886 research outputs found
A simple derivation of level spacing of quasinormal frequencies for a black hole with a deficit solid angle and quintessence-like matter
In this paper, we investigate analytically the level space of the imaginary
part of quasinormal frequencies for a black hole with a deficit solid angle and
quintessence-like matter by the Padmanabhan's method \cite{Padmanabhan}.
Padmanabhan presented a method to study analytically the imaginary part of
quasinormal frequencies for a class of spherically symmetric spacetimes
including Schwarzschild-de Sitter black holes which has an evenly spaced
structure. The results show that the level space of scalar and gravitational
quasinormal frequencies for this kind of black holes only depend on the surface
gravity of black-hole horizon in the range of -1 < w < -1/3, respectively . We
also extend the range of to , the results of which are similar
to that in -1 < w < -1/3 case. Particularly, a black hole with a deficit solid
angle in accelerating universe will be a Schwarzschild-de Sitter black hole,
fixing and . And a black hole with a deficit solid
angle in the accelerating universe will be a Schwarzschild black hole,when
and . In this paper, is the parameter of state
equation, is a parameter relating to a deficit solid angle and
is the density of static spherically symmetrical quintessence-like
matter at .Comment: 6 pages, Accepted for publication in Astrophysics & Space Scienc
Global Monopole in Asymptotically dS/AdS Spacetime
In this paper, we investigate the global monopole in asymptotically dS/Ads
spacetime and find that the mass of the monopole in the asymptotically dS
spacetime could be positive if the cosmological constant is greater than a
critical value. This shows that the gravitational field of the global monopole
could be attractive or repulsive depending on the value of the cosmological
constant.Comment: 5 pages, 1 figure, to appear in Phys. Rev.
On the Crystalline Approximants of the Al-Mn, Al-Pd and Al-Mn-Pd Type Decagonal Quasicrystals
On the basis of atomic clusters of decagonal quasicrystals, subunits of decagonal quasicrystals are proposed as the results of aggregation of the atomic clusters. The structure of the crystalline approximants can be characterized as some simple periodic tiling of the subunits. This is shown as a new approach to study the structure of the new crystalline approximants
Attractor Solution of Phantom Field
In light of recent study on the dark energy models that manifest an equation
of state , we investigate the cosmological evolution of phantom field in
a specific potential, exponential potential in this paper. The phase plane
analysis show that the there is a late time attractor solution in this model,
which address the similar issues as that of fine tuning problems in
conventional quintessence models. The equation of state is determined by
the attractor solution which is dependent on the parameter in the
potential. We also show that this model is stable for our present observable
universe.Comment: 9 pages, 3 ps figures; typos corrected, references updated, this is
the final version to match the published versio
Fermionic Casimir effect with helix boundary condition
In this paper, we consider the fermionic Casimir effect under a new type of
space-time topology using the concept of quotient topology. The relation
between the new topology and that in Ref. \cite{Feng,Zhai3} is something like
that between a M\"obius strip and a cylindric. We obtain the exact results of
the Casimir energy and force for the massless and massive Dirac fields in the
()-dimensional space-time. For both massless and massive cases, there is a
symmetry for the Casimir energy. To see the effect of the mass, we
compare the result with that of the massless one and we found that the Casimir
force approaches the result of the force in the massless case when the mass
tends to zero and vanishes when the mass tends to infinity.Comment: 7 pages, 4 figures, published in Eur. Phys. J.
Antibacterial effect of orange Monascus pigment against Staphylococcus aureus
The objective of this work was to research the antibacterial effects of orange pigment, which was separated from Monascus pigments, against Staphylococcus aureus. The increase of the diameter of inhibition zone treated with orange pigment indicated that orange pigment had remarkable antibacterial activities against S. aureus. Orange pigment (10 mg mlâ1) had a strong destructive effect on the membrane and structure of S. aureus by the analysis of scanning electron microscopy as well as transmission electron microscopy. Sodium dodecyl sulphate-polyacrylamide gel electrophoresis (SDS-PAGE) further demonstrated that the cell membrane was seriously damaged by orange pigment, which resulted in the leakage of protein from S. aureus cells. A significant decrease in the synthesis of DNA was also seen in S. aureus cells exposed to 10 mg mlâ1 orange pigment. All in all, orange pigment showed excellent antibacterial effects against S. aureus
Structural and molecular basis of the assembly of the TRPP2/PKD1 complex
Mutations in PKD1 and TRPP2 account for nearly all cases of autosomal dominant polycystic kidney disease (ADPKD). These 2 proteins form a receptor/ion channel complex on the cell surface. Using a combination of biochemistry, crystallography, and a single-molecule method to determine the subunit composition of proteins in the plasma membrane of live cells, we find that this complex contains 3 TRPP2 and 1 PKD1. A newly identified coiled-coil domain in the C terminus of TRPP2 is critical for the formation of this complex. This coiled-coil domain forms a homotrimer, in both solution and crystal structure, and binds to a single coiled-coil domain in the C terminus of PKD1. Mutations that disrupt the TRPP2 coiled-coil domain trimer abolish the assembly of both the full-length TRPP2 trimer and the TRPP2/PKD1 complex and diminish the surface expression of both proteins. These results have significant implications for the assembly, regulation, and function of the TRPP2/PKD1 complex and the pathogenic mechanism of some ADPKD-producing mutations
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