979 research outputs found
Stability of central finite difference schemes for the Heston PDE
This paper deals with stability in the numerical solution of the prominent
Heston partial differential equation from mathematical finance. We study the
well-known central second-order finite difference discretization, which leads
to large semi-discrete systems with non-normal matrices A. By employing the
logarithmic spectral norm we prove practical, rigorous stability bounds. Our
theoretical stability results are illustrated by ample numerical experiments
Continuous Equilibrium in Affine and Information-Based Capital Asset Pricing Models
We consider a class of generalized capital asset pricing models in continuous
time with a finite number of agents and tradable securities. The securities may
not be sufficient to span all sources of uncertainty. If the agents have
exponential utility functions and the individual endowments are spanned by the
securities, an equilibrium exists and the agents' optimal trading strategies
are constant. Affine processes, and the theory of information-based asset
pricing are used to model the endogenous asset price dynamics and the terminal
payoff. The derived semi-explicit pricing formulae are applied to numerically
analyze the impact of the agents' risk aversion on the implied volatility of
simultaneously-traded European-style options.Comment: 24 pages, 4 figure
Generalized pricing formulas for stochastic volatility jump diffusion models applied to the exponential Vasicek model
Path integral techniques for the pricing of financial options are mostly
based on models that can be recast in terms of a Fokker-Planck differential
equation and that, consequently, neglect jumps and only describe drift and
diffusion. We present a method to adapt formulas for both the path-integral
propagators and the option prices themselves, so that jump processes are taken
into account in conjunction with the usual drift and diffusion terms. In
particular, we focus on stochastic volatility models, such as the exponential
Vasicek model, and extend the pricing formulas and propagator of this model to
incorporate jump diffusion with a given jump size distribution. This model is
of importance to include non-Gaussian fluctuations beyond the Black-Scholes
model, and moreover yields a lognormal distribution of the volatilities, in
agreement with results from superstatistical analysis. The results obtained in
the present formalism are checked with Monte Carlo simulations.Comment: 9 pages, 2 figures, 1 tabl
Bessel Process and Conformal Quantum Mechanics
Different aspects of the connection between the Bessel process and the
conformal quantum mechanics (CQM) are discussed. The meaning of the possible
generalizations of both models is investigated with respect to the other model,
including self adjoint extension of the CQM. Some other generalizations such as
the Bessel process in the wide sense and radial Ornstein- Uhlenbeck process are
discussed with respect to the underlying conformal group structure.Comment: 28 Page
A robust spectral method for solving Hestonās model
In this paper, we consider the Hestonās volatility model (Heston in Rev.
Financ. Stud. 6: 327ā343, 1993]. We simulate this model using a combination of the
spectral collocation method and the Laplace transforms method. To approximate the
two dimensional PDE, we construct a grid which is the tensor product of the two
grids, each of which is based on the Chebyshev points in the two spacial directions.
The resulting semi-discrete problem is then solved by applying the Laplace transform
method based on Talbotās idea of deformation of the contour integral (Talbot in IMA
J. Appl. Math. 23(1): 97ā120, 1979)
A Subset of Replication Proteins Enhances Origin Recognition and Lytic Replication by the Epstein-Barr Virus ZEBRA Protein
ZEBRA is a site-specific DNA binding protein that functions as a transcriptional activator and as an origin binding protein. Both activities require that ZEBRA recognizes DNA motifs that are scattered along the viral genome. The mechanism by which ZEBRA discriminates between the origin of lytic replication and promoters of EBV early genes is not well understood. We explored the hypothesis that activation of replication requires stronger association between ZEBRA and DNA than does transcription. A ZEBRA mutant, Z(S173A), at a phosphorylation site and three point mutants in the DNA recognition domain of ZEBRA, namely Z(Y180E), Z(R187K) and Z(K188A), were similarly deficient at activating lytic DNA replication and expression of late gene expression but were competent to activate transcription of viral early lytic genes. These mutants all exhibited reduced capacity to interact with DNA as assessed by EMSA, ChIP and an in vivo biotinylated DNA pull-down assay. Over-expression of three virally encoded replication proteins, namely the primase (BSLF1), the single-stranded DNA-binding protein (BALF2) and the DNA polymerase processivity factor (BMRF1), partially rescued the replication defect in these mutants and enhanced ZEBRA's interaction with oriLyt. The findings demonstrate a functional role of replication proteins in stabilizing the association of ZEBRA with viral DNA. Enhanced binding of ZEBRA to oriLyt is crucial for lytic viral DNA replication
Explaining High Economic Growth in Small Tourism Countries with a Dynamic General Equilibrium Model
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