151 research outputs found
Constraint of the Nuclear Dissipation Coefficient in Fission of Hypernuclei
Experimental studies of nuclear fission induced by fusion, transfer,
spallation, fragmentation, and electromagnetic reactions in combination with
state-of-the-art calculations are successful to investigate the nuclear
dissipation mechanism in normal nuclear matter, containing only nucleons. The
dissipation mechanism has been widely studied by the use of many different
fission observables and nowadays the dissipation coefficients involved in
transport theories are well constrained. However, the existence of hypernuclei
and the possible presence of hyperons in neutron stars make it necessary to
extend the investigation of the nuclear dissipation coefficient to the
strangeness sector. In this Letter, we use fission reactions of hypernuclei to
constrain for the first time the dissipation coefficient in hypernuclear
matter, observing that this coefficient increases a factor of 6 in presence of
a single -hyperon with respect to normal nuclear matter.Comment: 6 pages, 2 figure
Intestinal current measurement versus nasal potential difference measurements for diagnosis of cystic fibrosis: a caseâcontrol study
Project TENDR: Targeting environmental neuro-developmental risks. the TENDR consensus statement
Children in America today are at an unacceptably high risk of developing neurodevelopmental disorders that affect the brain and nervous system including autism, attention deficit hyperactivity disorder, intellectual disabilities, and other learning and behavioral disabilities. These are complex disorders with multiple causesâgenetic, social, and environmental. The contribution of toxic chemicals to these disorders can be prevented. Approach: Leading scientific and medical experts, along with childrenâs health advocates, came together in 2015 under the auspices of Project TENDR: Targeting Environmental Neuro-Developmental Risks to issue a call to action to reduce widespread exposures to chemicals that interfere with fetal and childrenâs brain development. Based on the available scientific evidence, the TENDR authors have identified prime examples of toxic chemicals and pollutants that increase childrenâs risks for neurodevelopmental disorders. These include chemicals that are used extensively in consumer products and that have become widespread in the environment. Some are chemicals to which children and pregnant women are regularly exposed, and they are detected in the bodies of virtually all Americans in national surveys conducted by the U.S. Centers for Disease Control and Prevention. The vast majority of chemicals in industrial and consumer products undergo almost no testing for developmental neurotoxicity or other health effects. Conclusion: Based on these findings, we assert that the current system in the United States for evaluating scientific evidence and making health-based decisions about environmental chemicals is fundamentally broken. To help reduce the unacceptably high prevalence of neurodevelopmental disorders in our children, we must eliminate or significantly reduce exposures to chemicals that contribute to these conditions. We must adopt a new framework for assessing chemicals that have the potential to disrupt brain development and prevent the use of those that may pose a risk. This consensus statement lays the foundation for developing recommendations to monitor, assess, and reduce exposures to neurotoxic chemicals. These measures are urgently needed if we are to protect healthy brain development so that current and future generations can reach their fullest potential
Salivary Amylase Induction by Tannin-Enriched Diets as a Possible Countermeasure Against Tannins
Changes in the salivary protein profile of morbidly obese women either previously subjected to bariatric surgery or not
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