Can Tourism Business Development Cause Environmental Damage? A Perspective of Spatial Planning

Tourism industry is closely related to the environment. In tourism industry, natural or environmental beauty is one of the attractions for tourists. In reality, however, the development of tourism not only provides positive impacts on the environment but also results in significant negative impacts. The purpose of this research is to determine the effect of tourism business development on environmental damage and to determine preventive efforts to prevent the occurrence of environmental damage. This research is an empirical law study. This research used primary data and secondary data. Data were collected through interview technique to three officers in three different government institutions. The sample was determined using non-probability/non-random sampling technique. The findings showed the development of the tourism industry absolutely has a direct impact on the environment. This can be seen from the tourism business that causes environmental damage either directly or indirectly to the physical, chemical and/or biological changes to the environment that exceed the standard criteria of environmental degradation. Preventive measures that can be taken to prevent environmental damage are through control and licensing system

Strategi Pengolahan Sampah Plastik di Tempat Wisata

Kehadiran sampah plastik di tempat wisata menjadi salah satu masalah serius di berbagai tempat wisata di dunia termasuk di Indonesia. Sampah plastik dapat menyebabkan pencemaran serta kerusakan lingkungan membahayakan kesehatan penduduk di sekitarnya. Meskipun sering dianggap enteng, mengolah sampah plastik dari tempat wisata memerlukan ketelitian, ketalatenan dan usaha sungguh-sungguh. Namun cara yang paling sederhana untuk mencegah pencemaran plastik di tempat wisata dengan meningkatkan kepedulian pengunjung terhadap kebersihan tempat wisata melalui metode 3R (reduce, reuse dan recycle)

l-Arginine stimulates proliferation and prevents endotoxin-induced death of intestinal cells

This study tested the hypothesis that l-arginine (Arg) may stimulate cell proliferation and prevent lipopolysaccharide (LPS)-induced death of intestinal cells. Intestinal porcine epithelial cells (IPEC-1) were cultured for 4 days in Arg-free Dulbecco’s modified Eagle’s-F12 Ham medium (DMEM-F12) containing 10, 100 or 350 μM Arg and 0 or 20 ng/ml LPS. Cell numbers, protein concentrations, protein synthesis and degradation, as well as mammalian target of rapamycin (mTOR) and Toll-like receptor 4 (TLR4) signaling pathways were determined. Without LPS, IPEC-1 cells exhibited time- and Arg-dependent growth curves. LPS treatment increased cell death and reduced protein concentrations in IPEC-1 cells. Addition of 100 and 350 μM Arg to culture medium dose-dependently attenuated LPS-induced cell death and reduction of protein concentrations, in comparison with the basal medium containing 10 μM Arg. Furthermore, supplementation of 100 and 350 μM Arg increased protein synthesis and reduced protein degradation in both control and LPS-treated IPEC-1 cells. Consistent with the data on cell growth and protein turnover, addition of 100 or 350 μM Arg to culture medium increased relative protein levels for phosphorylated mTOR and phosphorylated ribosomal protein S6 kinase-1, while reducing the relative levels of TLR4 and phosphorylated levels of nuclear factor-κB in LPS-treated IPEC-1 cells. These results demonstrate a protective effect of Arg against LPS-induced enterocyte damage through mechanisms involving mTOR and TLR4 signaling pathways, as well as intracellular protein turnover

PERANCANGAN DAN REALISASI PENGHITUNG FREKUENSI DETAK JANTUNG BERBASIS MIKROKONTROLER AT89S52

Jantung adalah organ penting dalam tubuh manusia yang difungsikan untuk memompa darah ke seluruh tubuh. Salah satu cara untuk mengetahui bagaimana kondisi jantung yaitu dengan mengetahui frekuensi detak jantung. Pada tulisan ini akan dirancang dan dibuat sistem pendeteksi dan penghitung frekuensi detak jantung berbasis mikrokontroler AT89S52. Pada sistem ini mikrokontroler AT89S52 bekerja berdasarkan masukan dari rangkaian pendeteksi detak jantung. Mikrokontroler akan menambahkan dan menampilkan angka pada layar LCD pada setiap detak jantung yang terdeteksi. Sistem juga dilengkapi dengan RTC DS12887 sebagai sumber waktu konstan. Dengan dirancang dan direalisasikannya perangkat untuk menghitung frekuensi detak jantung berbasis mikrokontroler AT89S52 ini, diharapkan dapat digunakan untuk mengetahui frekuensi detak jantung secara umum serta dengan ukurannya yang relatif kecil diharapkan pengukuran frekuensi detak jantung menjadi lebih cepat dan mudah dilakukan.<br /