102 research outputs found
Production of Secondaries in High Energy d+Au Collisions
In the framework of Quark-Gluon String Model we calculate the inclusive
spectra of secondaries produced in d+Au collisions at intermediate (CERN SPS)
and at much higher (RHIC) energies. The results of numerical calculations at
intermediate energies are in reasonable agreement with the data. At RHIC
energies numerically large inelastic screening corrections (percolation
effects) should be accounted for in calculations. We extract these effects from
the existing RHIC experimental data on minimum bias and central d+Au
collisions. The predictions for p+Au interactions at LHC energy are also given.Comment: 18 pages and 10 figure
Feynman scaling violation on baryon spectra in pp collisions at LHC and cosmic ray energies
A significant asymmetry in baryon/antibaryon yields in the central region of
high energy collisions is observed when the initial state has non-zero baryon
charge. This asymmetry is connected with the possibility of baryon charge
diffusion in rapidity space. Such a diffusion should decrease the baryon charge
in the fragmentation region and translate into the corresponding decrease of
the multiplicity of leading baryons. As a result, a new mechanism for Feynman
scaling violation in the fragmentation region is obtained. Another numerically
more significant reason for the Feynman scaling violation comes from the fact
that the average number of cutted Pomerons increases with initial energy. We
present the quantitative predictions of the Quark-Gluon String Model (QGSM) for
the Feynman scaling violation at LHC energies and at even higher energies that
can be important for cosmic ray physics.Comment: 21 pages, 11 figures, and 1 table. arXiv admin note: substantial text
overlap with arXiv:1107.1615, arXiv:1007.320
Oral ulcer as an exclusive sign of gastric cancer: report of a rare case
BACKGROUND: The oral cavity is a rare but occasional target for metastases, which may masquerade as various benign and inflammatory lesions, and sometimes also be asymptomatic. Oral metastatic lesions have been described in various cancers, particularly lung, breast and kidney carcinoma. CASE PRESENTATION: We here describe an uncommon case of a hard palate mucosa and gingival metastasis from gastric carcinoma that was originally diagnosed as a periodontal disease. Histopathological examination of a biopsy of the lesion revealed a signet-ring cell carcinoma, and a subsequent biopsy of an ulcerated stomach lesion showed a poorly differentiated gastric carcinoma. The patient underwent gastric resection but died of heart failure on the tenth postoperative day; a post-mortem examination revealed a residual bilateral ovarian infiltration by gastric carcinoma (Krukenberg's tumor). CONCLUSION: An occult carcinoma of the stomach may rarely metastasise to the oral cavity even as a first and exclusive manifestation; it is important to bear this possibility in mind because such conditions may mimic a benign disease
Thermotomaculum hydrothermale gen. nov., sp. nov., a novel heterotrophic thermophile within the phylum Acidobacteria from a deep-sea hydrothermal vent chimney in the Southern Okinawa Trough
http://www.godac.jamstec.go.jp/darwin/cruise/natsushima/nt08-13/
Multiple Interactions and the Structure of Beam Remnants
Recent experimental data have established some of the basic features of
multiple interactions in hadron-hadron collisions. The emphasis is therefore
now shifting, to one of exploring more detailed aspects. Starting from a brief
review of the current situation, a next-generation model is developed, wherein
a detailed account is given of correlated flavour, colour, longitudinal and
transverse momentum distributions, encompassing both the partons initiating
perturbative interactions and the partons left in the beam remnants. Some of
the main features are illustrated for the Tevatron and the LHC.Comment: 69pp, 33 figure
Production of secondaries in soft p+pb collisions at LHC
We calculate the inclusive spectra of secondaries produced in soft (minimum
bias) p+Pb collisions in the framework of Quark-Gluon String Model at LHC
energy, and by taking into account the inelastic screening corrections
(percolation effects). The role of these effects is expected to be very large
at very high energies, and they should decrease the spectra about 3 times in
the midrapidity region and increase them about 2 times in the fragmentation
region at the energy of LHC.Comment: 18 pages and 10 figures. arXiv admin note: text overlap with
arXiv:0802.219
Lambda-Baryon Production in pi(+-)n Interactions
The process of Lambda-baryon production in pi-p collisions is considered. The
contribution of the string-junction mechanism to the strange baryon production
in meson-baryon scattering is anlysed. The results of numerical calculations in
the framework of the Quark-Gluon String model are in reasonable agreement with
the data.Comment: 10 pages and 5 figue
Characterisation of prostate cancer lesions in heterozygous Men1 mutant mice
<p>Abstract</p> <p>Background</p> <p>Mutations of the <it>MEN1 </it>gene predispose to multiple endocrine neoplasia type 1 (MEN1) syndrome. Our group and others have shown that <it>Men1 </it>disruption in mice recapitulates MEN1 pathology. Intriguingly, rare lesions in hormone-dependent tissues, such as prostate and mammary glands, were also observed in the <it>Men1 </it>mutant mice.</p> <p>Methods</p> <p>To study the occurrence of prostate lesions, we followed a male mouse cohort of 47 <it>Men1</it><sup>+/- </sup>mice and 23 age-matched control littermates, starting at 18 months of age, and analysed the prostate glands from the cohort.</p> <p>Results</p> <p>Six <it>Men1</it><sup>+/- </sup>mice (12.8%) developed prostate cancer, including two adenocarcinomas and four <it>in situ </it>carcinomas, while none of the control mice developed cancerous lesions. The expression of menin encoded by the <it>Men1 </it>gene was found to be drastically reduced in all carcinomas, and partial LOH of the wild-type <it>Men1 </it>allele was detected in three of the five analysed lesions. Using immunostaining for the androgen receptor and p63, a basal epithelial cell marker, we demonstrated that the menin-negative prostate cancer cells did not display p63 expression and that the androgen receptor was expressed but more heterogeneous in these lesions. Furthermore, our data showed that the expression of the cyclin-dependent kinase inhibitor CDKN1B (p27), a <it>Men1 </it>target gene known to be inactivated during prostate cell tumorigenesis, was notably decreased in the prostate cancers that developed in the mutant mice.</p> <p>Conclusion</p> <p>Our work suggests the possible involvement of <it>Men1 </it>inactivation in the tumorigenesis of the prostate gland.</p
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