Wildlife Damage Control in Eastern Cities and Suburbs

The topics covered in this paper are based mainly on experiences with people seeking help from the University of Maryland and the National Institute for Urban Wildlife. Although most residents of cities and suburbs enjoy and appreciate wildlife, we are frequently reminded that some do not, other people only tolerate and enjoy selected animal species, provided that these animals \u27\u27behave - they don\u27t make noise, keep their distance, don\u27t damage ornamentals, and are not messy. Some people are so intolerant of wildlife that they even complain about frog choruses from nearby ponds. Response to wildlife varies such that one resident may detest squirrels and seek to destroy all that visit his property, while his neighbor, on the other hand, enjoys having them in his yard and may even provide them with food and nesting shelters

United States Fish and Wildlife Service Reports

Report containing insights into fish and wildlife management, as well as examples for developing habitat around mining areas

Ohio's Status as a Game and Fur Producing State

Author Institution: U. S. Fish and Wildlife Service, Washington, D. C

United States Fish and Wildlife Service Report FWS/OBS-80/24

Report that contains information on effects of coal surface mining on the populations of fish and wildlife

An annotated bibliography on planning and management for urban-suburban wildlife /

"FWS/OBS-79/25."Includes index."Follow-up volume to Planning for wildlife in cities and suburbs by Daniel L. Leedy, Robert M. Maestro, and Thomas M. Franklin."Biological services program"--Cover.Mode of access: Internet

Highway-wildlife relationships. Volume 2. An annotated bibliography. Final report.

Federal Highway Administration, Office of Research and Development, Washington, D.C.Mode of access: Internet.Author corporate affiliation: Urban Wildlife Research Center, Inc., Ellicott City, Md.Subject code: CDBSubject code: CRJSubject code: EZASubject code: SDCDSubject code: SDESubject code: YE

Self-Compensation in Semiconductors: The Zn Vacancy in Ga-Doped ZnO

Self-compensation, the tendency of a crystal to lower its energy by forming point defects to counter the effects of a dopant, is here quantitatively proven. Based on a new theoretical formalism and several different experimental techniques, we demonstrate that the addition of 1.4 × 1021-cm−3 Ga donors in ZnO causes the lattice to form 1.7 × 1020-cm−3 Zn-vacancy acceptors. The calculated VZn formation energy of 0.2 eV is consistent with predictions from density functional theory. Our formalism is of general validity and can be used to investigate self-compensation in any degenerate semiconductor material

The Effects of Adolescent Methylphenidate Exposure on the Behavioral and Brain-Derived Neurotrophic Factor Response to Nicotine

This study analyzed the interaction of adolescent methylphenidate on the behavioral response to nicotine and the effects of these drug treatments on brain-derived neurotrophic factor in the nucleus accumbens and hippocampus in male and female Sprague-Dawley rats. Animals were intraperitoneal administered 1 mg/kg methylphenidate or saline using a “school day” regimen (five days on, two days off) beginning on postnatal day (P)28 and throughout behavioral testing. In Experiment 1, animals were intraperitoneal administered 0.5 mg/kg (free base) nicotine or saline every second day for 10 days from P45–P63 and tested after a three-day drug washout on the forced swim stress task on P67–P68. Results revealed that adolescent methylphenidate blunted nicotine behavioral sensitization. However, methylphenidate-treated rats given saline during sensitization demonstrated decreased latency to immobility and increased immobility time on the forced swim stress task in males that was reduced by nicotine. In Experiment 2, a different set of animals were conditioned to nicotine (0.6 mg/kg free base) or saline using the conditioned place preference behavioral paradigm from P44–P51, and given a preference test on P52. On P53, the nucleus accumbens and hippocampus were analyzed for brain-derived neurotrophic factor. Methylphenidate enhanced nicotine-conditioned place preference in females and nicotine produced conditioned place preference in males and females pre-exposed to saline in adolescence. In addition, methylphenidate and nicotine increased nucleus accumbens brain-derived neurotrophic factor in females and methylphenidate enhanced hippocampus brain-derived neurotrophic factor in males and females. Methylphenidate adolescent exposure using a clinically relevant dose and regimen results in changes in the behavioral and brain-derived neurotrophic factor responses to nicotine in adolescence that are sex-dependent