Flat type thick film inductive sensors

Two thick film flat-type inductive sensors are described and tested for distance and profile measurement. The first one is a single-layer spiral while the second one is a multi-layer structure consisting of ten spirals one over the other. The paper describes their geometric configurations together with their simulated magnetic fields and it reports the results from the characterization test i.e. the series-equivalent circuit parameters, the sensitivity and the cross-sensitivity to temperature. An experimental analysis of the sensitivity suggests that optimized values are obtained by an appropriate choice of the working frequency. The sensors are shielded against e.m. noise coming from the nonsensitive area. Moreover, two sensors have been tested in the laboratory using the single layer as a distance sensor and the multi-layer as a transducer for the measurement of a metallic object profile. The results of the tests show a maximum sensitivity of 14mV/µm and a resolution of 0.6 µm for the single layer, while the multi layer one reconstructs the profile with an axial resolution of a few microns and a lateral resolution better than 200 mm

A single-magnet nonlinear piezoelectric converter for enhanced energy harvesting from random vibrations

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Flat Type Thick Film Inductive Sensors

Two thick film flat-type inductive sensors are described and tested for distance and profile measurement. The first one is a single-layer spiral while the second one is a multi-layer structure consisting of ten spirals one over the other. The paper describes their geometric configurations together with their simulated magnetic fields and it reports the results from the characterization test i.e. the series-equivalent circuit parameters, the sensitivity and the cross-sensitivity to temperature. An experimental analysis of the sensitivity suggests that optimized values are obtained by an appropriate choice of the working frequency. The sensors are shielded against e.m. noise coming from the nonsensitive area. Moreover, two sensors have been tested in the laboratory using the single layer as a distance sensor and the multi-layer as a transducer for the measurement of a metallic object profile. The results of the tests show a maximum sensitivity of 14mV/µm and a resolution of 0.6 µm for the single layer, while the multi layer one reconstructs the profile with an axial resolution of a few microns and a lateral resolution better than 200 mm

Highly sensitive smart flow sensor with frequency and duty cycle output

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TREM-1 expression on neutrophils and monocytes of septic patients: relation to the underlying infection and the implicated pathogen

<p>Abstract</p> <p>Background</p> <p>Current knowledge on the exact ligand causing expression of TREM-1 on neutrophils and monocytes is limited. The present study aimed at the role of underlying infection and of the causative pathogen in the expression of TREM-1 in sepsis.</p> <p>Methods</p> <p>Peripheral venous blood was sampled from 125 patients with sepsis and 88 with severe sepsis/septic shock. The causative pathogen was isolated in 91 patients. Patients were suffering from acute pyelonephritis, community-acquired pneumonia (CAP), intra-abdominal infections (IAIs), primary bacteremia and ventilator-associated pneumonia or hospital-acquired pneumonia (VAP/HAP). Blood monocytes and neutrophils were isolated. Flow cytometry was used to estimate the TREM-1 expression from septic patients.</p> <p>Results</p> <p>Within patients bearing intrabdominal infections, expression of TREM-1 was significantly lower on neutrophils and on monocytes at severe sepsis/shock than at sepsis. That was also the case for severe sepsis/shock developed in the field of VAP/HAP. Among patients who suffered infections by Gram-negative community-acquired pathogens or among patients who suffered polymicrobial infections, expression of TREM-1 on monocytes was significantly lower at the stage of severe sepsis/shock than at the stage of sepsis.</p> <p>Conclusions</p> <p>Decrease of the expression of TREM-1 on the membrane of monocytes and neutrophils upon transition from sepsis to severe sepsis/septic shock depends on the underlying type of infection and the causative pathogen.</p