae subfamily, including bread wheat and related wheat species (Triticum and Aegilops spp.), barley (Hordeum vulgare) and rye (Secale cereale) of the Triticeae tribe. Also from the Pooideae subfamily: brachypodium (Brachypodium dystachion) of the Brachypodiae tribe. More diverse species are also included, comprising sorghum (Sorghum bicolor) and maize (Zea mays) of the Panicoideae subfamily, and rice (Oryza sativa) of the Ehrhartoideae subfamily. The asparagine synthetase gene families of the Triticeae species each comprise five genes per genome, with the genes assigned to four groups: 1, 2, 3 (subdivided into 3.1 and 3.2) and 4. Each species has a single gene per genome in each group, except that some bread wheat varieties (genomes AABBDD) and emmer wheat (Triticum dicoccoides; genomes AABB) lack a group 2 gene in the B genome. This raises questions about the ancestry of cultivated pasta wheat and the B genome donor of bread wheat, suggesting that the hybridisation event that gave rise to hexaploid bread wheat occurred more than once. In phylogenetic analyses, genes from the other species cluster with the Triticeae genes, but brachypodium, sorghum and maize lack a group 2 gene, while rice has only two genes, one group 3 and one group 4. This means that TaASN2, the most highly expressed asparagine synthetase gene in wheat grain, has no equivalent in maize, rice, sorghum or brachypodium. An evolutionary pathway is proposed in which a series of gene duplications gave rise to the five genes found in modern Triticeae species
    • …
    corecore