A novel noncontact ultrasound indentation system for measurement of tissue material properties using water jet compression

Author name used in this publication: M. H. LuAuthor name used in this publication: Y. P. ZhengAuthor name used in this publication: Q. H. Huang2004-2005 > Academic research: refereed > Publication in refereed journalAccepted ManuscriptPublishe

A novel method to obtain modulus image of soft tissues using ultrasound water jet indentation : a phantom study

2006-2007 > Academic research: refereed > Publication in refereed journalVersion of RecordPublishe

Ultrasound elastomicroscopy using water beam indentation : preliminary study

Author name used in this publication: M. H. LuAuthor name used in this publication: Y. P. ZhengAuthor name used in this publication: Q. H. Huang2004-2005 > Academic research: not refereed > Chapter in an edited book (author)Accepted ManuscriptPublishe

A complete MacWilliams theorem for convolutional codes

© 2014 IEEE. In this paper, we prove a MacWilliams identity for the weight adjacency matrices based on the constraint codes of a convolutional code (CC) and its dual. Our result improves upon a recent result by Gluesing-Luerssen and Schneider, where the requirement of a minimal encoder is assumed. We can also establish the MacWilliams identity for the input-parity weight adjacency matrices of a systematic CC and its dual. Most importantly, we show that a type of Hamming weight enumeration functions of all codewords of a CC can be derived from the weight adjacency matrix, which thus provides a connection between these two very different notions of weight enumeration functions in the convolutional code literature. Finally, the relations between various enumeration functions of a CC and its dual are summarized in a diagram. This explains why no MacWilliams identity exists for the free-distance enumerators

Evaluation of bone-tendon junction healing using water jet ultrasound indentation method

2009-2010 > Academic research: refereed > Publication in refereed journalAccepted ManuscriptPublishe

3-D measurement of body tissues based on ultrasound images with 3-D spatial information

2005-2006 > Academic research: refereed > Publication in refereed journalAccepted ManuscriptPublishe

Development of a portable 3D ultrasound imaging system for musculoskeletal tissues

Author name used in this publication: Q. H. HuangAuthor name used in this publication: Y. P. ZhengAuthor name used in this publication: M. H. LuAuthor name used in this publication: Z. R. ChiCentre for Signal Processing, Department of Electronic and Information EngineeringRehabilitation Engineering Centre2004-2005 > Academic research: refereed > Publication in refereed journalAccepted ManuscriptPublishe

Cerebellar defects in Pdss2 conditional knockout mice during embryonic development and in adulthood

PDSS2 is a gene that encodes one of the two subunits of trans-prenyl diphosphate synthase that is essential for ubiquinone biosynthesis. It is known that mutations in PDSS2 can cause primary ubiquinone deficiency in humans and a similar disease in mice. Cerebellum is the most often affected organ in ubiquinone deficiency, and cerebellar atrophy has been diagnosed in many infants with this disease. In this study, two Pdss2 conditional knockout mouse lines directed by Pax2-cre and Pcp2-cre were generated to investigate the effect of ubiquinone deficiency on cerebellum during embryonic development and in adulthood, respectively. The Pdss2 f/-; Pax2-cre mouse recapitulates some symptoms of ubiquinone deficiency in infants, including severe cerebellum hypoplasia and lipid accumulation in skeletal muscles at birth. During early cerebellum development (E12.5-14.5), Pdss2 knockout initially causes the delay of radial glial cell growth and neuron progenitor migration, so the growth of mutant cerebellum is retarded. During later development (E15.5-P0), increased ectopic apoptosis of neuroblasts and impaired cell proliferation result in the progression of cerebellum hypoplasia in the mutant. Thus, the mutant cerebellum contains fewer neurons at birth, and the cells are disorganized. The developmental defect of mutant cerebellum does not result from reduced Fgf8 expression before E12.5. Electron microscopy reveals mitochondrial defects and increased autophagic-like vacuolization that may arise in response to abnormal mitochondria in the mutant cerebellum. Nevertheless, the mutant mice die soon after birth probably due to cleft palate and micrognathia, which may result from Pdss2 knockout caused by ectopic Pax2-cre expression in the first branchial arch. On the other hand, the Pdss2 f/-; Pcp2-cre mouse is healthy at birth but gradually loses cerebellar Purkinje cells and develops ataxia-like symptoms at 9.5months; thus this conditional knockout mouse may serve as a model for ubiquinone deficiency in adult patients. In conclusion, this study provides two mouse models of Pdss2 based ubiquinone deficiency. During cerebellum development, Pdss2 knockout results in severe cerebellum hypoplasia by impairing cell migration and eliciting ectopic apoptosis, whereas Pdss2 knockout in Purkinje cells at postnatal stages leads to the development of cerebellar ataxia. © 2011 Elsevier Inc.postprin

A low cost and reliable dimmable ballast topology with inherent power regulation and insensitivity to lamp characteristics

The output power of the proposed dimmable ballast topology is self-regulated and insensitive to the lamp characteristics. Accurate dimming operation with rather constant filament power can be realized simply by open loop control of the switching frequency of the converter. It can provide reliable operation during lamp ignition with fault conditions or even short circuit without the need of current sensing circuits. Prototype was built for experimental verifications. For completeness, a simple ultra low dimming method is also proposed together with the theoretical dimming limit considered. © 2005 IEEE.published_or_final_versio

A new adaptive interpolation algorithm for 3D ultrasound imaging with speckle reduction and edge preservation

Author name used in this publication: Qinghua HuangAuthor name used in this publication: Yongping ZhengAuthor name used in this publication: Minhua Lu2008-2009 > Academic research: refereed > Publication in refereed journalAccepted ManuscriptPublishe