Multi-generational House

BUTOROVÁ, H.: Dvougenerační rodinný dům: Bakalářská práce. Ostrava: VŠB-Technická univerzita Ostrava, Fakulta stavební, Katedra architektury 226, 2017, 49 s. Vedoucí práce: Student, A. Předmětem bakalářské práce „Dvougenerační rodinný dům“ je vypracování částečné projektové dokumentace pro provádění stavby podle vyhlášky 499/2006 Sb., o dokumentaci staveb. Jako podklad bakalářské práce slouží architektonická studie vypracovaná v rámci předmětu Ateliérová tvorba I a dokumentace pro stavební povolení vypracovaná v předmětu Ateliérová tvorba Va. Rodinný dvougenerační dům je navržen v lázeňské oblasti Karviná-Darkov. Stavba je složena z části pro mladou rodinu a z části pro starší rodiče. Cílem bylo vytvořit společné zázemí obou rodin, avšak i dostatek soukromí. Koncepce domu je založena na přízemní části staršího páru a na dvoupodlažní části mladé čtyřčlenné rodiny.BUTOROVÁ, H.: Multi-generational House: Bachelor´s thesis. Ostrava: VŠB-Technical university of Ostrava, Faculty of Civil Engineering, Department of Architecture 226, 2017, 49 p. Thesis head: Student, A. The subject of bachelor’s thesis „Multi-generational House‟ is preparation of partial project documentation for construction of a building according to notice 499/2006 Sb., about documentation of buildings. As resource materials serves architectural study worked out from Studio Work I and a documentation for building permit worked out from Studio Work Va. Multi-generational House is projected in the spa area Karviná-Darkov. The building consists of a part for young family and a part for grandparents. The goal was to make a common base for both families, but also to secure enough privacy. The philosophy of the house is based on the ground part for older couple and on the two-floor part for young four-member family.226 - Katedra architekturyvelmi dobř

Risk factors for the metabolic syndrome, according to the NCEP-ATPIII criteria, in NFMA patients using four logistic regression models.

<p>Data represent odds ratio (95% confidence interval). RT, radiotherapy; VFD, visual field defects at presentation; Hypopit, hypopituitarism (number of deficient pituitary axes). OR₁: regression model 1 (age and gender). OR₂: regression model 2 (age, gender, BMI). OR₃: regression model 3 (age, gender, VFD, RT, hypopit). OR₄: regression model 4 (age, gender, VFD, RT, Hypopit, BMI). ^†according to the NCEP-ATP III <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0090602#pone.0090602-Grundy1" target="_blank">[10]</a>. *difference statistically significant (P ≤ 0.05).</p

Comparison of metabolic syndrome prevalence, according to the NCEP-ATP III criteria, between NFMA patients and the general population.

<p>O, observed; E, expected. ^†according to the NCEP-ATP III <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0090602#pone.0090602-Grundy1" target="_blank">[10]</a>.</p

Difference (95% CI) in BMD spine, BMD hip and RASM per unit change of sleep parameter in the whole cohort.

<p>Difference (95% CI) in BMD spine, BMD hip and RASM per unit change of sleep parameter in the whole cohort.</p

Characteristics of NFMA patients.

<p>Data represent number (percentage) or median (range). VFD: visual field defects. rhGH: recombinant human growth hormone. ^†Data represent average ± standard deviation. ^‡Percentage within growth hormone deficiency.</p

Effect of transient and continuous CORT treatment on circadian plasma CORT levels in female E3L.CETP mice at baseline (A), week 5 (B) and week 17 (C), as well as on thymus weight (D) and adrenal weight (E) at week 17 (Control group: white bars, transient group: grey bars and continuous group: black bars).

<p>Data are means ± SEM (n = 17–21), *^{, #}<i>P</i><0.05, **^{, ##}<i>P</i><0.01, ^###<i>P</i><0.0001, *versus control group and ^#transient group.</p

Effect of transient and continuous CORT treatment on atherosclerosis development: representative HPS-stained pictures of lesions (A), lesion area (B), lesion type as % of the total lesion (C), representative pictures of monocyte/macrophage staining (adhering monocytes shown by arrows) (D), adhering monocytes per segment (E), macrophage content of the plaque (F) and macrophages as % of the lesion area (G).

<p>Serum anti-ox-LDL specific antibodies (H) and serum M-CSF (I) were measured on week 17. (Control group: white bars, transient group: grey bars and continuous group: black bars). Data are means ± SEM (n = 17–21), Anova with Tukey’s post-hoc test, ^*#<i>P</i><0.05, **<i>P</i><0.01, *versus control group and ^#versus transient group.</p

Effect of transient and continuous CORT treatment on cholesterol distribution over lipoproteins fractioned by FPLC at baseline (A), week 5 (B) and 17 (C) (Control group: white circles, transient group: grey squares and continuous group: black triangles) and on plasma insulin (D), plasma glucose (E), HOMA-IR (F), (Control group: white bars, transient group: grey bars and continuous group: black bars) on week 17.

<p>Data are means ± SEM (n = 17–21), Anova with Tukey’s post-hoc test, **^{, ###}<i>P</i><0.001, *versus control group and ^#versus transient group.</p