17 research outputs found
New insights into seasonal foraging ranges and migrations of minke whales from the Salish Sea and coastal British Columbia.
In the Salish Sea and coastal waters of British Columbia, minke whales are known to establish small home ranges during the feeding season. Beyond the feeding season little is known of their movements or distribution. To determine movement patterns of minke whales in these waters we used photo-identification data that were collected opportunistically from 2005-2012. These data were from four non-overlapping areas between 48ºN and 53ºN. Despite year-round search effort, minke whales were only encountered between April and October. Most of the 44 unique minke whales identified in 405 encounters displayed fidelity to areas both within and among feeding seasons. Five of these individuals also made relatively large-scale intra-annual movements between areas on six occasions. They were documented to move up to at least 424km in a northerly direction early in the season and up to at least 398km in a southerly direction late in the season. We believe that the seasonal patterns of these movements provide new insight into the foraging ranges and migrations of individuals. Ecological markers provide further evidence that the minke whales we photographed undertake annual long distance migrations. Scars believed to be from cookiecutter shark bites were observed on 43 individuals and the majority of minke whales documented with good quality images each year had acquired new scars since the previous feeding season. Furthermore, the commensal barnacle Xenobalanus globicipitis was observed on three individuals. Since these sharks and barnacles are from warm waters, it can be inferred that they interacted with the minke whales at lower latitudes. These findings may have important implications for our understanding of minke whale populations in the Salish Sea and the management of this species in the North Pacific
Conventional Dendritic Cells Mount a Type I IFN Response against Candida spp. Requiring Novel Phagosomal TLR7-Mediated IFN-β Signaling
Access and allocation in earth system governance: Water and climate change compared
A significant percentage of the global population does not yet have access to safe drinking water, sufficient food or energy to live in dignity. There is a continuous struggle to allocate the earth's resources among users and uses. This article argues that distributional problems have two faces: access to basic resources or ecospace; and, the allocation of environmental resources, risks, burdens, and responsibilities for causing problems. Furthermore, addressing problems of access and allocation often requires access to social processes (science, movements and law). Analysts, however, have tended to take a narrow, disciplinary approach although an integrated conceptual approach may yield better answers. This article proposes a multi-disciplinary perspective to the problem of access and allocation and illustrates its application to water management and climate change. © The Author(s) 2010
Determination of traces of Sn, Pb, Cu, Cd in zinc and zinc salts by anodic stripping voltammetry
Through the Eyes of the Beholder—In Quest of Queer Approaches to Legal Writing on Water and Gender
Identification of a 52 kb deletion downstream of the SOST gene in patients with Buchem disease
Identification of a 52 kb deletion downstream of the SOST gene in patients with van Buchem disease
Van Buchem disease is an autosomal recessive skeletal dysplasia characterised by generalised bone overgrowth, predominantly in the skull and mandible. Clinical complications including facial nerve palsy, optic atrophy, and impaired hearing occur in most patients. These features are very similar to those of sclerosteosis and the two conditions are only differentiated by the hand malformations and the tall stature appearing in sclerosteosis. Using an extended Dutch inbred van Buchem family and two inbred sclerosteosis families, we mapped both disease genes to the same region on chromosome 17q12-q21, supporting the hypothesis that van Buchem disease and sclerosteosis are caused by mutations in the same gene. In a previous study, we positionally cloned a novel gene, called SOST, from the linkage interval and identified three different, homozygous mutations in the SOST gene in sclerosteosis patients leading to loss of function of the underlying protein. The present study focuses on the identification of a 52 kb deletion in all patients from the van Buchem family. The deletion, which results from a homologous recombination between Alu sequences, starts approximately 35 kb downstream of the SOST gene. Since no evidence was found for the presence of a gene within the deleted region, we hypothesise that the presence of the deletion leads to a down regulation of the transcription of the SOST gene by a cis regulatory action or a position effect