Time-dependent approach to many-particle tunneling in one-dimension

Employing the time-dependent approach, we investigate a quantum tunneling decay of many-particle systems. We apply it to a one-dimensional three-body problem with a heavy core nucleus and two valence protons. We calculate the decay width for two-proton emission from the survival probability, which well obeys the exponential decay-law after a sufficient time. The effect of the correlation between the two emitted protons is also studied by observing the time evolution of the two-particle density distribution. It is shown that the pairing correlation significantly enhances the probability for the simultaneous diproton decay.Comment: 9 pages, 10 eps figure

Modification of Angular Velocity by Inhomogeneous MRI Growth in Protoplanetary Disks

We have investigated evolution of magneto-rotational instability (MRI) in protoplanetary disks that have radially non-uniform magnetic field such that stable and unstable regions coexist initially, and found that a zone in which the disk gas rotates with a super-Keplerian velocity emerges as a result of the non-uniformly growing MRI turbulence. We have carried out two-dimensional resistive MHD simulations with a shearing box model. We found that if the spatially averaged magnetic Reynolds number, which is determined by widths of the stable and unstable regions in the initial conditions and values of the resistivity, is smaller than unity, the original Keplerian shear flow is transformed to the quasi-steady flow such that more flattened (rigid-rotation in extreme cases) velocity profile emerges locally and the outer part of the profile tends to be super-Keplerian. Angular momentum and mass transfer due to temporally generated MRI turbulence in the initially unstable region is responsible for the transformation. In the local super-Keplerian region, migrations due to aerodynamic gas drag and tidal interaction with disk gas are reversed. The simulation setting corresponds to the regions near the outer and inner edges of a global MRI dead zone in a disk. Therefore, the outer edge of dead zone, as well as the inner edge, would be a favorable site to accumulate dust particles to form planetesimals and retain planetary embryos against type I migration.Comment: 28 pages, 11figures, 1 table, accepted by Ap

Pressure-induced unconventional superconductivity in the heavy-fermion antiferromagnet CeIn3: An 115In-NQR study under pressure

We report on the pressure-induced unconventional superconductivity in the heavy-fermion antiferromagnet CeIn3 by means of nuclear-quadrupole-resonance (NQR) studies conducted under a high pressure. The temperature and pressure dependences of the NQR spectra have revealed a first-order quantum-phase transition (QPT) from an AFM to PM at a critical pressure Pc=2.46 GPa. Despite the lack of an AFM quantum critical point in the P-T phase diagram, we highlight the fact that the unconventional SC occurs in both phases of the AFM and PM. The nuclear spin-lattice relaxation rate 1/T1 in the AFM phase have provided evidence for the uniformly coexisting AFM+SC phase. In the HF-PM phase where AFM fluctuations are not developed, 1/T1 decreases without the coherence peak just below Tc, followed by a power-law like T dependence that indicates an unconventional SC with a line-node gap. Remarkably, Tc has a peak around Pc in the HF-PM phase as well as in the AFM phase. In other words, an SC dome exists with a maximum value of Tc = 230 mK around Pc, indicating that the origin of the pressure-induced HF SC in CeIn3 is not relevant to AFM spin fluctuations but to the emergence of the first-order QPT in CeIn3. When the AFM critical temperature is suppressed at the termination point of the first-order QPT, Pc = 2.46 GPa, the diverging AFM spin-density fluctuations emerge at the critical point from the AFM to PM. The results with CeIn3 leading to a new type of quantum criticality deserve further theoretical investigations

The deleted in brachydactyly B domain of ROR2 is required for receptor activation by recruitment of Src

The transmembrane receptor 'ROR2' resembles members of the receptor tyrosine kinase family of signalling receptors in sequence but its' signal transduction mechanisms remain enigmatic. This problem has particular importance because mutations in ROR2 are associated with two human skeletal dysmorphology syndromes, recessive Robinow Syndrome (RS) and dominant acting Brachydactyly type B (BDB). Here we show, using a constitutive dimerisation approach, that ROR2 exhibits dimerisation-induced tyrosine kinase activity and the ROR2 C-terminal domain, which is deleted in BDB, is required for recruitment and activation of the non-receptor tyrosine kinase Src. Native ROR2 phosphorylation is induced by the ligand Wnt5a and is blocked by pharmacological inhibition of Src kinase activity. Eight sites of Src-mediated ROR2 phosphorylation have been identified by mass spectrometry. Activation via tyrosine phosphorylation of ROR2 receptor leads to its internalisation into Rab5 positive endosomes. These findings show that BDB mutant receptors are defective in kinase activation as a result of failure to recruit Src