CA-ARBAC: privacy preserving using context-aware role-based access control on Android permission system

Existing mobile platforms are based on manual way of granting and revoking permissions to applications. Once the user grants a given permission to an application, the application can use it without limit, unless the user manually revokes the permission. This has become the reason for many privacy problems because of the fact that a permission that is harmless at some occasion may be very dangerous at another condition. One of the promising solutions for this problem is context-aware access control at permission level that allows dynamic granting and denying of permissions based on some predefined context. However, dealing with policy configuration at permission level becomes very complex for the user as the number of policies to configure will become very large. For instance, if there are A applications, P permissions, and C contexts, the user may have to deal with A × P × C number of policy configurations. Therefore, we propose a context-aware role-based access control model that can provide dynamic permission granting and revoking while keeping the number of policies as small as possible. Although our model can be used for all mobile platforms, we use Android platform to demonstrate our system. In our model, Android applications are assigned roles where roles contain a set of permissions and contexts are associated with permissions. Permissions are activated and deactivated for the containing role based on the associated contexts. Our approach is unique in that our system associates contexts with permissions as opposed to existing similar works that associate contexts with roles. As a proof of concept, we have developed a prototype application called context-aware Android role-based access control. We have also performed various tests using our application, and the result shows that our model is working as desired

Operational use of radar sensors for personal protection and area

Diplomová práce se zabývá možností použití radiolokačních senzorů, jako osobního prostředku včasné výstrahy před nenadálým útokem na chráněnou osobu ze zadní strany a dále jejich využitím pro mobilní zastřežení chráněného prostoru s dálkovým přenosem alarmu. Na základě provedeného měření reakčních dob osob na audiální percepci, byly navrženy experimenty, které prověřují vhodnost použití zkonstruovaného radiolokačního senzoru pro ochranu osob, tento senzor byl použit i pro ověření mobilního zastřežení chráněného prostoru. Závěrem jsou uvedeny výsledky práce a diskuse, kde jsou uvedeny možná řešení pro zdokonalení zkonstruovaného radiolokačního senzoru.The thesis deals with the possibility of using radar sensors, such as a personal instrument of early warning before the sudden attack on the protected person from the back and forth using their mobile arming of the protected area with remote transmission of alarm. Based on the measurement of reaction times of people to audio perception, were designed experiments that examine the appropriateness of using a radar sensor constructed to protect people, this sensor was also used to verify the mobile arming of the protected area. Conclusion contains the results of the work and discussions for a possible solution for improving constructed radar sensor.060 - Katedra bezpečnostních služebvýborn

Kaplan–Meier survival curves of rats with tracheal stenosis.

<p>At the start of the study, 10 untreated rats and nine FIR-SeV/ΔF-treated rats were included. A survival analysis was performed using the log-rank test. The FIR-SeV/ΔF-treated animals (solid line) displayed a significantly higher survival rate than the untreated control animals (dotted line) five days following the scraping of the tracheal mucosa. *<i>P</i> < 0.05 using the log-rank test.</p

Representative H&E-stained axial sections of the rat trachea five days following the scraping of the tracheal mucosa.

<p>The untreated controls (tracheal scraping only (n = 10)), (<b>a, c</b>) showed both hyperplasia of the airway epithelium and a thickened submucosal layer with extensive fibrosis, angiogenesis, and collagen deposition causing lumen stenosis. The FIR-SeV/ΔF-treated animals, in which FIR-SeV/ΔF was administered into the tracheal mucosa following tracheal scraping (n = 9), (<b>b, d</b>), showed a reduction in the extent of hyperplasia of the tracheal epithelium. The scale bar in (<b>a, b</b>) and (<b>c, d</b>) indicates 500 μm and 100 μm, respectively.</p

Quantitative analysis of tracheal stenosis.

<p>The percentage of stenosis five days following tracheal scraping was calculated using the following formula: (1 − the area of the mucosal surface lumen (solid line)/the area of the tracheal cartilage lumen (dotted line)) × 100 (<b>a</b>). The animals in which FIR-SeV/ΔF was administered (black) displayed a significantly lower percentage of stenosis than the untreated controls (white) (<b>b</b>). The results are expressed as the mean + SEM (bars). *P < 0.05 using the Mann–Whitney U test.</p

Daphne pseudo-mezereum A. Gray

原著和名: オニシバリ科名: ジンチョウゲ科 = Thymelaeaceae採集地: 千葉県 清澄山 (安房 清澄山)採集日: 1963/3/21採集者: 萩庭丈壽整理番号: JH037304国立科学博物館整理番号: TNS-VS-98730

Additional file 1: Figure S1. of Combination of a third generation bisphosphonate and replication-competent adenoviruses augments the cytotoxicity on mesothelioma

Genetic deletion or loss of expression of the p14 and p16 genes in mesothelioma. (A) Polymerase chain reactions to detect the INK4A/ARF region, encoding the p14 and p16 genes. The p14 gene is comprised by exon 1β, 2 and 3, and the p16 is exon 1α, 2 and 3. NCI-H28, EHMES-10 and NCI-H2452 cells were defective of the p14 and p16 genes. (B) Reverse transcription-polymerase chain reactions to detect the p14 and the p16 transcripts with primers that amplified between exon 1β and 2 (for the p14) and exon 1α and 2 (for the p16). Met-5A cells and GAPDH (glyceraldehyde-3-phosphate dehydrogenase) were used for a positive and a loading control, respectively. MSTO-211H and NCI-H226 cells were negative for the p14 and the p16 transcripts. (PPTX 46 kb

Combinatory effects with ZOL and CDDP in an orthotopic animal model.

<p>MSTO-211H cells (1×10⁶) were inoculated into the pleural cavity of BALB/c <i>nu/nu</i> mice (n = 6) (day 1), and then ZOL (25 µg, day 3) was administrated into the pleural cavity and/or CDDP (100 µg, day 5) into the peritoneal cavity (CDDP). PBS was used as a control. Tumor weights were measured on day 24. The SE bars are also shown. * P<0.05, ** P<0.01.</p

Combinatory effects of ZOL and CDDP.

<p>(A) Cells were treated with different doses of ZOL and CDDP at a constant concentration ratio (ZOL∶CDDP = 3∶2 at each concentration in MSTO-211H and 4∶3 in EHMES-10 cells) for 3 days and the cell viabilities were measured with the WST assay. Means of triplicated samples and the SD bars are shown. (B) CI values based on the cell viabilities as shown in (A) were calculated at different Fa points with CalcuSyn software. The SE bars are also indicated. (C) Sub-G1 phase populations of PI-stained MSTO-211H cells that were treated with ZOL (15 µM) and/or CDDP (4 µM) for 24 h were calculated with flow cytometry. Means of triplicated samples and the SE bars are shown. * P<0.01.</p