60 research outputs found

    Global data set of long-term summertime vertical temperature profiles in 153 lakes

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    peer reviewedClimate change and other anthropogenic stressors have led to long-term changes in the thermal structure, including surface temperatures, deepwater temperatures, and vertical thermal gradients, in many lakes around the world. Though many studies highlight warming of surface water temperatures in lakes worldwide, less is known about long-term trends in full vertical thermal structure and deepwater temperatures, which have been changing less consistently in both direction and magnitude. Here, we present a globally-expansive data set of summertime in-situ vertical temperature profiles from 153 lakes, with one time series beginning as early as 1894. We also compiled lake geographic, morphometric, and water quality variables that can influence vertical thermal structure through a variety of potential mechanisms in these lakes. These long-term time series of vertical temperature profiles and corresponding lake characteristics serve as valuable data to help understand changes and drivers of lake thermal structure in a time of rapid global and ecological change. © 2021, The Author(s)

    Angiotensin-converting enzyme inhibitors and renal failure in diabetes

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    Consequences of lake and river ice loss on cultural ecosystem services

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    People extensively use lakes and rivers covered by seasonal ice. Although ice cover duration has been declining over the past 150 years for Northern Hemisphere freshwaters, we know relatively little about how ice loss directly affects humans. We synthesize the cultural ecosystem services (i.e., services that provide intangible or nonmaterial benefits) and associated benefits supported by inland ice. We also provide, for the first time, empirical examples that give quantitative evidence for a winter warming effect on a wide range of ice-related cultural ecosystem services and benefits. We show that in recent decades, warmer air temperatures delayed the opening date of winter ice roads and led to cancellations of spiritual ceremonies, outdoor ice skating races, and ice fishing tournaments. Additionally, our synthesis effort suggests unexploited datasets that allow for the use of integrative approaches to evaluate the interplay between inland ice loss and society

    The ECHORD Project: A General Perspective

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    Neoproterozoic cap carbonates: a critical appraisal of existing models and the plumeworld hypothesis

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    Evidence for glaciation during the mid-late Neoproterozoic is widespread on Earth, reflecting three or more ice ages between 730 Ma and 580 Ma. Of these, the late Neoproterozoic Marinoan glaciation of approximately 635 Ma stands out because of its ubiquitous association with a characteristic, microcrystalline cap dolostone that drapes glacially influenced rock units worldwide. The Marinoan glaciation is also peculiar in that evidence for low altitude glaciation at equatorial latitudes is compelling. Three models have been proposed linking abrupt deglaciation with this global carbonate precipitation event: (i) overturn of an anoxic deep ocean; (ii) catastrophically accelerated rates of chemical weathering because of supergreenhouse conditions following global glaciation (Snowball Earth Hypothesis); and (iii) massive release of carbonate alkalinity from destabilized methane clathrates. All three models invoke extreme alkalinity fluxes into seawater during deglaciation but none explains how such alkalinity excess from point sources could be distributed homogeneously around the globe. In addition, none explains the consistent sequence of precipitation events observed within cap carbonate successions, specifically: (i) the global blanketing of carbonate powder in shallow marine environments during deglaciation; (ii) widespread and disruptive precipitation of dolomite cement; followed by (iii) localized barite precipitation and seafloor cementation by aragonite. The conceptual model presented here proposes that low latitude deglaciation was so massive and abrupt that the resultant meltwater plume could extend worldwide, physically separating the surface and deep ocean reservoirs for ≥103 years. It is proposed that cap dolostones formed primarily by microbially mediated precipitation of carbonate whitings during algal blooms within this low salinity plumeworld rather than by abiotic precipitation from normal salinity seawater. Many of the disruption features that are characteristic of cap dolostones can be explained by microbially mediated, early diagenetic dolomitization and cementation. The re-initiation of whole ocean circulation degassed CO2 into the atmosphere in areas of upwelling, triggering localized, abiotic CaCO3 precipitation in the form of aragonite fans that overlie cap dolostones in NW Canada and Namibia. The highly oxygenated shallow marine environments of the glacial and post-glacial Neoproterozoic world provided consistently favourable conditions for the evolutionary development of animals and other oxygenophiles

    Supplementary Material for: Venous Thromboembolism and the Risk of Death and Graft Loss in Kidney Transplant Recipients

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    <p><b><i>Background:</i></b> The implications of venous thromboembolism (VTE) for morbidity and mortality in kidney transplant recipients are not well described. <b><i>Methods:</i></b> We conducted a retrospective study using linked healthcare databases in Ontario, Canada to determine the risk and complications of VTE in kidney transplant recipients from 2003 to 2013. We compared the incidence rate of VTE in recipients (<i>n</i> = 4,343) and a matched (1:4) sample of the general population (<i>n</i> = 17,372). For recipients with evidence of a VTE posttransplant, we compared adverse clinical outcomes (death, graft loss) to matched (1:2) recipients without evidence of a VTE posttransplant. <b><i>Results:</i></b> During a median follow-up of 5.2 years, 388 (8.9%) recipients developed a VTE compared to 254 (1.5%) in the matched general population (16.3 vs. 2.4 events per 1,000 person-years; hazard ratio [HR] 7.1, 95% CI 6.0-8.4; <i>p</i> < 0.0001). Recipients who experienced a posttransplant VTE had a higher risk of death (28.5 vs. 11.2%; HR 4.1, 95% CI 2.9-5.8; <i>p</i> < 0.0001) and death-censored graft loss (13.1 vs. 7.5%; HR 2.3, 95% CI 1.4-3.6; <i>p</i> = 0.0006) compared to matched recipients who did not experience a posttransplant VTE. <b><i>Conclusions:</i></b> Kidney transplant recipients have a sevenfold higher risk of VTE compared to the general population with VTE conferring an increased risk of death and graft loss.</p
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