A simple derivation of level spacing of quasinormal frequencies for a black hole with a deficit solid angle and quintessence-like matter

In this paper, we investigate analytically the level space of the imaginary part of quasinormal frequencies for a black hole with a deficit solid angle and quintessence-like matter by the Padmanabhan's method \cite{Padmanabhan}. Padmanabhan presented a method to study analytically the imaginary part of quasinormal frequencies for a class of spherically symmetric spacetimes including Schwarzschild-de Sitter black holes which has an evenly spaced structure. The results show that the level space of scalar and gravitational quasinormal frequencies for this kind of black holes only depend on the surface gravity of black-hole horizon in the range of -1 < w < -1/3, respectively . We also extend the range of $w$ to $w \leq -1$, the results of which are similar to that in -1 < w < -1/3 case. Particularly, a black hole with a deficit solid angle in accelerating universe will be a Schwarzschild-de Sitter black hole, fixing $w = -1$ and $\epsilon^2 = 0$. And a black hole with a deficit solid angle in the accelerating universe will be a Schwarzschild black hole,when $\rho_0 = 0$ and $\epsilon^2 = 0$. In this paper, $w$ is the parameter of state equation, $\epsilon^2$ is a parameter relating to a deficit solid angle and $\rho_0$ is the density of static spherically symmetrical quintessence-like matter at $r = 1$.Comment: 6 pages, Accepted for publication in Astrophysics & Space Scienc

Global Monopole in Asymptotically dS/AdS Spacetime

In this paper, we investigate the global monopole in asymptotically dS/Ads spacetime and find that the mass of the monopole in the asymptotically dS spacetime could be positive if the cosmological constant is greater than a critical value. This shows that the gravitational field of the global monopole could be attractive or repulsive depending on the value of the cosmological constant.Comment: 5 pages, 1 figure, to appear in Phys. Rev.

On the Crystalline Approximants of the Al-Mn, Al-Pd and Al-Mn-Pd Type Decagonal Quasicrystals

On the basis of atomic clusters of decagonal quasicrystals, subunits of decagonal quasicrystals are proposed as the results of aggregation of the atomic clusters. The structure of the crystalline approximants can be characterized as some simple periodic tiling of the subunits. This is shown as a new approach to study the structure of the new crystalline approximants

Attractor Solution of Phantom Field

In light of recent study on the dark energy models that manifest an equation of state $w<-1$, we investigate the cosmological evolution of phantom field in a specific potential, exponential potential in this paper. The phase plane analysis show that the there is a late time attractor solution in this model, which address the similar issues as that of fine tuning problems in conventional quintessence models. The equation of state $w$ is determined by the attractor solution which is dependent on the $\lambda$ parameter in the potential. We also show that this model is stable for our present observable universe.Comment: 9 pages, 3 ps figures; typos corrected, references updated, this is the final version to match the published versio

Fermionic Casimir effect with helix boundary condition

In this paper, we consider the fermionic Casimir effect under a new type of space-time topology using the concept of quotient topology. The relation between the new topology and that in Ref. \cite{Feng,Zhai3} is something like that between a M\"obius strip and a cylindric. We obtain the exact results of the Casimir energy and force for the massless and massive Dirac fields in the ($D+1$)-dimensional space-time. For both massless and massive cases, there is a $Z_2$ symmetry for the Casimir energy. To see the effect of the mass, we compare the result with that of the massless one and we found that the Casimir force approaches the result of the force in the massless case when the mass tends to zero and vanishes when the mass tends to infinity.Comment: 7 pages, 4 figures, published in Eur. Phys. J.

Antibacterial effect of orange Monascus pigment against Staphylococcus aureus

The objective of this work was to research the antibacterial effects of orange pigment, which was separated from Monascus pigments, against Staphylococcus aureus. The increase of the diameter of inhibition zone treated with orange pigment indicated that orange pigment had remarkable antibacterial activities against S. aureus. Orange pigment (10 mg ml−1) had a strong destructive effect on the membrane and structure of S. aureus by the analysis of scanning electron microscopy as well as transmission electron microscopy. Sodium dodecyl sulphate-polyacrylamide gel electrophoresis (SDS-PAGE) further demonstrated that the cell membrane was seriously damaged by orange pigment, which resulted in the leakage of protein from S. aureus cells. A significant decrease in the synthesis of DNA was also seen in S. aureus cells exposed to 10 mg ml−1 orange pigment. All in all, orange pigment showed excellent antibacterial effects against S. aureus

Structural and molecular basis of the assembly of the TRPP2/PKD1 complex

Mutations in PKD1 and TRPP2 account for nearly all cases of autosomal dominant polycystic kidney disease (ADPKD). These 2 proteins form a receptor/ion channel complex on the cell surface. Using a combination of biochemistry, crystallography, and a single-molecule method to determine the subunit composition of proteins in the plasma membrane of live cells, we find that this complex contains 3 TRPP2 and 1 PKD1. A newly identified coiled-coil domain in the C terminus of TRPP2 is critical for the formation of this complex. This coiled-coil domain forms a homotrimer, in both solution and crystal structure, and binds to a single coiled-coil domain in the C terminus of PKD1. Mutations that disrupt the TRPP2 coiled-coil domain trimer abolish the assembly of both the full-length TRPP2 trimer and the TRPP2/PKD1 complex and diminish the surface expression of both proteins. These results have significant implications for the assembly, regulation, and function of the TRPP2/PKD1 complex and the pathogenic mechanism of some ADPKD-producing mutations