4 research outputs found

    Biometric changes in highly myopic eyes before and after phacoemulsification with intraocular lense implantation

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    High short-sightedness is a complicating factor in phacoemulsification cataract affecting the achievement of high visual results. There are no data in the literature that would reflect changes in the biometric parameters of eyes with high myopia after cataract phacoemulsification with intraocular lens implantation. The authors have shown the reliability of this surgical treatment, the absence of complications and a negative effect on the hydrodynamics of the eye. Patients underwent the ophthalmic examination and for patients preparing for phacoemulsification + IOL: A- and B-scanning, endothelial microscopy, optical biometry. In the group of patients with high myopia, regardless of the age, ranged from 3.1 mmto 3.9 mmwith a mean of 3.52 Β± 0.34 mm(variation within 10%), while in the group of patients with myopia and concomitant cataract the fluctuations in anterior chamber depth were recorded in the range from 2.6 mmto 4.4 Β± 0.1 mmwith average values of 3.14 Β± 0.038 mm(variation within 25%). In the dynamics of treatment, a significant reduction in intraocular pressure according to Maklakov by 6.9% was noted in 6 months from baseline (p <0.001) - from 18.6 Β± 2.34 to 17.4 Β± 1.09 mmHg. Art. At the same time, it should be emphasized that the main effect of reducing intraocular pressure was expected already within 1 month (17.9 Β± 1.06 mmHg) after surgery (3.9%, p = 0.006), followed by decrease by 3.1% after 6 months of follow-up (p = 0.008). The research results showed a direct correlation between the endothelial layer density and the anterior chamber depth. Thus, before surgery r = 0.248, and after surgery r = 0.119, i.e. after phacoemulsification with IOL implantation, the density of the endothelial layer depends on changes in the anterior chamber depth (p <0.05). This research has shown that phacoemulsification with IOL implantation is accompanied by positive changes in the size of the mutually located structures of the anterior chamber of the eyeball

    Impact of corneal astigmatism on refractive outcomes after phacoemulsification with implantation of a spherical IOL

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    Background: To date, particular emphasis is being put to correction of preoperative corneal astigmatism in phacoemulsification, since approximately 30% of the world’s population has astigmatism of at least 0.75D which results in decreased visual acuity after cataract surgery. Purpose: To assess the effect of corneal astigmatism on refractive outcomes of phacoemulsification with implantation of a spheric intraocular lens (IOL). Material and Methods: We retrospectively analyzed the outpatient medical records of 39 patients (50 eyes) who received phacoemulsification with a spherical IOL and had corneal astigmatism of 0.5-3.75D (as assessed by keratometry). Eyes were divided into four groups based on the degree of corneal astigmatism. We assessed changes in visual acuity and corneal astigmatism and refractive outcomes of cylindrical correction at one month after surgery. Results: The greater the presurgical astigmatism, the lower was uncorrected visual acuity at one month after surgery. There was no significant difference (Ρ€ > 0.05) in change in corneal astigmatism values after phacoemulsification. In group 1 (preoperative astigmatism of 0.75 D or less) and group 2 (preoperative astigmatism of 1.0 to 1.5 D), the mean increase in visual acuity after cylindrical refractive correction was 10% or less, and had no significant impact on the quality of vision. In group 3 (preoperative astigmatism of 1.75 to 2.5 D) and group 4 (preoperative astigmatism of 2.75 D or more), the mean increase in visual acuity was 15% and 25%, respectively. Conclusion: Surgically induced astigmatism after phacoemulsification had no substantial impact on refractive outcomes. When planning refractive outcomes for eyes with astigmatism after cataract surgery, it should be taken into account that implanting a spherical IOL is acceptable only for eyes with an amount of preoperative astigmatism of 0.75 D or less and vertical axis of astigmatism. Cataract patients with preoperative corneal astigmatism of >0.75 D will require implantation of a toric IOL or a plan for astigmatism correction with another method

    Anatomical optical, biomechanical and morphometric parameters of the eye in children with acquired myopia and syndrome of undifferentiated connective tissue dysplasia

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    Aim of the research – to make a comprehensive assessment of anatomical, optical, biomechanical and morphometric parameters of the eye in children with acquired myopia and syndrome of undifferentiated connective tissue dysplasia. Materials and methods. We examined patients of 2 groups: the I group – 44 children (88 eyes) with myopia of mild degree and phenotypic manifestations of connective tissue dysplasia, the II group – 40 patients (80 eyes) with myopia and without signs of connective tissue dysplasia. We measured the following parameters: corneal refractive power, corneal diameter and radius, thickness of the cornea, anterior chamber depth, lens thickness, sagittal length of the vitreous body, axial length of the eye, corneal hysteresis, peripapillary retinal nerve fiber layer thickness. Results. We revealed significant differences in the anatomical, optical and biomechanical parameters of the visual analyzer in children with myopia of mild degree on the background of the UCTD, in contrast to children with myopia and without connective tissue dysplasia, which consisted in corneal refractive power reduction to an average of 41.25 [40.62; 41.75] D, increase in the corneal radius to an average of 8.13 [7.97; 8.28] mm, the corneal diameter to an average of 12.5 [12.1; 12.7] mm, the anterior chamber depth to an average of 3.8 [3.4; 4.0] mm, the sagittal length of the vitreous body to an average of 17.6 [17.3; 18.0] mm, the axial length of eye to an average of 24.9 [24.4; 25.4] mm, decrease in corneal hysteresis to an average of 11.2 [10.6; 11.7] mm Hg. Peripapillary retinal nerve fiber layer thickness was reduced to an average of 87 [85; 93] ΞΌm. Conclusions. The obtained data can be useful in a comprehensive assessment of ophthalmic manifestations of undifferentiated connective tissue dysplasia syndrome, in myopic process development prediction and in individual treatment tactics determination

    Анатомо-ΠΎΠΏΡ‚ΠΈΡ‡Π½Ρ–, Π±Ρ–ΠΎΠΌΠ΅Ρ…Π°Π½Ρ–Ρ‡Π½Ρ– Ρ‚Π° ΠΌΠΎΡ€Ρ„ΠΎΠΌΠ΅Ρ‚Ρ€ΠΈΡ‡Π½Ρ– ΠΏΠ°Ρ€Π°ΠΌΠ΅Ρ‚Ρ€ΠΈ ΠΎΠΊΠ° Π² Π΄Ρ–Ρ‚Π΅ΠΉ Ρ–Π· Π½Π°Π±ΡƒΡ‚ΠΎΡŽ ΠΌΡ–ΠΎΠΏΡ–Ρ”ΡŽ Ρ‚Π° синдромом Π½Π΅Π΄ΠΈΡ„Π΅Ρ€Π΅Π½Ρ†Ρ–ΠΉΠΎΠ²Π°Π½ΠΎΡ— дисплазії сполучної Ρ‚ΠΊΠ°Π½ΠΈΠ½ΠΈ

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    Aim of the research – to make a comprehensive assessment of anatomical, optical, biomechanical and morphometric parameters of the eye in children with acquired myopia and syndrome of undifferentiated connective tissue dysplasia.Materials and methods. We examined patients of 2 groups: the I group – 44 children (88 eyes) with myopia of mild degree and phenotypic manifestations of connective tissue dysplasia, the II group – 40 patients (80 eyes) with myopia and without signs of connective tissue dysplasia. We measured the following parameters: corneal refractive power, corneal diameter and radius, thickness of the cornea, anterior chamber depth, lens thickness, sagittal length of the vitreous body, axial length of the eye, corneal hysteresis, peripapillary retinal nerve fiber layer thickness.Results. We revealed significant differences in the anatomical, optical and biomechanical parameters of the visual analyzer in children with myopia of mild degree on the background of the UCTD, in contrast to children with myopia and without connective tissue dysplasia, which consisted in corneal refractive power reduction to an average of 41.25 [40.62; 41.75] D, increase in the corneal radius to an average of 8.13 [7.97; 8.28] mm, the corneal diameter to an average of 12.5 [12.1; 12.7] mm, the anterior chamber depth to an average of 3.8 [3.4; 4.0] mm, the sagittal length of the vitreous body to an average of 17.6 [17.3; 18.0] mm, the axial length of eye to an average of 24.9 [24.4; 25.4] mm, decrease in corneal hysteresis to an average of 11.2 [10.6; 11.7] mm Hg. Peripapillary retinal nerve fiber layer thickness was reduced to an average of 87 [85; 93] ΞΌm.Conclusions. The obtained data can be useful in a comprehensive assessment of ophthalmic manifestations of undifferentiated connective tissue dysplasia syndrome, in myopic process development prediction and in individual treatment tactics determination.ЦСль Ρ€Π°Π±ΠΎΡ‚Ρ‹ – провСсти ΠΊΠΎΠΌΠΏΠ»Π΅ΠΊΡΠ½ΡƒΡŽ ΠΎΡ†Π΅Π½ΠΊΡƒ Π°Π½Π°Ρ‚ΠΎΠΌΠΎ-оптичСских, биомСханичСских ΠΈ морфомСтричСских ΠΏΠΎΠΊΠ°Π·Π°Ρ‚Π΅Π»Π΅ΠΉ Π³Π»Π°Π·Π° Ρƒ Π΄Π΅Ρ‚Π΅ΠΉ с ΠΏΡ€ΠΈΠΎΠ±Ρ€Π΅Ρ‚Π΅Π½Π½ΠΎΠΉ ΠΌΠΈΠΎΠΏΠΈΠ΅ΠΉ ΠΈ синдромом Π½Π΅Π΄ΠΈΡ„Ρ„Π΅Ρ€Π΅Π½Ρ†ΠΈΡ€ΠΎΠ²Π°Π½Π½ΠΎΠΉ дисплазии ΡΠΎΠ΅Π΄ΠΈΠ½ΠΈΡ‚Π΅Π»ΡŒΠ½ΠΎΠΉ Ρ‚ΠΊΠ°Π½ΠΈ.ΠœΠ°Ρ‚Π΅Ρ€ΠΈΠ°Π»Ρ‹ ΠΈ ΠΌΠ΅Ρ‚ΠΎΠ΄Ρ‹. ОбслСдовали ΠΏΠ°Ρ†ΠΈΠ΅Π½Ρ‚ΠΎΠ² 2 Π³Ρ€ΡƒΠΏΠΏ: Π† Π³Ρ€ΡƒΠΏΠΏΠ° – 44 Ρ€Π΅Π±Π΅Π½ΠΊΠ° (88 Π³Π»Π°Π·) с ΠΌΠΈΠΎΠΏΠΈΠ΅ΠΉ слабой стСпСни ΠΈ фСнотипичСскими проявлСниями ΡΠΎΠ΅Π΄ΠΈΠ½ΠΈΡ‚Π΅Π»ΡŒΠ½ΠΎΡ‚ΠΊΠ°Π½Π½ΠΎΠΉ дисплазии, Π†Π† Π³Ρ€ΡƒΠΏΠΏΠ° – 40 ΠΏΠ°Ρ†ΠΈΠ΅Π½Ρ‚ΠΎΠ² (80 Π³Π»Π°Π·) с ΠΌΠΈΠΎΠΏΠΈΠ΅ΠΉ ΠΈ Π±Π΅Π· ΠΏΡ€ΠΈΠ·Π½Π°ΠΊΠΎΠ² ΡΠΎΠ΅Π΄ΠΈΠ½ΠΈΡ‚Π΅Π»ΡŒΠ½ΠΎΡ‚ΠΊΠ°Π½Π½ΠΎΠΉ дисплазии. Π˜Π·ΠΌΠ΅Ρ€ΡΠ»ΠΈ ΠΏΠΎΠΊΠ°Π·Π°Ρ‚Π΅Π»ΡŒ ΠΏΡ€Π΅Π»ΠΎΠΌΠ»ΡΡŽΡ‰Π΅ΠΉ силы Ρ€ΠΎΠ³ΠΎΠ²ΠΈΡ†Ρ‹, Π΄ΠΈΠ°ΠΌΠ΅Ρ‚Ρ€ Ρ€ΠΎΠ³ΠΎΠ²ΠΈΡ†Ρ‹, радиус ΠΊΡ€ΠΈΠ²ΠΈΠ·Π½Ρ‹ Ρ€ΠΎΠ³ΠΎΠ²ΠΈΡ†Ρ‹, Ρ‚ΠΎΠ»Ρ‰ΠΈΠ½Ρƒ Ρ€ΠΎΠ³ΠΎΠ²ΠΈΡ†Ρ‹, Π³Π»ΡƒΠ±ΠΈΠ½Ρƒ ΠΏΠ΅Ρ€Π΅Π΄Π½Π΅ΠΉ ΠΊΠ°ΠΌΠ΅Ρ€Ρ‹, Ρ‚ΠΎΠ»Ρ‰ΠΈΠ½Ρƒ хрусталика, ΡΠ°Π³ΠΈΡ‚Ρ‚Π°Π»ΡŒΠ½ΡƒΡŽ Π΄Π»ΠΈΠ½Ρƒ стСкловидного Ρ‚Π΅Π»Π°, Π°ΠΊΡΠΈΠ°Π»ΡŒΠ½ΡƒΡŽ Π΄Π»ΠΈΠ½Ρƒ Π³Π»Π°Π·Π½ΠΎΠ³ΠΎ яблока, ΠΊΠΎΡ€Π½Π΅Π°Π»ΡŒΠ½Ρ‹ΠΉ гистСрСзис, ΠΏΠ΅Ρ€ΠΈΠΏΠ°ΠΏΠΈΠ»Π»ΡΡ€Π½ΡƒΡŽ Ρ‚ΠΎΠ»Ρ‰ΠΈΠ½Ρƒ слоя Π½Π΅Ρ€Π²Π½Ρ‹Ρ… Π²ΠΎΠ»ΠΎΠΊΠΎΠ½.Π Π΅Π·ΡƒΠ»ΡŒΡ‚Π°Ρ‚Ρ‹. УстановлСны достовСрныС различия Π² Π°Π½Π°Ρ‚ΠΎΠΌΠΎ-оптичСских, биомСханичСских ΠΈ морфомСтричСских показатСлях Π·Ρ€ΠΈΡ‚Π΅Π»ΡŒΠ½ΠΎΠ³ΠΎ Π°Π½Π°Π»ΠΈΠ·Π°Ρ‚ΠΎΡ€Π° Ρƒ Π΄Π΅Ρ‚Π΅ΠΉ с ΠΌΠΈΠΎΠΏΠΈΠ΅ΠΉ слабой стСпСни Π½Π° Ρ„ΠΎΠ½Π΅ БНДБВ ΠΈ Π΄Π΅Ρ‚Π΅ΠΉ с ΠΌΠΈΠΎΠΏΠΈΠ΅ΠΉ Π±Π΅Π· ΡΠΎΠ΅Π΄ΠΈΠ½ΠΈΡ‚Π΅Π»ΡŒΠ½ΠΎΡ‚ΠΊΠ°Π½Π½ΠΎΠΉ дисплазии, ΠΊΠΎΡ‚ΠΎΡ€Ρ‹Π΅ Π·Π°ΠΊΠ»ΡŽΡ‡Π°ΡŽΡ‚ΡΡ Π² сниТСнии ΠΏΡ€Π΅Π»ΠΎΠΌΠ»ΡΡŽΡ‰Π΅ΠΉ силы Ρ€ΠΎΠ³ΠΎΠ²ΠΈΡ†Ρ‹ Π² срСднСм Π΄ΠΎ 41,25 [40.62; 41.75] Π΄ΠΏΡ‚Ρ€, ΡƒΠ²Π΅Π»ΠΈΡ‡Π΅Π½ΠΈΠΈ радиуса Ρ€ΠΎΠ³ΠΎΠ²ΠΈΡ†Ρ‹ Π² срСднСм Π΄ΠΎ 8,13 [7.97; 8.28] ΠΌΠΌ, Π΄ΠΈΠ°ΠΌΠ΅Ρ‚Ρ€Π° Ρ€ΠΎΠ³ΠΎΠ²ΠΈΡ†Ρ‹ Π² срСднСм Π΄ΠΎ 12,5 [12.1; 12.7] ΠΌΠΌ, ΡƒΠ²Π΅Π»ΠΈΡ‡Π΅Π½ΠΈΠΈ Π³Π»ΡƒΠ±ΠΈΠ½Ρ‹ ΠΏΠ΅Ρ€Π΅Π΄Π½Π΅ΠΉ ΠΊΠ°ΠΌΠ΅Ρ€Ρ‹ Π² срСднСм Π΄ΠΎ 3,8 [3.4; 4.0] ΠΌΠΌ, ΡƒΠ²Π΅Π»ΠΈΡ‡Π΅Π½ΠΈΠΈ ΡΠ°Π³ΠΈΡ‚Ρ‚Π°Π»ΡŒΠ½ΠΎΠΉ Π΄Π»ΠΈΠ½Ρ‹ стСкловидного Ρ‚Π΅Π»Π° Π² срСднСм Π΄ΠΎ 17,6 [17.3; 18.0] ΠΌΠΌ, ΡƒΠ²Π΅Π»ΠΈΡ‡Π΅Π½ΠΈΠΈ аксиальной Π΄Π»ΠΈΠ½Ρ‹ Π³Π»Π°Π·Π° Π² срСднСм Π΄ΠΎ 24,9 [24.4; 25.4] ΠΌΠΌ, сниТСнии ΠΊΠΎΡ€Π½Π΅Π°Π»ΡŒΠ½ΠΎΠ³ΠΎ гистСрСзиса Π² срСднСм Π΄ΠΎ 11,2 [10.6; 11.7] ΠΌΠΌ Ρ€Ρ‚. ст., сниТСнии Ρ‚ΠΎΠ»Ρ‰ΠΈΠ½Ρ‹ слоя пСрипапиллярных Π½Π΅Ρ€Π²Π½Ρ‹Ρ… Π²ΠΎΠ»ΠΎΠΊΠΎΠ½ Π² срСднСм Π΄ΠΎ 87 [85; 93] ΞΌm.Π’Ρ‹Π²ΠΎΠ΄Ρ‹. Π Π΅Π·ΡƒΠ»ΡŒΡ‚Π°Ρ‚Ρ‹ ΠΌΠΎΠ³ΡƒΡ‚ Π±Ρ‹Ρ‚ΡŒ ΠΏΠΎΠ»Π΅Π·Π½Ρ‹ΠΌΠΈ Π² комплСксной ΠΎΡ†Π΅Π½ΠΊΠ΅ ΠΎΡ„Ρ‚Π°Π»ΡŒΠΌΠΎΠ»ΠΎΠ³ΠΈΡ‡Π΅ΡΠΊΠΈΡ… проявлСний синдрома Π½Π΅Π΄ΠΈΡ„Ρ„Π΅Ρ€Π΅Π½Ρ†ΠΈΡ€ΠΎΠ²Π°Π½Π½ΠΎΠΉ дисплазии ΡΠΎΠ΅Π΄ΠΈΠ½ΠΈΡ‚Π΅Π»ΡŒΠ½ΠΎΠΉ Ρ‚ΠΊΠ°Π½ΠΈ, ΠΏΡ€ΠΎΠ³Π½ΠΎΠ·ΠΈΡ€ΠΎΠ²Π°Π½ΠΈΠΈ развития миопичСского процСсса ΠΈ ΠΎΠΏΡ€Π΅Π΄Π΅Π»Π΅Π½ΠΈΠΈ ΠΈΠ½Π΄ΠΈΠ²ΠΈΠ΄ΡƒΠ°Π»ΡŒΠ½ΠΎΠΉ Ρ‚Π°ΠΊΡ‚ΠΈΠΊΠΈ лСчСния.Β ΠœΠ΅Ρ‚Π° Ρ€ΠΎΠ±ΠΎΡ‚ΠΈ – Π²ΠΈΠΊΠΎΠ½Π°Ρ‚ΠΈ комплСкснС ΠΎΡ†Ρ–Π½ΡŽΠ²Π°Π½Π½Ρ Π°Π½Π°Ρ‚ΠΎΠΌΠΎ-ΠΎΠΏΡ‚ΠΈΡ‡Π½ΠΈΡ…, Π±Ρ–ΠΎΠΌΠ΅Ρ…Π°Π½Ρ–Ρ‡Π½ΠΈΡ… Ρ– ΠΌΠΎΡ€Ρ„ΠΎΠΌΠ΅Ρ‚Ρ€ΠΈΡ‡Π½ΠΈΡ… ΠΏΠΎΠΊΠ°Π·Π½ΠΈΠΊΡ–Π² ΠΎΠΊΠ° Π² Π΄Ρ–Ρ‚Π΅ΠΉ Ρ–Π· Π½Π°Π±ΡƒΡ‚ΠΎΡŽ ΠΌΡ–ΠΎΠΏΡ–Ρ”ΡŽ Ρ‚Π° синдромом Π½Π΅Π΄ΠΈΡ„Π΅Ρ€Π΅Π½Ρ†Ρ–ΠΉΠΎΠ²Π°Π½ΠΎΡ— дисплазії сполучної Ρ‚ΠΊΠ°Π½ΠΈΠ½ΠΈ.ΠœΠ°Ρ‚Π΅Ρ€Ρ–Π°Π»ΠΈ Ρ‚Π° ΠΌΠ΅Ρ‚ΠΎΠ΄ΠΈ. ΠžΠ±ΡΡ‚Π΅ΠΆΠΈΠ»ΠΈ ΠΏΠ°Ρ†Ρ–Ρ”Π½Ρ‚Ρ–Π² Π΄Π²ΠΎΡ… Π³Ρ€ΡƒΠΏ: Π† – 44 Π΄ΠΈΡ‚ΠΈΠ½ΠΈ (88 ΠΎΡ‡Π΅ΠΉ) Π· ΠΌΡ–ΠΎΠΏΡ–Ρ”ΡŽ слабкого ступСня Ρ‚Π° Ρ„Π΅Π½ΠΎΡ‚ΠΈΠΏΠΎΠ²ΠΈΠΌΠΈ проявами сполучнотканинної дисплазії, Π†Π† Π³Ρ€ΡƒΠΏΠ° – 40 ΠΏΠ°Ρ†Ρ–Ρ”Π½Ρ‚Ρ–Π² (80 ΠΎΡ‡Π΅ΠΉ) Ρ–Π· ΠΌΡ–ΠΎΠΏΡ–Ρ”ΡŽ Ρ‚Π° Π±Π΅Π· ΠΎΠ·Π½Π°ΠΊ сполучнотканинної дисплазії. Π’ΠΈΠΌΡ–Ρ€ΡŽΠ²Π°Π»ΠΈ ΠΏΠΎΠΊΠ°Π·Π½ΠΈΠΊ Π·Π°Π»ΠΎΠΌΠ»ΡŽΠ²Π°Π½ΠΎΡ— сили Ρ€ΠΎΠ³Ρ–Π²ΠΊΠΈ, Π΄Ρ–Π°ΠΌΠ΅Ρ‚Ρ€ Ρ€ΠΎΠ³Ρ–Π²ΠΊΠΈ, радіус ΠΊΡ€ΠΈΠ²ΠΈΠ·Π½ΠΈ Ρ€ΠΎΠ³Ρ–Π²ΠΊΠΈ, Ρ‚ΠΎΠ²Ρ‰ΠΈΠ½Ρƒ Ρ€ΠΎΠ³Ρ–Π²ΠΊΠΈ, Π³Π»ΠΈΠ±ΠΈΠ½Ρƒ ΠΏΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΡ— ΠΊΠ°ΠΌΠ΅Ρ€ΠΈ, Ρ‚ΠΎΠ²Ρ‰ΠΈΠ½Ρƒ ΠΊΡ€ΠΈΡˆΡ‚Π°Π»ΠΈΠΊΠ°, ΡΠ°Π³Ρ–Ρ‚Π°Π»ΡŒΠ½Ρƒ Π΄ΠΎΠ²ΠΆΠΈΠ½Ρƒ склоподібного Ρ‚Ρ–Π»Π°, Π°ΠΊΡΡ–Π°Π»ΡŒΠ½Ρƒ Π΄ΠΎΠ²ΠΆΠΈΠ½Ρƒ ΠΎΡ‡Π½ΠΎΠ³ΠΎ яблука, ΠΊΠΎΡ€Π΅Π°Π»ΡŒΠ½ΠΈΠΉ гістСрСзис, пСрипапілярну Ρ‚ΠΎΠ²Ρ‰ΠΈΠ½Ρƒ ΡˆΠ°Ρ€Ρƒ Π½Π΅Ρ€Π²ΠΎΠ²ΠΈΡ… Π²ΠΎΠ»ΠΎΠΊΠΎΠ½.Π Π΅Π·ΡƒΠ»ΡŒΡ‚Π°Ρ‚ΠΈ. Виявили Π²Ρ–Ρ€ΠΎΠ³Ρ–Π΄Π½Ρ– відмінності Π·Π° Π°Π½Π°Ρ‚ΠΎΠΌΠΎ-ΠΎΠΏΡ‚ΠΈΡ‡Π½ΠΈΠΌΠΈ, Π±Ρ–ΠΎΠΌΠ΅Ρ…Π°Π½Ρ–Ρ‡Π½ΠΈΠΌΠΈ ΠΉ ΠΌΠΎΡ€Ρ„ΠΎΠΌΠ΅Ρ‚Ρ€ΠΈΡ‡Π½ΠΈΠΌΠΈ ΠΏΠΎΠΊΠ°Π·Π½ΠΈΠΊΠ°ΠΌΠΈ Π·ΠΎΡ€ΠΎΠ²ΠΎΠ³ΠΎ Π°Π½Π°Π»Ρ–Π·Π°Ρ‚ΠΎΡ€Π° Π΄Ρ–Ρ‚Π΅ΠΉ Ρ–Π· ΠΌΡ–ΠΎΠΏΡ–Ρ”ΡŽ слабкого ступСня Π½Π° Ρ‚Π»Ρ– БНДБВ Ρ– Π΄Ρ–Ρ‚Π΅ΠΉ Ρ–Π· ΠΌΡ–ΠΎΠΏΡ–Ρ”ΡŽ Π±Π΅Π· сполучнотканинної дисплазії, Ρ‰ΠΎ ΠΏΠΎΠ»ΡΠ³Π°ΡŽΡ‚ΡŒ Ρƒ Π·Π½ΠΈΠΆΠ΅Π½Π½Ρ– Π·Π°Π»ΠΎΠΌΠ»ΡŽΠ²Π°Π½ΠΎΡ— сили Ρ€ΠΎΠ³Ρ–Π²ΠΊΠΈ Π² ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 41,25 [40.62; 41.75] Π΄ΠΏΡ‚Ρ€, Π·Π±Ρ–Π»ΡŒΡˆΠ΅Π½Π½Ρ– радіуса Ρ€ΠΎΠ³Ρ–Π²ΠΊΠΈ Π² ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 8,13 [7.97; 8.28] ΠΌΠΌ, Π΄Ρ–Π°ΠΌΠ΅Ρ‚Ρ€Π° Ρ€ΠΎΠ³Ρ–Π²ΠΊΠΈ Π² ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 12,5 [12.1; 12.7] ΠΌΠΌ, Π·Π±Ρ–Π»ΡŒΡˆΠ΅Π½Π½Ρ– Π³Π»ΠΈΠ±ΠΈΠ½ΠΈ ΠΏΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΡ— ΠΊΠ°ΠΌΠ΅Ρ€ΠΈ Π² ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 3,8 [3.4; 4.0] ΠΌΠΌ, Π·Π±Ρ–Π»ΡŒΡˆΠ΅Π½Π½Ρ ΡΠ°Π³Ρ–Ρ‚Π°Π»ΡŒΠ½ΠΎΡ— Π΄ΠΎΠ²ΠΆΠΈΠ½ΠΈ склоподібного Ρ‚Ρ–Π»Π° Π² ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 17,6 [17.3; 18.0] ΠΌΠΌ, Π·Π±Ρ–Π»ΡŒΡˆΠ΅Π½Π½Ρ– Π°ΠΊΡΡ–Π°Π»ΡŒΠ½ΠΎΡ— Π΄ΠΎΠ²ΠΆΠΈΠ½ΠΈ ΠΎΠΊΠ° Π² ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 24,9 [24.4; 25.4] ΠΌΠΌ, Π·Π½ΠΈΠΆΠ΅Π½Π½Ρ– ΠΊΠΎΡ€Π½Π΅Π°Π»ΡŒΠ½ΠΎΠ³ΠΎ гістСрСзису Π² ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 11,2 [10.6; 11.7] ΠΌΠΌ Ρ€Ρ‚. ст., Π·Π½ΠΈΠΆΠ΅Π½Π½Ρ– Ρ‚ΠΎΠ²Ρ‰ΠΈΠ½ΠΈ ΡˆΠ°Ρ€Ρƒ пСрипапілярних Π½Π΅Ρ€Π²ΠΎΠ²ΠΈΡ… Π²ΠΎΠ»ΠΎΠΊΠΎΠ½ Ρƒ ΡΠ΅Ρ€Π΅Π΄Π½ΡŒΠΎΠΌΡƒ Π΄ΠΎ 87 [85; 93] ΞΌm.Висновки. Π Π΅Π·ΡƒΠ»ΡŒΡ‚Π°Ρ‚ΠΈ ΠΌΠΎΠΆΡƒΡ‚ΡŒ Π±ΡƒΡ‚ΠΈ корисними Π² комплСксному ΠΎΡ†Ρ–Π½ΡŽΠ²Π°Π½Π½Ρ– ΠΎΡ„Ρ‚Π°Π»ΡŒΠΌΠΎΠ»ΠΎΠ³Ρ–Ρ‡Π½ΠΈΡ… проявів синдрому Π½Π΅Π΄ΠΈΡ„Π΅Ρ€Π΅Π½Ρ†Ρ–ΠΉΠΎΠ²Π°Π½ΠΎΡ— дисплазії сполучної Ρ‚ΠΊΠ°Π½ΠΈΠ½ΠΈ, ΠΏΡ€ΠΎΠ³Π½ΠΎΠ·ΡƒΠ²Π°Π½Π½Ρ– Ρ€ΠΎΠ·Π²ΠΈΡ‚ΠΊΡƒ ΠΌΡ–ΠΎΠΏΡ–Ρ‡Π½ΠΎΠ³ΠΎ процСсу Ρ– Π²ΠΈΠ·Π½Π°Ρ‡Π΅Π½Π½Ρ– Ρ–Π½Π΄ΠΈΠ²Ρ–Π΄ΡƒΠ°Π»ΡŒΠ½ΠΎΡ— Ρ‚Π°ΠΊΡ‚ΠΈΠΊΠΈ лікування.
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