9 research outputs found
Helicobacter pylori on oral mucosa - topographic distribution
Svrha rada bila je ispitati topografsku distribuciju bakterije Helicobacter pylori na oralnoj sluznici u upalnim, ulceroznim i keratotiÄnim lezijama, kandidijazi i sindromu peÄenja usta i usporediti s nalazom na zdravoj sluznici.
Primijenili smo nested PCR na citoloÅ”kim uzorcima uzetim s 9 topografski razliÄitih mjesta oralne sluznice i s razliÄitih oralnih lezija.
Od ukupno 426 ispitanika njih 66 (15,49 %) imalo je pozitivan nalaz H. pylori na oralnoj sluznici, bez predilekcije po spolu, ali s veÄom ÄestoÄom nalaza u starijih ispitanika u usporedbi s mlaÄima te gotovo jednakom distribucijom na svim ispitanim topografskim stranama
oralne sluznice. H. pylori otkriven je u svim ispitanim lezijama i sindromu peÄenja usta osim u kandidijazi s veÄom prevalencijom na sluznici dorzuma jezika i u ulceroznim lezijama, premda bez statistiÄki znatne razlike izmeÄu lezija i zdrave sluznice (p = 0,059).
H. pylori nije patogen za oralnu sluznicu jer kolonizira gotovo s istom ÄestoÄom zdravu i oboljelu oralnu sluznicu. TakoÄer ne postoje preferirana mjesta na oralnoj sluznici za naseljavanje H. pylori.The purpose of this study was to examine the topographic distribution of Helicobacter pylori on oral mucosa in various oral lesions such as inflammation, oral ulceration, keratosis, candidiasis and burning mouth syndrome in comparison with healthy oral mucosa.
We applied nested PCR on cytologic specimens collected from 9 topographic sites of the healthy oral mucosa and from various oral lesions.
Of a total 426 patients 66 (15.49 %) were positive for H. pylori,with no predilection for sex but with higher frequency in older patients, and with almost equal distribution on all examined topographic sites of the oral mucosa. H. pylori was detected in all examined diseases, except in candidiasis, with prevalence on tongue mucosa and in
ulcerous lesions although without significant differences between lesions and healthy oral mucosa (p = 0.059).
H.pylori is non-pathogenic for oral mucosa since it colonizes almost with the same frequency healthy and diseased oral mucosa. There is no preferential oral site or lesion for colonization of H. pylori
Post-Traumatic Stress Disorder Patients and Periodontal Health
Progresija parodontitisa ovisi o istodobnoj pojavi nekoliko Äimbenika bolesti. Osim mikroorganizama subgingivnoga plaka pokazano je da stres, promjenom odgovora domaÄina, pridonosi destrukciji tkiva u parodontnoj bolesti. Ratni stres može izazvati trajne posljedice, ukljuÄujuÄi posttraumatski stresni poremeÄaj (PTSP). Željeli smo ispitati prevalenciju nekih parodontnih patogena, povezanost tih mikroorganizama s kliniÄkim parametrima parodontitisa i utjecaj ratnoga stresa na parodontno zdravlje u pacijenata s PTSP-om. Istraživanje je provedeno na 130 ispitanika: 50 ratnih veterana oboljelih i lijeÄenih od PTSP-a, 50 osoba s progresivnim parodontitisom (PP) i 30 parodontno zdravih osoba. Skupina s progresivnim parodontitisom ukljuÄuje oboljele od kroniÄnog (CO) i agresivnog (AP) parodontitisa. Jedino je skupina ratnih veterana bila neposredno izložena ratnome stresu. Proveden je potanki parodontni pregled i uzeti su uzorci subgingivne mikroflore svakog ispitanika. KliniÄki pregled je ukljuÄivao odreÄivanje plak indeksa (PI), indeksa krvareÄeg sulkusa (SBI), dubinu sondiranja (PD) i kliniÄki gubitak priÄvrstka (CAL). Metoda lanÄane reakcije polimeraze (PCR) upotrijebljena je za identifikaciju sljedeÄih bakterija: Actinobacillus actinomycetemcomitans(Aa), Porphyromonas gingivalis (Pg) i Eikenella corrodens (Es). Prevalencija u svim skupinama za Ec bila je 81% , za Aa 36% i za Pg 19%. Prevalencija za Ec bila je 74% u PTSP skupni, 80% u PP skupni i 93% u parodontno zdravih ispitanika. Prevalencija za Aa bila je 30% u PTSP skupini, 46% u PP skupini i 30% u parodontno zdravih ispitanika. Prevalencija za Pg bila je 16% u PTSP skupini, 16% u PP skupini i 26% u kontrolnoj skupini. S obzirom na vrstu parodontitisa prevalencija bakterija bila je : za Ec 74% u CP i 83% u AP pacijenata; za Aa 37% u CP i 39% u Ap pacijenata; za Pg 16% u CP i 18% u AP pacijnata. Ispitanici s Ec imali su niže PI, SBI i CAL vrijednosti. Oboljeli od PTSP-a imali su najveÄe PI i SBI vrijednosti, a bili su sliÄni PP pacijentima po PD i CAL vrijednostima. SliÄna prevalencija parodontnih patogena u PTSP i PP pacijenata pokazuje da se oboljeli od PTSP-a mogu smatrati riziÄnom skupinom za inicijaciju i progresiju parodontne bolesti.Progression of periodontitis depends on simultaneous occurrence of several factors of the disease. Besides subgingival plaque microorganisms, stress, through modifying host response, has been shown to contribute to tissue destruction seen in periodontal disease. War stress can cause permanent effects, including post-traumatic stress dosorder (PTSD). We wanted to study the prevalence of periodontal pathogens, association of these microorganisms with the clinical parameters of periodontitis and the influence of war stress on periodontal health in patients with PTSD. The investigation was conducted on 130 subjects: 50 war veterans diagnosed and treated for PTSD, 50 subjects with progressive periodontitis (PP) and 30 periodontally healthy subjects. The progressive periodontitis group included subjects with chronic (CP) and agressive peiodontitis (AP). Only the war veterans group has been exposed directly to war stress. Detailed periodontal examination and subgingival microbial ampling were conducted for each participant. Clinical examination included assessment of plaque index (PI), sulcus bleeding index (SBI), probing depth (PD) and clinical attachment loss (CAL). Polymerase chain reaction (PCR) was used for microbial identification of the following bacteria: Actinobacillus actinomycetemcomitans (Aa), Porphyromonas gingivalis (Pg) and Eikenella corrodens (Ec). The prevalence in all groups for Ec was 81%, for Aa 36% and for Pg 19%. Prevalence for Ec was 74% in PTSD group, 80% in PP group and 93% in periodontally healthy subjects. Prevalence for Aa was 30% in PTSD group, 46% in PP group and 30% in periodontally healthy subjects. Prevalence of Pg was 16% in PTSD group, 16% in PP group and 26% in the control gorup. Considering the periodontal diagnosis, the prevalence of bacteria was : for Ec 74% in CP and 83% in AP patients; for Aa 37% in CP and 39% in AP patients; for Pg 16% in CP and 18% in AP patients. Subjects harbouring Ec had lower PI, SBI and CAL values. PTSD patients had the highest PI and SBI values, and concerning PD an CAL were similar to PP patients. The similar prevalence of periodontal pathogens in PTSD and PP subjects indicates PTSD subjects as a risk group for periodontal disease initiation or progression
Novel Alleles of the D16S752 Polymorphic Genetic Marker Linked to E-Cadherin Gene ā A Potential Population Marker
Seven DNA variants that polymorphic genetic marker D16S752 reveals in Croatian
population are reported in this paper. The marker is a GATA tetranucleotide repeat
linked to human E-cadherin gene (CDH1). Prior studies involving this marker revealed
only four DNA allele variants. The reported DNA variants contribute to the collection of
hypervariable DNA polymorphisms data useful in the field of anthropological and population
genetic and forensic medicine