Improve IT Business Process Performance Using AI and Deep Learning

Perfection is an ambitious goal. However, it’s right to say that in-Service Management we aspire to do the best we possibly can. We improve our performance through practice, but for real acceleration, we need to take a fresh approach. “IT is the backbone of the modern enterprise”—if this is the case and we demand a consistently high level of performance from our IT staff now is the time to think about how best to achieve this. With the use of AI powered autonomous micro-learning coupled with machine learning, employees can now be evaluated and coached by providing instance learning and feedback in real time so as to improve process performance

Founding properties on non-ferrous liquid metals

ALTHOUGH the liquid state is the first step of all founding operations no fundamental measure is available to evaluate the capacity of liquid metals to flow adeq-uately into intricate moulds. Whilst a consideration of viscosity & surface tension is natural in any discussion of the flow of liquid metals, it is not adequate to describe their founding characteristics.In this paper an attempt has been made to evolve a fundamental basis of viscosity and surface tension and their impact on casting fluidity

Modulation of impact of obesity in pathological and physiological cardiac hypertrophy by orlistat

High fat diet (30 % fat) was used to induce obesity in rats. Male wistar rats were kept at high fat diet for 90 days and subjected to partial abdominal aortic constriction (PAAC) at 62nd day and continued upto 90th day. Similarly rats were kept at high fat diet for 90 days and subjected to chronic swimming training (CST) at 46th day and continued upto 90th day. Obesity was assessed by measuring body weight, WHR ratio, obesity index and adiposity index. Cardiac hypertrophy was assessed by measuring left ventricular weight, left ventricular weight to body weight, left ventricular wall thickness, cardiomyocyte diameter, left ventricular protein content and left ventricular collagen content. Mean arterial blood pressure (MABP) was also recorded. Oxidative stress was assessed in terms of thiobarbituric acid reactive species (TBARS) level, superoxide anion generation level and reduced glutathione level in left ventricular tissue. Obesity, cardiac hypertrophy and oxidative stress were increased in high fat diet groups. Orlistat, lipase inhibitor, significantly attenuated the impact of obesity in experimental cardiac hypertrophy. Furthermore orlistat significantly attenuated the oxidative stress. So it can be concluded that by decreasing the impact of obesity, orlistat significantly attenuated the extent of cardiac hypertrophy.Colegio de Farmacéuticos de la Provincia de Buenos Aire

Single Valued Neutrosophic Signedgraphs

Motivated by the notion of single valued neutrosophic graphs defined by Broumi, Talea, Bakali and Smarandache[2] and notion of intuitionistic fuzzy signed graphs defined by Mishra and Pal[8], we introduce the concept of single valued neutrosophic signed graphs and examine the properties of this new concept and examples

Effect of sodium tungstate on obesity induced cardiac hypertrophy and oxidative stress

Obesity was induced by high fat diet (30 % fat by weight). Wistar rats of 225-250 g were kept on high fat diet to induce obesity for 90 days. Sodium tungstate (2 mg/ml, in drinking water) was administered for 90 days. Obesity was assessed by measuring % age change in body weight, WHR ratio, adiposity index and obesity index. Left ventricular cardiac hypertrophy was assessed in terms of left ventricular weight, left ventricular wall thickness, left ventricular protein content and left ventricular collagen content. Oxidative stress was measured in terms of levels of thiobarbituric acid reactive substances (TBARS), superoxide anion generation (SAG) and level of reduced glutathione. Sodium tungstate significantly attenuated the increase in body weight adiposity index, obesity index, TBARS, SAG and reduced glutathione, whereas no significant change was observed the parameters of cardiac hypertrophy. So, it can be concluded that sodium tungstate significantly attenuated high fat diet induced obesity and oxidative stress but no significant decrease was observed in the parameters of cardiac hypertrophy.Colegio de Farmacéuticos de la Provincia de Buenos Aire

Modulation of impact of obesity in pathological and physiological cardiac hypertrophy by orlistat

High fat diet (30 % fat) was used to induce obesity in rats. Male wistar rats were kept at high fat diet for 90 days and subjected to partial abdominal aortic constriction (PAAC) at 62nd day and continued upto 90th day. Similarly rats were kept at high fat diet for 90 days and subjected to chronic swimming training (CST) at 46th day and continued upto 90th day. Obesity was assessed by measuring body weight, WHR ratio, obesity index and adiposity index. Cardiac hypertrophy was assessed by measuring left ventricular weight, left ventricular weight to body weight, left ventricular wall thickness, cardiomyocyte diameter, left ventricular protein content and left ventricular collagen content. Mean arterial blood pressure (MABP) was also recorded. Oxidative stress was assessed in terms of thiobarbituric acid reactive species (TBARS) level, superoxide anion generation level and reduced glutathione level in left ventricular tissue. Obesity, cardiac hypertrophy and oxidative stress were increased in high fat diet groups. Orlistat, lipase inhibitor, significantly attenuated the impact of obesity in experimental cardiac hypertrophy. Furthermore orlistat significantly attenuated the oxidative stress. So it can be concluded that by decreasing the impact of obesity, orlistat significantly attenuated the extent of cardiac hypertrophy.Colegio de Farmacéuticos de la Provincia de Buenos Aire

Effect of sodium tungstate on obesity induced cardiac hypertrophy and oxidative stress

Obesity was induced by high fat diet (30 % fat by weight). Wistar rats of 225-250 g were kept on high fat diet to induce obesity for 90 days. Sodium tungstate (2 mg/ml, in drinking water) was administered for 90 days. Obesity was assessed by measuring % age change in body weight, WHR ratio, adiposity index and obesity index. Left ventricular cardiac hypertrophy was assessed in terms of left ventricular weight, left ventricular wall thickness, left ventricular protein content and left ventricular collagen content. Oxidative stress was measured in terms of levels of thiobarbituric acid reactive substances (TBARS), superoxide anion generation (SAG) and level of reduced glutathione. Sodium tungstate significantly attenuated the increase in body weight adiposity index, obesity index, TBARS, SAG and reduced glutathione, whereas no significant change was observed the parameters of cardiac hypertrophy. So, it can be concluded that sodium tungstate significantly attenuated high fat diet induced obesity and oxidative stress but no significant decrease was observed in the parameters of cardiac hypertrophy.Colegio de Farmacéuticos de la Provincia de Buenos Aire