Enacted task design: tasks as written in the classroom

This paper presents and describes the construct of enacted task design, which considers the way tasks are “written” (designed) by teachers. Two enactments by different teachers based on the same written algebra task were analyzed and compared using the math story framework (Dietiker, 2015). Variations in these stories highlight four dimensions of the teacher’s design work

Prologomenon to an Empirical Restatement of Conflicts

Symposium: Preparing for the Next Century-A New Restatement of Conflicts

Quenched Hadron Spectrum and Decay Constants on the lattice

In this talk we present the results obtained from a study of ${\cal O}(2000)$ (quenched) lattice configurations from the APE collaboration, at $6.0\le\beta\le 6.4$, using both the Wilson and the SW-Clover fermion action. We determine the light hadronic spectrum and the meson decay constants. For the light-light systems we find an agreement with the experimental data of $\sim 5$ for mesonic masses and $\sim 10$ for baryonic masses and pseudoscalar decay constants; a larger deviation is present for the vector decay constants. For the heavy-light decay constants we find $f_{D_s}=237 \pm 16 MeV, f_{D} = 221 \pm 17 MeV (f_{D_s}/f_D=1.07(4)), f_{B_s} = 205 \pm 35 MeV, f_{B} = 180 \pm 32 MeV (f_{B_s}/f_B=1.14(8))$, in good agreement with previous estimates.Comment: 8 pages, latex, Talk given at XXV ITEP Winter School of Physics, Moscow - Russia, 18-27 Feb 199

Appellate Justice Bureaucracy and Scholarship

Many of the other Articles in this Symposium demonstrate that a single great piece of legal scholarship can have an enormous impact on the development of legal doctrine. This Article differs in two respects. First, it focuses not on a single seminal work, but rather on a developing literature authored by a large group of scholars. Second, it attempts to assess the impact of that literature not on the growth of legal theory, but on the development of a single legal institution-the United States Courts of Appeals

Isolation and characterisation of the chick orthologue of the Opitz syndrome gene, Mid1, supports a conserved role in vertebrate development

© UBC PressThe X-linked form of Opitz syndrome (OS) is caused by loss of function of the microtubule-associated MID1 protein. The phenotype of OS includes defects along the central body axis, namely hypertelorism, cleft lip and palate, hypospadias and cardiac structural anomalies. Here we describe the isolation and characterisation of full-length cDNA clones representing the chick Mid1 gene and the detailed profile of its expression in stage 7 to 28 chick embryos. Consistent with the remarkable sequence conservation of MID1 between human and chick was the good correlation of the pattern of cMid1 expression with the tissues affected in OS. In stage 10 embryos, transcripts were concentrated in the head mesenchyme which includes migratory neural crest cells. However, the incomplete overlap with a neural crest marker, Sox10, suggests that Mid1 is a marker for somitomeric mesoderm and potentially for a subset of neural crest cells. Consistent with this, cMid1 expression was also detected at later stages in neural crest-derived facial mesenchyme, in the myotome and in the condensing muscle blocks of the limb. Expression of cMid1 was observed in the neural epithelium of the forebrain beginning at stage 7 with increased signal in presumptive rhombomeres 2/3. By stage 15, expression is highest in the diencephalon. Other areas with high expression are certain facial epithelia and the midgut that will give rise to the oesophagus and trachea. These data indicate that Mid1 plays an evolutionarily conserved developmental function in vertebrates that may involve effects on cellular proliferation, tissue interactions and morphogenesis.Joy M. Richman, Katherine K. Fu, Liza L. Cox, Jane P. Sibbons and Timothy C. Co

Natural entropy fluctuations discriminate similar looking electric signals emitted from systems of different dynamics

Complexity measures are introduced, that quantify the change of the natural entropy fluctuations at different length scales in time-series emitted from systems operating far from equilibrium. They identify impending sudden cardiac death (SD) by analyzing fifteen minutes electrocardiograms, and comparing to those of truly healthy humans (H). These measures seem to be complementary to the ones suggested recently [Phys. Rev. E {\bf 70}, 011106 (2004)] and altogether enable the classification of individuals into three categories: H, heart disease patients and SD. All the SD individuals, who exhibit critical dynamics, result in a common behavior.Comment: Published in Physical Review