1,418 research outputs found
High domain wall velocities induced by current in ultrathin Pt/Co/AlOx wires with perpendicular magnetic anisotropy
Current-induced domain wall (DW) displacements in an array of ultrathin
Pt/Co/AlOx wires with perpendicular magnetic anisotropy have been directly
observed by wide field Kerr microscopy. DWs in all wires in the array were
driven simultaneously and their displacement on the micrometer-scale was
controlled by the current pulse amplitude and duration. At the lower current
densities where DW displacements were observed (j less than or equal to 1.5 x
10^12 A/m^2), the DW motion obeys a creep law. At higher current density (j =
1.8 x 10^12 A/m^2), zero-field average DW velocities up to 130 +/- 10 m/s were
recorded.Comment: Minor changes to Fig. 1(b) and text, correcting for the fact that
domain walls were subsequently found to move counter to the electron flow.
References update
Interplay between magnetic anisotropy and interlayer coupling in nanosecond magnetization reversal of spin-valve trilayers
The influence of magnetic anisotropy on nanosecond magnetization reversal in
coupled FeNi/Cu/Co trilayers was studied using a photoelectron emission
microscope combined with x-ray magnetic circular dicroism. In quasi-isotropic
samples the reversal of the soft FeNi layer is determined by domain wall
pinning that leads to the formation of small and irregular domains. In samples
with uniaxial magnetic anisotropy, the domains are larger and the influence of
local interlayer coupling dominates the domain structure and the reversal of
the FeNi layer
Dynamics of magnetic domain wall motion after nucleation: Dependence on the wall energy
The dynamics of magnetic domain wall motion in the FeNi layer of a
FeNi/Al2O3/Co trilayer has been investigated by a combination of x-ray magnetic
circular dichroism, photoelectron emission microscopy, and a stroboscopic
pump-probe technique. The nucleation of domains and subsequent expansion by
domain wall motion in the FeNi layer during nanosecond-long magnetic field
pulses was observed in the viscous regime up to the Walker limit field. We
attribute an observed delay of domain expansion to the influence of the domain
wall energy that acts against the domain expansion and that plays an important
role when domains are small.Comment: Accepted for publication in Physical Review Letter
Domain wall tilting in the presence of the Dzyaloshinskii-Moriya interaction in out-of-plane magnetized magnetic nanotracks
We show that the Dzyaloshinskii-Moriya interaction (DMI) can lead to a
tilting of the domain wall (DW) surface in perpendicularly magnetized magnetic
nanotracks when DW dynamics is driven by an easy axis magnetic field or a spin
polarized current. The DW tilting affects the DW dynamics for large DMI and the
tilting relaxation time can be very large as it scales with the square of the
track width. The results are well explained by an analytical model based on a
Lagrangian approach where the DMI and the DW tilting are included. We propose a
simple way to estimate the DMI in a magnetic multilayers by measuring the
dependence of the DW tilt angle on a transverse static magnetic field. Our
results shed light on the current induced DW tilting observed recently in Co/Ni
multilayers with inversion asymmetry, and further support the presence of DMI
in these systems.Comment: 12 pages, 3 figures, 1 Supplementary Material
Influence of topography and Co domain walls on the magnetization reversal of the FeNi layer in FeNi/AlO/Co magnetic tunnel junctions
We have studied the magnetization reversal dynamics of FeNi/AlO/Co
magnetic tunnel junctions deposited on step-bunched Si substrates using
magneto-optical Kerr effect and time-resolved x-ray photoelectron emission
microscopy combined with x-ray magnetic circular dichroism (XMCD-PEEM).
Different reversal mechanisms have been found depending on the substrate miscut
angle. Larger terraces (smaller miscut angles) lead to a higher nucleation
density and stronger domain wall pinning. The width of domain walls with
respect to the size of the terraces seems to play an important role in the
reversal. We used the element selectivity of XMCD-PEEM to reveal the strong
influence of the stray field of domain walls in the hard magnetic layer on the
magnetic switching of the soft magnetic layer.Comment: 8 Pages, 7 Figure
Electric-field control of domain wall nucleation and pinning in a metallic ferromagnet
The electric (E) field control of magnetic properties opens the prospects of
an alternative to magnetic field or electric current activation to control
magnetization. Multilayers with perpendicular magnetic anisotropy (PMA) have
proven to be particularly sensitive to the influence of an E-field due to the
interfacial origin of their anisotropy. In these systems, E-field effects have
been recently applied to assist magnetization switching and control domain wall
(DW) velocity. Here we report on two new applications of the E-field in a
similar material : controlling DW nucleation and stopping DW propagation at the
edge of the electrode
Association between Resistin Levels and All-Cause and Cardiovascular Mortality: A New Study and a Systematic Review and Meta-Analysis.
CONTEXT: Studies concerning the association between circulating resistin and mortality risk have reported, so far, conflicting results.
OBJECTIVE: To investigate the association between resistin and both all-cause and cardiovascular (CV) mortality risk by 1) analyzing data from the Gargano Heart Study (GHS) prospective design (n=359 patients; 81 and 58 all-cause and CV deaths, respectively); 2) performing meta-analyses of all published studies addressing the above mentioned associations.
DATA SOURCE AND STUDY SELECTION: MEDLINE and Web of Science search of studies reporting hazard ratios (HR) of circulating resistin for all-cause or CV mortality.
DATA EXTRACTION: Performed independently by two investigators, using a standardized data extraction sheet.
DATA SYNTHESIS: In GHS, adjusted HRs per one standard deviation (SD) increment in resistin concentration were 1.28 (95% CI: 1.07-1.54) and 1.32 (95% CI: 1.06-1.64) for all-cause and CV mortality, respectively. The meta-analyses included 7 studies (n=4016; 961 events) for all-cause mortality and 6 studies (n=4,187: 412 events) for CV mortality. Pooled HRs per one SD increment in resistin levels were 1.21 (95% CI: 1.03-1.42, Q-test p for heterogeneity<0.001) and 1.05 (95% CI: 1.01-1.10, Q-test p for heterogeneity=0.199) for all-cause and CV mortality, respectively. At meta-regression analyses, study mean age explained 9.9% of all-cause mortality studies heterogeneity. After adjusting for age, HR for all-cause mortality was 1.24 (95% CI: 1.06-1.45).
CONCLUSIONS: Our results provide evidence for an association between circulating resistin and mortality risk among high-risk patients as are those with diabetes and coronary artery disease
5-En-androstene-3 beta,17 beta-diol inhibits the growth of MCF-7 breast cancer cells when oestrogen receptors are blocked by oestradiol.
Adrenal androgens show a dual and apparently opposite effect on the growth of oestrogen-responsive breast cancer: they stimulate growth on their own, but counteract the growth-stimulatory effect of oestrogens. Focusing on the inhibitory action we have studied the effects of 5-en-androstene-3 beta,17 beta-diol (ADIOL) on the growth of oestrogen-responsive MCF-7 breast cancer cells in the presence of oestrogens (oestradiol and diethylstilboestrol), antiestrogens (tamoxifen) and antiandrogens (hydroxyflutamide). The inhibition of oestrogen-stimulated growth, attained with nanomolar concentrations of ADIOL, was not modified by increasing concentrations of diethylstilboestrol up to 100 nM. This inhibition was counteracted by antiandrogens, which were unable to block the ADIOL stimulatory effect in steroid-free medium. On the other hand, in the presence of tamoxifen ADIOL showed an additive antiproliferative activity also in steroid-free medium, rather than the usual stimulatory effect. These results suggest that ADIOL stimulates breast cancer cell growth via oestrogen receptors, but inhibits oestrogen-stimulated growth via androgen receptors
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