The developmental course of illicit substance use from age 12 to 22: links with depressive, anxiety, and behavior disorders at age 18

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/72027/1/j.1469-7610.2008.01915.x.pd

Classes of Intimate Partner Violence from Late Adolescence to Young Adulthood

Researchers do not agree on how intimate partner violence (IPV) emerges and changes from adolescence to young adulthood. This may be because change in these behaviors varies across individuals. The present study uses a longitudinal, person-centered approach to examine whether there are multiple classes or patterns of change in the perpetration of IPV during the transitional period from adolescence (age 18) to young adulthood (age 25) using data collected annually from a community sample of 484 participants. Latent class analysis was the analytic approach used. Results revealed three patterns for psychological IPV (Little-to-None, Minor/Increasing, and Extensive/Increasing) and two patterns for physical IPV (Little-to-None and Extensive). Patterns varied greatly in number of representatives, although they were more balanced in size for psychological than physical IPV. Variations in IPV behaviors were also revealed across classes, although as expected in a community sample, minor forms of IPV were more common than severe forms. In addition, classes differed in demographic and relationship status variables. These findings suggest that IPV may occur in multiple distinct patterns as opposed to one average pattern across a population. This suggests that interventions for IPV may need to be geared to differences in patterns to enhance their efficacy

Developmental mediation of genetic variation in response to the Fast Track Prevention Program

We conducted a developmental analysis of genetic moderation of the effect of the Fast Track intervention on adult externalizing psychopathology. The Fast Track intervention enrolled 891 children at high risk to develop externalizing behavior problems when they were in kindergarten. Half of the enrolled children were randomly assigned to receive 10 years of treatment, with a range of services and resources provided to the children and their families, and the other half to usual care (controls). We previously showed that the effect of the Fast Track intervention on participants\u27 risk of externalizing psychopathology at age 25 years was moderated by a variant in the glucocorticoid receptor gene. Children who carried copies of the A allele of the single nucleotide polymorphism rs10482672 had the highest risk of externalizing psychopathology if they were in the control arm of the trial and the lowest risk of externalizing psychopathology if they were in the treatment arm. In this study, we test a developmental hypothesis about the origins of this for better and for worse Gene × Intervention interaction (G × I): that the observed G × I effect on adult psychopathology is mediated by the proximal impact of intervention on childhood externalizing problems and adolescent substance use and delinquency. We analyzed longitudinal data tracking the 270 European American children in the Fast Track randomized control trial with available genetic information (129 intervention children, 141 control group peers, 69% male) from kindergarten through age 25 years. Results show that the same pattern of for better and for worse susceptibility to intervention observed at the age 25 follow-up was evident already during childhood. At the elementary school follow-ups and at the middle/high school follow-ups, rs10482672 predicted better adjustment among children receiving the Fast Track intervention and worse adjustment among children in the control condition. In turn, these proximal G × I effects early in development mediated the ultimate G × I effect on externalizing psychopathology at age 25 years. We discuss the contribution of these findings to the growing literature on genetic susceptibility to environmental intervention

Developmental mediation of genetic variation in response to the Fast Track Prevention Program

We conducted a developmental analysis of genetic moderation of the effect of the Fast Track intervention on adult externalizing psychopathology. The Fast Track intervention enrolled 891 children at high risk to develop externalizing behavior problems when they were in kindergarten. Half of the enrolled children were randomly assigned to receive 10 years of treatment, with a range of services and resources provided to the children and their families, and the other half to usual care (controls). We previously showed that the effect of the Fast Track intervention on participants\u27 risk of externalizing psychopathology at age 25 years was moderated by a variant in the glucocorticoid receptor gene. Children who carried copies of the A allele of the single nucleotide polymorphism rs10482672 had the highest risk of externalizing psychopathology if they were in the control arm of the trial and the lowest risk of externalizing psychopathology if they were in the treatment arm. In this study, we test a developmental hypothesis about the origins of this for better and for worse Gene × Intervention interaction (G × I): that the observed G × I effect on adult psychopathology is mediated by the proximal impact of intervention on childhood externalizing problems and adolescent substance use and delinquency. We analyzed longitudinal data tracking the 270 European American children in the Fast Track randomized control trial with available genetic information (129 intervention children, 141 control group peers, 69% male) from kindergarten through age 25 years. Results show that the same pattern of for better and for worse susceptibility to intervention observed at the age 25 follow-up was evident already during childhood. At the elementary school follow-ups and at the middle/high school follow-ups, rs10482672 predicted better adjustment among children receiving the Fast Track intervention and worse adjustment among children in the control condition. In turn, these proximal G × I effects early in development mediated the ultimate G × I effect on externalizing psychopathology at age 25 years. We discuss the contribution of these findings to the growing literature on genetic susceptibility to environmental intervention

Temporal Associations between Psychological and Physical Intimate Partner Violence: A Cross-Lag Analysis

The present study examined the relationship between psychological and physical forms of intimate partner violence (IPV) across four waves of data during the developmental period of young adulthood. The links from early psychological aggression to later physical aggression and from early physical aggression to later psychological aggression across waves were tested while controlling for their cross-time stabilities and concurrent associations. IPV data were collected annually from 434 young adult respondents involved in a romantic relationship at least once during the respective years from the ages of 22-25. On average, participants provided IPV data for three out of the four years covered by the study (M = 2.82; SD = 1.14). Results of a cross-lagged SEM model indicated significant cross-time stabilities as well as significant, positive concurrent associations for both forms of aggression. Most important to this study were the findings that, controlling for these stabilities and concurrent associations, early psychological aggression was a consistent positive predictor of later physical aggression across waves whereas the opposite direction from early physical aggression to later psychological aggression was either non-significant or significant and negative

Predicting Patterns of Intimate Partner Violence Perpetration from Late Adolescence to Young Adulthood

Saint-Eloi Cadely et al. (2017) found longitudinal patterns for the perpetration of both psychological and physical intimate partner violence (IPV), including actively and minimally aggressive patterns. The current study builds on these findings by examining four theory-derived variables (interparental aggression, social-information processing (SIP) biases, relationship insecurities (preoccupied and fearful), and discontinuity in relationship partner over time) as predictors of membership within these patterns using multinomial logistic regression. The analysis sample consisted of 484 participants who were romantically involved at least once during the 8 waves of data collection from the ages of 18-25. In predicting psychological IPV, more SIP biases, higher levels of a preoccupied insecurity, and less discontinuity in relationship partners over time differentiated the actively aggressive patterns from the minimally aggressive pattern. Additionally, two actively aggressive patterns of psychological IPV differed in terms of SIP biases and discontinuity in romantic partners. Specifically, more SIP biases and less discontinuity in romantic partnerships distinguished the extensively aggressive pattern from the pattern that mainly consisted of minor types of aggression. In predicting physical IPV, the aggressive pattern differed from the non-aggressive pattern in terms of more interparental aggression, more SIP biases and more relationship insecurities. The findings that developmental patterns of IPV can be predicted by social and psychological factors may aid both developmental theory and practice

Country, sex, and parent occupational status: Moderators of the continuity of aggression from childhood to adulthood

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/109352/1/ab21546.pd

CATS II long-term anthropometric and metabolic effects of maternal sub-optimal thyroid function in offspring and mothers

Context and Objectives The Controlled Antenatal Thyroid Screening Study I (CATS-I) was a randomized controlled trial investigating the effects of levothyroxine therapy for suboptimal gestational thyroid function (SGTF), comparing outcomes in children of treated (SGTF-T) with untreated (SGTF-U) women during pregnancy. This follow-up study, CATS-II, reports the long-term effects on anthropometric, bone, and cardiometabolic outcomes in mothers and offspring and includes a group with normal gestational thyroid function (NGTF). Design & Participants 332 mothers (197 NGTF, 56 SGTF-U, 79 SGTF-T) aged 41.2±5.3 years (mean±SD) and 326 paired children assessed 9.3±1.0 years after birth for (i) body mass index (BMI); (ii) lean, fat, and bone mass by dual-energy X-ray absorptiometry; (iii) blood pressure, augmentation index, and aortic pulse-wave-velocity; and (iv) thyroid function, lipids, insulin, and adiponectin. The difference between group means was compared using linear regression. Results Offspring’s measurements were similar between groups. Although maternal BMI was similar between groups at CATS-I, after 9 years (at CATS-II) SGTF-U mothers showed higher BMI (median [interquartile ratio] 28.3 [24.6-32.6] kg/m2) compared with NGTF (25.8 [22.9-30.0] kg/m2; P = 0.029), driven by fat mass increase. At CATS-II SGTF-U mothers also had higher thyroid-stimulating hormone (TSH) values (2.45 [1.43-3.50] mU/L) than NGTF (1.54 [1.12-2.07] mU/L; P = 0.015), since 64% had never received levothyroxine. At CATS-II, SGTF-T mothers had BMI (25.8 [23.1-29.8] kg/m2, P = 0.672) and TSH (1.68 [0.89-2.96] mU/L; P = 0.474) values similar to NGTF mothers. Conclusions Levothyroxine supplementation of women with SGTF did not affect long-term offspring anthropometric, bone, and cardiometabolic measurements. However, absence of treatment was associated with sustained long-term increase in BMI and fat mass in women with SGTF

Parental agreement of reporting parent to child aggression using the Conflict Tactics Scales

Objectives: This study examined mothers’ and fathers’ reporting congruency using the Parent–Child Conflict Tactics Scales. We asked if the mother’s report of the father’s parenting aggression was consistent with the father’s self-report of parenting aggression and if the father’s report of the mother’s parenting aggression was consistent with the mother’s selfreport of those same behaviors. We assessed moderators of parental reporting congruency: severity of the aggression, interparental conflict, child temperament, and child gender. Methods: Participants were from the Child Development Project, a longitudinal study beginning when children were in kindergarten. The analyses herein included 163 children for whom 2 parents provided data about their own and their spouse or partner’s behavior toward the child. Most parents (87%) were married. Mothers and fathers independently completed the Parent–Child Conflict Tactics Scale, both with respect to their own behavior toward the child and with respect to their partner’s behavior toward the child. Mothers completed the retrospective Infant Characteristics Questionnaire to assess child temperament. Mothers and fathers completed measures of interparental conflict. Results: Both fathers and mothers self-reported more frequently engaging in each behavior than the other parent reported they did. Parents were more congruent on items assessing harsher parenting behavior. Furthermore, there was more agreement between parents regarding fathers’ behavior than mothers’ behavior. Analyses of interparental conflict, child difficult temperament, and child gender as moderators yielded findings suggesting that mothers’ and fathers’ reports of their own and their spouses’ harsh parenting behaviors were more concordant in couples with low levels of conflict, for children with easy temperaments, and for boys versus girls. Conclusions: Prior studies indicate only a moderate level of agreement in couples’ reports of violence between intimate partners and suggest that perpetrators tend to underreport their use of aggression. The results of this study suggest that parents may be more consistent in their reports of parent to child violence using the Parent–Child Conflict Tactics Scales than they are when reporting intimate partner violence. The results suggest that parental reports of their spouse’s parent to child aggression are reliable.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/106161/1/2012-Lee Lansford et al CTSPC.pd