142 research outputs found
Absence of spin superradiance in resonatorless magnets
A spin system is considered with a Hamiltonian typical of molecular magnets,
having dipole-dipole interactions and a single-site magnetic anisotropy. In
addition, spin interactions through the common radiation field are included. A
fully quantum-mechanical derivation of the collective radiation rate is
presented. An effective narrowing of the dipole-dipole attenuation, due to high
spin polarization is taken into account. The influence of the radiation rate on
spin dynamics is carefully analysed. It is shown that this influence is
completely negligible. No noticeable collective effects, such as superradiance,
can appear in molecular magnets, being caused by electromagnetic spin
radiation. Spin superradiance can arise in molecular magnets only when these
are coupled to a resonant electric circuit, as has been suggested earlier by
one of the authors in Laser Phys. {\bf 12}, 1089 (2002).Comment: Latex file, 14 pages, 5 figure
Thermal compression of atomic hydrogen on helium surface
We describe experiments with spin-polarized atomic hydrogen gas adsorbed on
liquid He surface. The surface gas density is increased locally by
thermal compression up to cm at 110 mK. This
corresponds to the onset of quantum degeneracy with the thermal de-Broglie
wavelength being 1.5 times larger than the mean interatomic spacing. The atoms
were detected directly with a 129 GHz electron-spin resonance spectrometer
probing both the surface and the bulk gas. This, and the simultaneous
measurement of the recombination power, allowed us to make accurate studies of
the adsorption isotherm and the heat removal from the adsorbed hydrogen gas.
From the data, we estimate the thermal contact between 2D hydrogen gas and
phonons of the helium film. We analyze the limitations of the thermal
compression method and the possibility to reach the superfluid transition in 2D
hydrogen gas.Comment: 20 pages, 11 figure
Vortex dynamics and upper critical fields in ultrathin Bi films
Current-voltage (I-V) characteristics of quench condensed, superconducting,
ultrathin films in a magnetic field are reported. These I-V's show
hysteresis for all films, grown both with and without thin underlayers.
Films on Ge underlayers, close to superconductor-insulator transition (SIT),
show a peak in the critical current, indicating a structural transformation of
the vortex solid (VS). These underlayers, used to make the films more
homogeneous, are found to be more effective in pinning the vortices. The upper
critical fields (B) of these films are determined from the resistive
transitions in perpendicular magnetic field. The temperature dependence of the
upper critical field is found to differ significantly from Ginzburg-Landau
theory, after modifications for disorder.Comment: Phys Rev B, to be published Figure 6 replaced with correct figur
Whole-genome sequencing reveals host factors underlying critical COVID-19
Critical COVID-19 is caused by immune-mediated inflammatory lung injury. Host genetic variation influences the development of illness requiring critical care1 or hospitalization2,3,4 after infection with SARS-CoV-2. The GenOMICC (Genetics of Mortality in Critical Care) study enables the comparison of genomes from individuals who are critically ill with those of population controls to find underlying disease mechanisms. Here we use whole-genome sequencing in 7,491 critically ill individuals compared with 48,400 controls to discover and replicate 23 independent variants that significantly predispose to critical COVID-19. We identify 16 new independent associations, including variants within genes that are involved in interferon signalling (IL10RB and PLSCR1), leucocyte differentiation (BCL11A) and blood-type antigen secretor status (FUT2). Using transcriptome-wide association and colocalization to infer the effect of gene expression on disease severity, we find evidence that implicates multiple genes—including reduced expression of a membrane flippase (ATP11A), and increased expression of a mucin (MUC1)—in critical disease. Mendelian randomization provides evidence in support of causal roles for myeloid cell adhesion molecules (SELE, ICAM5 and CD209) and the coagulation factor F8, all of which are potentially druggable targets. Our results are broadly consistent with a multi-component model of COVID-19 pathophysiology, in which at least two distinct mechanisms can predispose to life-threatening disease: failure to control viral replication; or an enhanced tendency towards pulmonary inflammation and intravascular coagulation. We show that comparison between cases of critical illness and population controls is highly efficient for the detection of therapeutically relevant mechanisms of disease
Comparison of optical appearance and infrared emission of some high latitude extended dust clouds
Wetensch. publicatieFaculteit der Wiskunde en Natuurwetenschappe
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