‘Are You Fed up with Your PC? Get A Mac.’ Swedish Popular Media Representations of Digital Technologies and the Stockpiling Behavior of Consumers

This article offers a speculative reading on the linkage between Swedish popular media representations of digital technologies and peoples’ stockpiling behavior. The hypothesis is that Swedish popular media representations of digital technologies contribute to fuelling an understanding of these technologies as resource-free, consumable goods, and that this in turn might affect peoples’ tendencies to stockpile obsolete digital technologies in their homes. Previous studies have shown that stockpiling of obsolete electrical and electronic equipment is a common phenomenon in many countries (see for comparison Grossman, 2006; Saphores et al., 2006, 2009; Lescak, 2008; Gabrys, 2011). At the same time, little light is shed on why people stockpile obsolete electrical and electronic equipment in their homes. This article focuses particularly on digital technologies such as computers, televisions, smart phones, cell phones, laptops and tablets. Digital technologies are interesting as they account for a large – and increasing – amount of electrical and electronic equipment. Looking at the ways in which Swedish popular media represent digital technologies, this article suggests that popular media representations of digital technologies matter, not only in the ways people imagine and use digital technologies, but also the ways in which we think (or choose not to think) about their afterlife. The hypothesis is that the ways in which Swedish popular media represent digital technologies obstruct environmentally sound disposal and reuse options, something that in turn serves to cement and/or reinforce contemporary consumption behaviors and the environmental impacts that follow. By this, this article provides additional insights into what appears to be a western tendency to store obsolete electrical and electronic equipment at home

Altered plasma protein profiles in genetic FTD – a GENFI study

Background: Plasma biomarkers reflecting the pathology of frontotemporal dementia would add significant value to clinical practice, to the design and implementation of treatment trials as well as our understanding of disease mechanisms. The aim of this study was to explore the levels of multiple plasma proteins in individuals from families with genetic frontotemporal dementia. Methods: Blood samples from 693 participants in the GENetic Frontotemporal Dementia Initiative study were analysed using a multiplexed antibody array targeting 158 proteins. Results: We found 13 elevated proteins in symptomatic mutation carriers, when comparing plasma levels from people diagnosed with genetic FTD to healthy non-mutation controls and 10 proteins that were elevated compared to presymptomatic mutation carriers. Conclusion: We identified plasma proteins with altered levels in symptomatic mutation carriers compared to non-carrier controls as well as to presymptomatic mutation carriers. Further investigations are needed to elucidate their potential as fluid biomarkers of the disease process.</p

Altered plasma protein profiles in genetic FTD - a GENFI study

BACKGROUND: Plasma biomarkers reflecting the pathology of frontotemporal dementia would add significant value to clinical practice, to the design and implementation of treatment trials as well as our understanding of disease mechanisms. The aim of this study was to explore the levels of multiple plasma proteins in individuals from families with genetic frontotemporal dementia. METHODS: Blood samples from 693 participants in the GENetic Frontotemporal Dementia Initiative study were analysed using a multiplexed antibody array targeting 158 proteins. RESULTS: We found 13 elevated proteins in symptomatic mutation carriers, when comparing plasma levels from people diagnosed with genetic FTD to healthy non-mutation controls and 10 proteins that were elevated compared to presymptomatic mutation carriers. CONCLUSION: We identified plasma proteins with altered levels in symptomatic mutation carriers compared to non-carrier controls as well as to presymptomatic mutation carriers. Further investigations are needed to elucidate their potential as fluid biomarkers of the disease process

Altered plasma protein profiles in genetic FTD – a GENFI study

© The Author(s) 2023. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.Background: Plasma biomarkers reflecting the pathology of frontotemporal dementia would add significant value to clinical practice, to the design and implementation of treatment trials as well as our understanding of disease mechanisms. The aim of this study was to explore the levels of multiple plasma proteins in individuals from families with genetic frontotemporal dementia. Methods: Blood samples from 693 participants in the GENetic Frontotemporal Dementia Initiative study were analysed using a multiplexed antibody array targeting 158 proteins. Results: We found 13 elevated proteins in symptomatic mutation carriers, when comparing plasma levels from people diagnosed with genetic FTD to healthy non-mutation controls and 10 proteins that were elevated compared to presymptomatic mutation carriers. Conclusion: We identified plasma proteins with altered levels in symptomatic mutation carriers compared to non-carrier controls as well as to presymptomatic mutation carriers. Further investigations are needed to elucidate their potential as fluid biomarkers of the disease process.Open access funding provided by Karolinska Institute. C.G. received funding from EU Joint Programme—Neurodegenerative Disease Research -Prefrontals Vetenskapsrådet Dnr 529–2014-7504, Vetenskapsrådet 2015–02926, Vetenskapsrådet 2018–02754, the Swedish FTD Inititative-Schörling Foundation, Alzheimer Foundation, Brain Foundation, Dementia Foundation and Region Stockholm ALF-project. PN received funding from KTH Center for Applied Precision Medicine (KCAP) funded by the Erling-Persson Family Foundation, the Swedish FTD Inititative-Schörling Foundation and Åhlén foundation. D.G. received support from the EU Joint Programme—Neurodegenerative Disease Research and the Italian Ministry of Health (PreFrontALS) grant 733051042. E.F. has received funding from a Canadian Institute of Health Research grant #327387. F.M. received funding from the Tau Consortium and the Center for Networked Biomedical Research on Neurodegenerative Disease. J.B.R. has received funding from the Welcome Trust (103838) and is supported by the Cambridge University Centre for Frontotemporal Dementia, the Medical Research Council (SUAG/051 G101400) and the National Institute for Health Research Cambridge Biomedical Research Centre (BRC-1215–20014). J.C.V.S. was supported by the Dioraphte Foundation grant 09–02-03–00, Association for Frontotemporal Dementias Research Grant 2009, Netherlands Organization for Scientific Research grant HCMI 056–13-018, ZonMw Memorabel (Deltaplan Dementie, project number 733 051 042), Alzheimer Nederland and the Bluefield Project. J.D.R. is supported by the Bluefield Project and the National Institute for Health and Care Research University College London Hospitals Biomedical Research Centre, and has received funding from an MRC Clinician Scientist Fellowship (MR/M008525/1) and a Miriam Marks Brain Research UK Senior Fellowship. M.M. has received funding from a Canadian Institute of Health Research operating grant and the Weston Brain Institute and Ontario Brain Institute. M.O. has received funding from Germany’s Federal Ministry of Education and Research (BMBF). R.S-V. is supported by Alzheimer’s Research UK Clinical Research Training Fellowship (ARUK-CRF2017B-2) and has received funding from Fundació Marató de TV3, Spain (grant no. 20143810). R.V. has received funding from the Mady Browaeys Fund for Research into Frontotemporal Dementia. This work was also supported by the EU Joint Programme—Neurodegenerative Disease Research GENFI-PROX grant [2019–02248; to J.D.R., M.O., B.B., C.G., J.C.V.S. and M.S.info:eu-repo/semantics/publishedVersio

Taking place - augmenting space : spatial diffusion in times of technological change

Godkänd; 2010; 20100907 (jenolo); DISPUTATION Ämnesområde: Genus och teknik/Gender and Technology Opponent: Professor Trevor Pinch, Cornell University, New York, USA Ordförande: Professor Ulf Mellström, Luleå tekniska universitet Tid: Fredag den 10 december 2010, kl 10.00 Plats: F341, Luleå tekniska universite

It is unmanly to drive using the first gear? : driving as an embodied performance in multiple forms

Godkänd; 2009; 20090914 (andbra

Negotiating figurations for feminist methodologies : a manifest for the fl@neur

Validerad; 2008; 20080811 (jenolo

Negotiating figurations for feminist methodologies : a manifest for the fl@neur

Validerad; 2008; 20080811 (jenolo

Aktivitet: International Workshop on Gender &amp; Technology

Evenemang (konferens etc): International Workshop on Gender &amp; Technology : New Theoretical Perspectives 20/06/2011 - 21/06/2011; Startdatum: 20/06/2011; Slutdatum: 21/06/2011; Roll: Deltagare; Typ: Organiserade av och deltagande i konferenser, workshops, kurser, seminarier - Deltagande i workshop, seminarium, kurs</p

A computer is a screen, is a cell phone, is plastics, is a microwave, is a copper coil… : On the fluidity of electronic waste

Informed by previous studies that employ Actor Network-Theory (ANT), this article discusses electronic waste (e-waste) as fluid objects. The concept of ‘fluidity’ or ‘fluid’ includes an object’s ability to flow and thereby change shape, and therefore obstructs any attempt to explain e-waste in terms of a single state of being. Traditionally, fluidity has been used as a means to capture the changing characters of objects while they are being used. Elaborating on the notion of fluidity or the fluid in relation to e-waste serves both to expand previous understandings of fluidity to also encompass the status of objects as they are discarded, and to challenge traditional understandings of e-waste as the end point in the object’s lifecycle.Mobile trash issue</p