Circular Polarized Antenna for Radio Frequency Energy Harvesting Applications in the Smart Cities

Objectives: In this work, the Circularly Polarized (CP) broadband antenna with a simple monopole structure is proposed. Method: The antenna includes the rectangular patch antenna slotted with the four arcs in a circular shape and Top-right, and down-left vertices are removed to obtain the CP and to harvest power from the frequency 1.95-3.05 GHz, it covers the LTE 2100 GHz and Wi-Fi 2.45 GHz, bands. The defected ground structure is considered to design the broadband frequency. The antenna is excited using microstrip feeding. The proposed antenna is simulated, fabricated, and measured. The Dimensions of the antenna are 45 mm x 50 mm x 1.6 mm. Findings: The maximum gain obtained was 4.71dB at a frequency of 2.45 GHz with a Return loss of -43.21 dB, the axial ratio (AR) obtained 1.98dB (less than 3dB) in the same frequency. The impedance bandwidth of 59.8% and 44.89% simulated and measured, respectively, at 2.45 GHz, and VSWR is 0.128 and 1.142 at 2.45 GHz for the simulated and measured, respectively. Novelty: The designed antenna radiation Phenomenon resembles omnidirectional characteristics. Rectenna is designed at 2.45GHz and is connected to the antenna with the help of an impedance matching network. It is used to achieve the RF- DC power conversion efficiency of 84.52%

OPTIMIZATION AND CHARACTERIZATION OF RIVASTIGMINE-LOADED NANOSTRUCTURED LIPID CARRIERS

Objective: The objective of the present study was to develop Nanostructured lipid carriers (NLCs) for improvement in the oral bioavailability of RT. Methods: RT-loaded NLCs were prepared by high shear homogenization technique using fish oil and flaxseed oil respectively. The prepared RT-NLCs were characterized using a phase-contrast microscope, scanning electron microscope (SEM), atomic force microscope (AFM), Fourier transform-infrared spectroscopy (FTIR) and differential scanning calorimetry (DSC). Further, particle size, entrapment efficiency and sustained release of the drug were also studied. Results: SEM results revealed that the RT-NLCs were spherical in shape with a smooth surface. AFM results confirmed the formation of spherical particle dispersions by the NLCs in nanoscale. FTIR spectroscopy and DSC analyses revealed that there is no chemical interaction between the ingredients of RT-NLCs. The particle size of the RT-NLCs was found to be exponentially decreased with the increase in a surfactant solution. Conclusion: The results confirmed pronounced improvement in entrapment efficiency of optimized formulation of RT-NLCs. In vitro, drug release studies showed that RT-NLCs were capable of releasing the drug in a sustained manner. The experimental results showed that the NLCs are potential carriers for providing sustained delivery of rivastigmine

Conditional spatial transition reduction data encoding technique for VLSI interconnects

In this paper the DSM (Deep sub micron) and lower node technologies device dimensions are reduced. Compared to logic functional blocks interconnects dynamic power consumption plays most precarious factor. The most effective way to save the dynamic power in wires is to reduce the number of transitions by using the data encoder. In this paper we proposed a inverse based algorithm with mathematical proof and simple architecture. it is having the different options lower bit inversion, upper bit inversion, all bit inversions and no bit inversion, the data selection based on the which option saves the more power. It is having the simple and compact architecture and it offers less delay compared to related methods

CHARACTERIZATION OF PORCINE AND OVINE MUSCLE MYOGLOBINS

Abstract not availabl

The Parafibromin Tumor Suppressor Protein Is Part of a Human Paf1 Complex

Parafibromin, the product of the HRPT2 (hyperparathyroidism-jaw tumor syndrome 2) tumor suppressor gene, is the human homologue of yeast Cdc73, part of the yeast RNA polymerase II/Paf1 complex known to be important for histone modification and connections to posttranscriptional events. By purifying cellular parafibromin and characterizing its associated proteins, we have identified a human counterpart to the yeast Paf1 complex including homologs of Leo1, Paf1, and Ctr9. Like the yeast complex, the parafibromin complex associates with the nonphosphorylated and Ser2 and Ser5 phosphorylated forms of the RNA polymerase II large subunit. Immunofluorescence experiments show that parafibromin is a nuclear protein. In addition, cotransfection data suggest that parafibromin can interact with a histone methyltransferase complex that methylates histone H3 on lysine 4. Some mutant forms of parafibromin lack association with hPaf1 complex members and with the histone methyltransferase complex, suggesting that disruption of these complexes may correlate with the oncogenic process

Purkinje cell-specific males absent on the first (mMof) gene deletion results in an ataxia-telangiectasia-like neurological phenotype and backward walking in mice

The brains of ataxia telangiectasia (AT) patients display an aberrant loss of Purkinje cells (PCs) that is postulated to contribute to the observed deficits in motor coordination as well as in learning and cognitive function. AT patients have mutations in the ataxia telangiectasia mutated (ATM) gene [Savitsky et al. (1995) Science 268:1749–1753]. However, in Atm-deficient mice, the neurological defects are limited, and the PCs are not deformed or lost as observed in AT patients [Barlow et al. (1996) Cell 86:159–171]. Here we report that PC-specific deletion of the mouse males absent on the first (mMof) gene (Cre−), which encodes a protein that specifically acetylates histone H4 at lysine 16 (H4K16ac) and influences ATM function, is critical for PC longevity. Mice deficient for PC-specific Mof display impaired motor coordination, ataxia, a backward-walking phenotype, and a reduced life span. Treatment of MofF/F/Pcp2-Cre+ mice with histone deacetylase inhibitors modestly prolongs PC survival and delays death. Therefore, Mof expression and H4K16 acetylation are essential for PC survival and function, and their absence leads to PC loss and cerebellar dysfunction similar to that observed in AT patients