Vulnerabilidad en dispositivos Bluetooth

El objetivo de este proyecto es el estudio de las posibles vulnerabilidades que puede tener un teléfono móvil por medio de la tecnología Bluetooth, además del estudio de viabilidad de una herramienta que ayude a vigilar el “medio” Bluetooth para evitar un mal uso del mismo. Hoy en día hay herramientas que realizan esta labor en otro tipo de redes, son los husmeadores o sniffers. Este proyecto abordará el estudio de si se podría realizar un sniffer para Bluetooth sobre un dispositivo móvil que contenga un sistema operativo Symbian. [ABSTRACT] The objective of the project is research about the bad use of the Bluetooth technology, and the research of the viability about any tool that help us to control the Bluetooth technology allowing us to check the right use of this technology. At the moment, there are several tools that make this job in other kind of net (sniffers). This project will tackle the viability about the creation of a sniffer to the Bluetooth, always into the Symbian environment

Experimental verification of hybrid renewable systems as feasible energy sources

[EN] Renewable energies are a central element in the search for energy sustainability, so they are becoming a substantial component of the energy scenario of every country, both as systems connected to the grid or in stand-alone applications. Feasibility of these renewable energy systems could be necessary not only in their application in isolated areas, but also in systems connected to the grid, in this last case when their contribution reaches a substantial fraction of the total electricity demand. To overcome this reliability problem, hybrid renewable systems could become essential and activities to optimize their design should be addressed, both in the simulation and in the experimental areas. In this paper, a laboratory to simulate and verify the reliability of hybrid renewable systems is presented and its application to the feasibility analysis of multicomponent systems including photovoltaic panels, wind generator and biomass gasification plant, plus energy storage in a battery bank, are described.Pérez-Navarro, Á.; Alfonso-Solar, D.; Ariza-Chacón, HE.; Cárcel Carrasco, FJ.; Correcher Salvador, A.; Escrivá-Escrivá, G.; Hurtado, E.... (2016). Experimental verification of hybrid renewable systems as feasible energy sources. Renewable Energy. 86(2):384-391. doi:10.1016/j.renene.2015.08.030S38439186

40 years of surgical advances

Changes in surgery over the last 40 years have been extraordinary. We want to reflect the progress of this period in the District Hospital of Alcoy, Spain. Technology and staffing have improved in an extraordinary way and allows a basic hospital to offer citizens the quality of the most sophisticated centers

JAK2 mediates lung fibrosis, pulmonary vascular remodelling and hypertension in idiopathic pulmonary fibrosis: an experimental study

Background Pulmonary hypertension (PH) is a common disorder in patients with idiopathic pulmonary fibrosis (IPF) and portends a poor prognosis. Recent studies using vasodilators approved for PH have failed in improving IPF mainly due to ventilation (V)/perfusion (Q) mismatching and oxygen desaturation. Janus kinase type 2 (JAK2) is a non-receptor tyrosine kinase activated by a broad spectrum of profibrotic and vasoactive mediators, but its role in PH associated to PH is unknown. Objective The study of JAK2 as potential target to treat PH in IPF. Methods and results JAK2 expression was increased in pulmonary arteries (PAs) from IPF (n=10; 1.93-fold; P=0.0011) and IPF+PH (n=9; 2.65-fold; P<0.0001) compared with PA from control subjects (n=10). PA remodelling was evaluated in human pulmonary artery endothelial cells (HPAECs) and human pulmonary artery smooth muscle cells (HPASMCs) from patients with IPF in vitro treated with the JAK2 inhibitor JSI-124 or siRNA-JAK2 and stimulated with transforming growth factor beta. Both JSI-124 and siRNA-JAK2 inhibited the HPAEC to mesenchymal transition and the HPASMCs to myofibroblast transition and proliferation. JAK2 inhibition induced small PA relaxation in precision-cut lung slice experiments. PA relaxation was dependent of the large conductance calcium-activated potassium channel (BKCa). JAK2 inhibition activated BKCa channels and reduced intracellular Ca2+. JSI-124 1 mg/kg/day, reduced bleomycin-induced lung fibrosis, PA remodelling, right ventricular hypertrophy, PA hypertension and V/Q mismatching in rats. The animal studies followed the ARRIVE guidelines. Conclusions JAK2 participates in PA remodelling and tension and may be an attractive target to treat IPF associated to PH