Censorship and Taboo Maintenance in L1 and LX Swearing

In this paper, we consider the censorship of public swear word usage as a function of, and continued maintenance of, taboo with a focus on L1 and LX swearing and its management. In research with multilingual speakers, first-language swear words are consistently perceived as more taboo, and thus more emotional/powerful than equivalent words from a second or third language. While the public use of English-language swear words may be subject to censorship in Anglophone contexts, it is not censored to the same extent in LX contexts. On the other hand, L1 swear words are censored. Such perceptions of differences in strength between one’s L1 and LX languages also seem to affect the work of language professionals: translators’ tendency to self-censor may at least in part be explained by this bias. The existence of a two-tier system of swearing and censorship serves to reinvigorate L1 swear words, while diminishing the power of English swear words. We thus examine how censorship works as a means of maintaining and/or attenuating taboo, potentially moderating the power of swearing itself in cross-linguistic and multilingual contexts

Confucius and the varifocal stance

We put the bifocal stance theory (BST) into dialogue with the Confucian approach to ritual. The aim of the commentary is two-fold: To draw on BST to provide an explanatory framework for a Confucian approach to social learning and, while doing so, to show how Chinese (Confucian) philosophy can contribute to debates in cultural evolution. In response to: Jagiello, R., Heyes, C., & Whitehouse, H. (2022). Tradition and invention: The bifocal stance theory of cultural evolution. Behavioral and Brain Sciences, 45, E249. doi:10.1017/S0140525X2200038

Pbx homeodomain proteins pattern both the zebrafish retina and tectum

<p>Abstract</p> <p>Background</p> <p><it>Pbx </it>genes encode TALE class homeodomain transcription factors that pattern the developing neural tube, pancreas, and blood. Within the hindbrain, Pbx cooperates with Hox proteins to regulate rhombomere segment identity. Pbx cooperates with Eng to regulate midbrain-hindbrain boundary maintenance, and with MyoD to control fast muscle cell differentiation. Although previous results have demonstrated that Pbx is required for proper eye size, functions in regulating retinal cell identity and patterning have not yet been examined.</p> <p>Results</p> <p>Analysis of retinal ganglion cell axon pathfinding and outgrowth in <it>pbx2/4 </it>null embryos demonstrated a key role for <it>pbx </it>genes in regulating neural cell behavior. To identify Pbx-dependent genes involved in regulating retino-tectal pathfinding, we conducted a microarray screen for Pbx-dependent transcripts in zebrafish, and detected genes that are specifically expressed in the eye and tectum. A subset of Pbx-dependent retinal transcripts delineate specific domains in the dorso-temporal lobe of the developing retina. Furthermore, we determined that some Pbx-dependent transcripts also require Meis1 and Gdf6a function. Since <it>gdf6a </it>expression is also dependent on Pbx, we propose a model in which Pbx proteins regulate expression of the growth factor <it>gdf6a</it>, which in turn regulates patterning of the dorso-temporal lobe of the retina. This, in concert with aberrant tectal patterning in <it>pbx2/4 </it>null embryos, may lead to the observed defects in RGC outgrowth.</p> <p>Conclusion</p> <p>These data define a novel role for Pbx in patterning the vertebrate retina and tectum in a manner required for proper retinal ganglion cell axon outgrowth.</p