10 research outputs found
Secondary Instabilities of Surface Waves on Viscous Fluids in the Faraday Instability
Secondary instabilities of Faraday waves show three regimes: (1) As seen
previously, low-viscosity (nu) fluids destabilize first into squares. At higher
driving accelerations a, squares show low-frequency modulations corresponding
to the motion of phase defects, while theory predicts a stationary transverse
amplitude modulation (TAM). (2) High-nu fluids destabilize first to stripes.
Stripes then show an oscillatory TAM whose frequency is incommensurate with the
driving frequency. At higher a, the TAM undergoes a phase instability. At still
higher a, edge dislocations form and fluid droplets are ejected. (3)
Intermediate-nu fluids show a complex coexistence of squares and stripes, as
well as stationary and oscillatory TAM instabilities of the stripes.Comment: REVTEX, with 3 separate uuencoded figures, to appear in Europhys.
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Local estimates for entropy densities in coupled map lattices
We present a method to derive an upper bound for the entropy density of
coupled map lattices with local interactions from local observations. To do
this, we use an embedding technique being a combination of time delay and
spatial embedding. This embedding allows us to identify the local character of
the equations of motion. Based on this method we present an approximate
estimate of the entropy density by the correlation integral.Comment: 4 pages, 5 figures include
A Phase Front Instability in Periodically Forced Oscillatory Systems
Multiplicity of phase states within frequency locked bands in periodically
forced oscillatory systems may give rise to front structures separating states
with different phases. A new front instability is found within bands where
(). Stationary fronts shifting the
oscillation phase by lose stability below a critical forcing strength and
decompose into traveling fronts each shifting the phase by . The
instability designates a transition from stationary two-phase patterns to
traveling -phase patterns
Aberrant Splicing of the Senataxin Gene in a Patient with Ataxia with Oculomotor Apraxia Type 2
Ataxia with oculomotor apraxia type 2 (AOA2) is caused by a diversity of mutations within the coding region of the senataxin gene. Recently, rare noncoding senataxin mutations affecting RNA processing have been identified in AOA2. Here, we report the case of an 18-year-old woman, with classic clinical features of AOA2, who was found to harbor a mutation within senataxin intron 16. This mutation disrupts the local 5′ splice site architecture via a novel intronic frameshift mechanism, causing skipping of exon 16 with predicted disruption of the conserved DNA/RNA helicase domain. RNA processing mutations expand the growing complexity of pathogenic senataxin mutations
Random Pinning of Localized States and the Birth of Deterministic Disorder within Gradient Models
The birth of spatial disorder from almost regular initial conditions within the Swift-Hohenberg model equation with subcritical bifurcation is considered. The complexity of the space series Imeasured by the spatial K.,-entropy) grows with time and reaches a stationary value depending on the period of the initial regular disturbance. A qualitative model is suggested describing the process via the birth of localized structures and its subsequent disordering due to weak interaction